Neurod1 suppresses hair cell differentiation in ear ganglia and regulates hair cell subtype development in the cochlea.
about
Beyond generalized hair cells: molecular cues for hair cell typesThe genetics of hair cell development and regenerationConditional gene expression in the mouse inner ear using Cre-loxP.Sensory hair cell development and regeneration: similarities and differencesWhere hearing starts: the development of the mammalian cochleaLGR4 and LGR5 Regulate Hair Cell Differentiation in the Sensory Epithelium of the Developing Mouse Cochlea.Neurog1 can partially substitute for Atoh1 function in hair cell differentiation and maintenance during organ of Corti developmentThe quest for restoring hearing: Understanding ear development more completely.N-Myc and L-Myc are essential for hair cell formation but not maintenanceConditional deletion of Atoh1 using Pax2-Cre results in viable mice without differentiated cochlear hair cells that have lost most of the organ of Corti.Inner ear development: building a spiral ganglion and an organ of Corti out of unspecified ectodermEvolution and development of the tetrapod auditory system: an organ of Corti-centric perspective.Evolution of vertebrate mechanosensory hair cells and inner ears: toward identifying stimuli that select mutation driven altered morphologies.The molecular basis of making spiral ganglion neurons and connecting them to hair cells of the organ of CortiDissecting the molecular basis of organ of Corti development: Where are we now?Signaling and Transcription Factors during Inner Ear Development: The Generation of Hair Cells and Otic Neurons.Nonviral Reprogramming of Human Wharton's Jelly Cells Reveals Differences Between ATOH1 Homologues.Evolving gene regulatory networks into cellular networks guiding adaptive behavior: an outline how single cells could have evolved into a centralized neurosensory system.The Role of Atonal Factors in Mechanosensory Cell Specification and FunctionConnecting the ear to the brain: Molecular mechanisms of auditory circuit assembly.Atoh1 expression and function during auditory hair cell regeneration in post-hatch chickens.A review of gene delivery and stem cell based therapies for regenerating inner ear hair cellsCorrect timing of proliferation and differentiation is necessary for normal inner ear development and auditory hair cell viability.Expression and function of scleraxis in the developing auditory systemUnderstanding the evolution and development of neurosensory transcription factors of the ear to enhance therapeutic translation.Atoh1, an essential transcription factor in neurogenesis and intestinal and inner ear development: function, regulation, and context dependency.BDNF gene therapy induces auditory nerve survival and fiber sprouting in deaf Pou4f3 mutant mice.Regeneration of Hair Cells: Making Sense of All the Noise.Conditional deletion of N-Myc disrupts neurosensory and non-sensory development of the ear.Mutational ataxia resulting from abnormal vestibular acquisition and processing is partially compensated forDirecting human induced pluripotent stem cells into a neurosensory lineage for auditory neuron replacement.Insights into electrosensory organ development, physiology and evolution from a lateral line-enriched transcriptomeA novel Atoh1 "self-terminating" mouse model reveals the necessity of proper Atoh1 level and duration for hair cell differentiation and viability.Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.The prosensory function of Sox2 in the chicken inner ear relies on the direct regulation of Atoh1.Opportunities and limits of the one gene approach: the ability of Atoh1 to differentiate and maintain hair cells depends on the molecular contextIn vivo generation of immature inner hair cells in neonatal mouse cochleae by ectopic Atoh1 expressionInner ear hair cells deteriorate in mice engineered to have no or diminished innervationOveractivation of Notch1 signaling induces ectopic hair cells in the mouse inner ear in an age-dependent manner.Expression and Localization of CaBP Ca2+ Binding Proteins in the Mouse Cochlea.
P2860
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P2860
Neurod1 suppresses hair cell differentiation in ear ganglia and regulates hair cell subtype development in the cochlea.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@ast
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@en
type
label
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@ast
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@en
prefLabel
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@ast
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@en
P2093
P2860
P1433
P1476
Neurod1 suppresses hair cell d ...... pe development in the cochlea.
@en
P2093
Israt Jahan
Jennifer Kersigo
P2860
P304
P356
10.1371/JOURNAL.PONE.0011661
P407
P577
2010-07-22T00:00:00Z