Reactive oxygen species are required for maintenance and differentiation of primary lung fibroblasts in idiopathic pulmonary fibrosis.
about
Therapeutic targeting of redox signaling in myofibroblast differentiation and age-related fibrotic diseaseTGF-β signaling in tissue fibrosis: redox controls, target genes and therapeutic opportunitiesSystemic sclerosis (scleroderma) and cancer risk: systematic review and meta-analysis of observational studies.MicroRNAs in idiopathic pulmonary fibrosis: involvement in pathogenesis and potential use in diagnosis and therapeuticsNADPH oxidase activation is required in reactive oxygen species generation and cell transformation induced by hexavalent chromiumEnvironmental particulate (PM2.5) augments stiffness-induced alveolar epithelial cell mechanoactivation of transforming growth factor betaRole of areca nut induced JNK/ATF2/Jun axis in the activation of TGF-β pathway in precancerous Oral Submucous Fibrosis.Sulforaphane prevents bleomycin‑induced pulmonary fibrosis in mice by inhibiting oxidative stress via nuclear factor erythroid 2‑related factor‑2 activation.Age-dependent decline in mouse lung regeneration with loss of lung fibroblast clonogenicity and increased myofibroblastic differentiationIncreased constitutive αSMA and Smad2/3 expression in idiopathic pulmonary fibrosis myofibroblasts is KCa3.1-dependent.The angiotensin-converting enzyme 2/angiotensin (1-7)/Mas axis protects against lung fibroblast migration and lung fibrosis by inhibiting the NOX4-derived ROS-mediated RhoA/Rho kinase pathway.Low-dose paclitaxel ameliorates pulmonary fibrosis by suppressing TGF-β1/Smad3 pathway via miR-140 upregulation.NRF2 deficiency reduces life span of mice administered thoracic irradiation.Protective roles of pulmonary rehabilitation mixture in experimental pulmonary fibrosis in vitro and in vivo.Caveolin-1 and accelerated host aging in the breast tumor microenvironment: chemoprevention with rapamycin, an mTOR inhibitor and anti-aging drugMolecular and cellular mechanisms of pulmonary fibrosis.C-type natriuretic peptide ameliorates pulmonary fibrosis by acting on lung fibroblasts in mice.Lung extracellular matrix and redox regulationSalidroside protects against bleomycin-induced pulmonary fibrosis: activation of Nrf2-antioxidant signaling, and inhibition of NF-κB and TGF-β1/Smad-2/-3 pathwaysMatrices of physiologic stiffness potently inactivate idiopathic pulmonary fibrosis fibroblasts.The matricellular protein CCN1 enhances TGF-β1/SMAD3-dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to lung injury.Detecting splicing variants in idiopathic pulmonary fibrosis from non-differentially expressed genes.Triptolide mitigates radiation-induced pulmonary fibrosis via inhibition of axis of alveolar macrophages-NOXes-ROS-myofibroblasts.Hyperoxia induces alveolar epithelial-to-mesenchymal cell transition.A Systematic Review of the Role of Dysfunctional Wound Healing in the Pathogenesis and Treatment of Idiopathic Pulmonary Fibrosis.New insights into the role of oxidative stress in scleroderma fibrosis.Interstitial lung diseases-can pathologists arrive at an etiology-based diagnosis? A critical update.Redox signaling as a therapeutic target to inhibit myofibroblast activation in degenerative fibrotic diseaseThe Role of Aging in Idiopathic Pulmonary Fibrosis.Senescent stroma promotes prostate cancer progression: the role of miR-210.The SPARC protein: an overview of its role in lung cancer and pulmonary fibrosis and its potential role in chronic airways disease.Secreted protein acidic and rich in cysteine (SPARC) is upregulated by transforming growth factor (TGF)-β and is required for TGF-β-induced hydrogen peroxide production in fibroblastsDesensitization of idiopathic pulmonary fibrosis fibroblasts to Alternaria alternata extract-mediated necrotic cell death.Mitochondrial reactive oxygen species regulate transforming growth factor-β signaling.Caveolin-1 serves as a negative effector in senescent human gingival fibroblasts during Fusobacterium nucleatum infection.Oxidative stress, tumor microenvironment, and metabolic reprogramming: a diabolic liaison.Targeting the Myofibroblastic Cancer-Associated Fibroblast Phenotype Through Inhibition of NOX4.Age-associated intracellular superoxide dismutase deficiency potentiates dermal fibroblast dysfunction during wound healing.Emodin Attenuates Bleomycin-Induced Pulmonary Fibrosis via Anti-Inflammatory and Anti-Oxidative Activities in Rats.A Chinese Herbal Formula Ameliorates Pulmonary Fibrosis by Inhibiting Oxidative Stress via Upregulating Nrf2.
P2860
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P2860
Reactive oxygen species are required for maintenance and differentiation of primary lung fibroblasts in idiopathic pulmonary fibrosis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@ast
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@en
type
label
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@ast
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@en
prefLabel
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@ast
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@en
P2093
P2860
P1433
P1476
Reactive oxygen species are re ...... idiopathic pulmonary fibrosis.
@en
P2093
Alessandro Sanduzzi
Armando Gabrielli
Domenico Grieco
Enrico V Avvedimento
Evelina Fagone
Marialuisa Bocchino
Savina Agnese
Silvia Svegliati
P2860
P304
P356
10.1371/JOURNAL.PONE.0014003
P407
P577
2010-11-16T00:00:00Z