Induction of lytic Epstein-Barr virus (EBV) infection by synergistic action of rituximab and dexamethasone renders EBV-positive lymphoma cells more susceptible to ganciclovir cytotoxicity in vitro and in vivo
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Short, discontinuous exposure to butyrate effectively sensitizes latently EBV-infected lymphoma cells to nucleoside analogue antiviral agents.Histone deacetylase inhibitors are potent inducers of gene expression in latent EBV and sensitize lymphoma cells to nucleoside antiviral agentsAdvances in Virus-Directed Therapeutics against Epstein-Barr Virus-Associated MalignanciesLMP1-Induced Sumoylation Influences the Maintenance of Epstein-Barr Virus Latency through KAP1An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Evidence that amphotericin B mediates reactivation of latent Epstein-Barr virus in Hodgkin's lymphoma allowing cytotoxicity by acyclovirBendamustine reactivates latent Epstein-Barr virus.Association between Epstein-Barr virus infection and chemoresistance to docetaxel in gastric carcinoma.Cancer and viruses: a double-edged sword.Lenalidomide, Thalidomide, and Pomalidomide Reactivate the Epstein-Barr Virus Lytic Cycle through Phosphoinositide 3-Kinase Signaling and Ikaros Expression.Arsenic mediated disruption of promyelocytic leukemia protein nuclear bodies induces ganciclovir susceptibility in Epstein-Barr positive epithelial cells.Treatment of Akata EBV-positive cells with doxorubicin causes more EBV reactivation than treatment with etoposide.Inhibitory activities of microalgal extracts against Epstein-Barr virus DNA release from lymphoblastoid cells.Co-treatment with arsenic trioxide and ganciclovir reduces tumor volume in a murine xenograft model of nasopharyngeal carcinoma.In vivo intratumoral Epstein-Barr virus replication is associated with XBP1 activation and early-onset post-transplant lymphoproliferative disorders with prognostic implications.Complete and durable responses in Primary Central Nervous System Post-Transplant Lymphoproliferative Disorder with Zidovudine, Ganciclovir, Rituximab and Dexamethasone.Therapies based on targeting Epstein-Barr virus lytic replication for EBV-associated malignancies.
P2860
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P2860
Induction of lytic Epstein-Barr virus (EBV) infection by synergistic action of rituximab and dexamethasone renders EBV-positive lymphoma cells more susceptible to ganciclovir cytotoxicity in vitro and in vivo
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@ast
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@en
type
label
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@ast
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@en
prefLabel
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@ast
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@en
P2093
P2860
P921
P1433
P1476
Induction of lytic Epstein-Bar ...... otoxicity in vitro and in vivo
@en
P2093
Hirokuni Taguchi
Isao Miyoshi
Kentaro Bandobashi
Masanori Daibata
Masayuki Kuroda
Shosuke Imai
P2860
P304
P356
10.1128/JVI.79.9.5875-5879.2005
P577
2005-05-01T00:00:00Z