Carboxyethylpyrrole oxidative protein modifications stimulate neovascularization: Implications for age-related macular degeneration. - Wikidata - wikimedia - TriplyDB

Carboxyethylpyrrole oxidative protein modifications stimulate neovascularization: Implications for age-related macular degeneration.

Carboxyethylpyrrole oxidative protein modifications stimulate neovascularization: Implications for age-related macular degeneration.

http://www.wikidata.org/entity/Q33255664

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The proteomics of drusen Lack of involvement of CEP adducts in TLR activation and in angiogenesis The innate immune response to products of phospholipid peroxidation CEP biomarkers as potential tools for monitoring therapeutics Circulating Autoantibodies in Age-Related Macular Degeneration Recognize Human Macular Tissue Antigens Implicated in Autophagy, Immunomodulation, and Protection from Oxidative Stress and Apoptosis Complement factor h is critical in the maintenance of retinal perfusion.Polyunsaturated phospholipids promote the oxidation and fragmentation of gamma-hydroxyalkenals: formation and reactions of oxidatively truncated ether phospholipids A 13-Oxo-9,10-epoxytridecenoate phospholipid analogue of the genotoxic 4,5-epoxy-2E-decenal: detection in vivo, chemical synthesis, and adduction with DNA.Retinal ultrastructure of murine models of dry age-related macular degeneration (AMD).Quantitative proteomics: comparison of the macular Bruch membrane/choroid complex from age-related macular degeneration and normal eyes Nutritional risk factors for age-related macular degeneration.Iron, the retina and the lens: a focused review.Inflammation and Cell Death in Age-Related Macular Degeneration: An Immunopathological and Ultrastructural Model.Complement activation and choriocapillaris loss in early AMD: implications for pathophysiology and therapy.An (1)O2 route to γ-hydroxyalkenal phospholipids by vitamin E-induced fragmentation of hydroperoxydiene-derived endoperoxides Progress and perspectives on the role of RPE cell inflammatory responses in the development of age-related macular degeneration.Discovery of carboxyethylpyrroles (CEPs): critical insights into AMD, autism, cancer, and wound healing from basic research on the chemistry of oxidized phospholipids Molecular mechanisms of (R,R)ZX-5 on NO synthesis and its anti-angiogenic effect Receptor-Mediated Mechanism Controlling Tissue Levels of Bioactive Lipid Oxidation Products Structural identification and cardiovascular activities of oxidized phospholipids.Engagement of platelet toll-like receptor 9 by novel endogenous ligands promotes platelet hyperreactivity and thrombosis.Retinal pigment epithelium lipofuscin proteomics.The future implications and indications of anti-vascular endothelial growth factor therapy in ophthalmic practice.Carboxyethylpyrrole plasma biomarkers in age-related macular degeneration.Lipid metabolites in the pathogenesis and treatment of neovascular eye disease.Assessing susceptibility to age-related macular degeneration with proteomic and genomic biomarkers.Pretreatment with pyridoxamine mitigates isolevuglandin-associated retinal effects in mice exposed to bright light.Plasma protein pentosidine and carboxymethyllysine, biomarkers for age-related macular degeneration Aging is not a disease: distinguishing age-related macular degeneration from aging.Synthesis and structural characterization of carboxyethylpyrrole-modified proteins: mediators of age-related macular degeneration.Oxidative stress in angiogenesis and vascular disease Oxidation and age-related macular degeneration: insights from molecular biology.The pathogenic role of Maillard reaction in the aging eye.Oxidation as "the stress of life".Therapeutic potential of targeting lipid aldehydes and lipoxidation end-products in the treatment of ocular disease.Inflammation and oxidative stress in angiogenesis and vascular disease.Age-related macular degeneration and changes in the extracellular matrix.Retinal pigment epithelium and microglia express the CD5 antigen-like protein, a novel autoantigen in age-related macular degeneration.4-Hydroxy-7-oxo-5-heptenoic Acid Lactone Induces Angiogenesis through Several Different Molecular Pathways.Metabolism of 4-Hydroxy-7-oxo-5-heptenoic Acid (HOHA) Lactone by Retinal Pigmented Epithelial Cells.

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Carboxyethylpyrrole oxidative protein modifications stimulate neovascularization: Implications for age-related macular degeneration.

http://www.wikidata.org/entity/Q33255664

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2006 nî lūn-bûn

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2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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2006年の論文

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Carboxyethylpyrrole oxidative ...... -related macular degeneration.

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Carboxyethylpyrrole oxidative ...... -related macular degeneration.

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Carboxyethylpyrrole oxidative ...... -related macular degeneration.

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PNAS.0601552103

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Proceedings of the National Academy of Sciences of the United States of America

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Carboxyethylpyrrole oxidative ...... -related macular degeneration.

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Amit Vasanji

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Bela Anand-Apte

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John W Crabb

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Jonathan Sears

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Liang Lu

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Quteba Ebrahem

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Robert G Salomon

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Xiaorong Gu

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P2860

A common haplotype in the complement regulatory gene factor H (HF1/CFH) predisposes individuals to age-related macular degeneration

A randomized, placebo-controlled, clinical trial of high-dose supplementation with vitamins C and E, beta carotene, and zinc for age-related macular degeneration and vision loss: AREDS report no. 8

Complement factor H polymorphism in age-related macular degeneration

Complement factor H variant increases the risk of age-related macular degeneration

Role of complement and complement membrane attack complex in laser-induced choroidal neovascularization

Identification of a novel family of oxidized phospholipids that serve as ligands for the macrophage scavenger receptor CD36

Complement factor H polymorphism and age-related macular degeneration

Maximum tolerated dose of a humanized anti-vascular endothelial growth factor antibody fragment for treating neovascular age-related macular degeneration.

Targeted disruption of the FGF2 gene does not prevent choroidal neovascularization in a murine model.

Oxidative damage and age-related macular degeneration

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Kutralanathan Renganathan

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6849595

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neovascularization

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