Virally targeted therapies for EBV-associated malignancies.
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Regulation of the latent-lytic switch in Epstein-Barr virusControl of Kaposi's sarcoma-associated herpesvirus reactivation induced by multiple signalsSystematic identification of cellular signals reactivating Kaposi sarcoma-associated herpesvirus.Immunodominance of lytic cycle antigens in Epstein-Barr virus-specific CD4+ T cell preparations for therapyRole of the ubiquitin system and tumor viruses in AIDS-related cancer.Evaluation of a prediction protocol to identify potential targets of epigenetic reprogramming by the cancer associated Epstein Barr virusKleemeier Award Lecture 2008--the canary in the coal mine: telomeres and human healthspan.The Methylation Status and Expression of Epstein-Barr Virus Early Genes BARF1 and BHRF1 in Epstein-Barr Virus-Associated Gastric Carcinomas.LY294002 may overcome 5-FU resistance via down-regulation of activated p-AKT in Epstein-Barr virus-positive gastric cancer cells.Induction of lytic Epstein-Barr virus (EBV) infection by synergistic action of rituximab and dexamethasone renders EBV-positive lymphoma cells more susceptible to ganciclovir cytotoxicity in vitro and in vivoHistone deacetylase inhibitors in Hodgkin lymphoma.Phase I clinical trial of valacyclovir and standard of care cyclophosphamide in children with endemic Burkitt lymphoma in Malawi.Theodore E. Woodward Award: development of novel, EBV-targeted therapies for EBV-positive tumorsShort, discontinuous exposure to butyrate effectively sensitizes latently EBV-infected lymphoma cells to nucleoside analogue antiviral agents.Histone deacetylase inhibitors are potent inducers of gene expression in latent EBV and sensitize lymphoma cells to nucleoside antiviral agentsTargeted therapy for Epstein-Barr virus-associated gastric carcinoma using low-dose gemcitabine-induced lytic activation.Advances in Virus-Directed Therapeutics against Epstein-Barr Virus-Associated MalignanciesEmerging therapies for herpes viral infections (types 1 - 8).Enhanced antitumor effect of combining chemotherapy with Epstein-Barr virus (EBV)-specific cytotoxic T lymphocytes in mice with EBV-related non-Hodgkin's lymphoma.Evidence that amphotericin B mediates reactivation of latent Epstein-Barr virus in Hodgkin's lymphoma allowing cytotoxicity by acyclovirEpstein-Barr virus infection is common in inflamed gastrointestinal mucosa.Bendamustine reactivates latent Epstein-Barr virus.Laboratory assays for Epstein-Barr virus-related disease.CpG methylation analysis--current status of clinical assays and potential applications in molecular diagnostics: a report of the Association for Molecular Pathology.Role of CD4+ cytotoxic T lymphocytes in the control of viral diseases and cancer.Epstein-Barr virus-associated B-cell lymphomas: pathogenesis and clinical outcomes.Children's Oncology Group's 2013 blueprint for research: Hodgkin lymphoma.Multiple infections and cancer: implications in epidemiology.Epigenetic Pathways of Oncogenic Viruses: Therapeutic Promises.(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.Parthenolide induces apoptosis and lytic cytotoxicity in Epstein-Barr virus-positive Burkitt lymphoma.Treatment of Akata EBV-positive cells with doxorubicin causes more EBV reactivation than treatment with etoposide.Valproic acid antagonizes the capacity of other histone deacetylase inhibitors to activate the Epstein-barr virus lytic cycle.The Epstein-Barr virus (EBV)-encoded protein kinase, EBV-PK, but not the thymidine kinase (EBV-TK), is required for ganciclovir and acyclovir inhibition of lytic viral productionEpstein-Barr virus infection in immortalized nasopharyngeal epithelial cells: regulation of infection and phenotypic characterization.Upregulation of STAT3 marks Burkitt lymphoma cells refractory to Epstein-Barr virus lytic cycle induction by HDAC inhibitorsSmall molecule perturbation of the CAND1-Cullin1-ubiquitin cycle stabilizes p53 and triggers Epstein-Barr virus reactivation.Epstein-Barr virus latency type and spontaneous reactivation predict lytic induction levels.Unexpected structure of Epstein-Barr virus lytic cycle activator Zta.Activation of lytic cycle of Epstein-Barr virus by suberoylanilide hydroxamic acid leads to apoptosis and tumor growth suppression of nasopharyngeal carcinoma.
P2860
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P2860
Virally targeted therapies for EBV-associated malignancies.
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Virally targeted therapies for EBV-associated malignancies.
@ast
Virally targeted therapies for EBV-associated malignancies.
@en
type
label
Virally targeted therapies for EBV-associated malignancies.
@ast
Virally targeted therapies for EBV-associated malignancies.
@en
prefLabel
Virally targeted therapies for EBV-associated malignancies.
@ast
Virally targeted therapies for EBV-associated malignancies.
@en
P2860
P356
P1433
P1476
Virally targeted therapies for EBV-associated malignancies.
@en
P2093
Bruce F Israel
Shannon C Kenney
P2860
P2888
P304
P356
10.1038/SJ.ONC.1206548
P407
P577
2003-08-01T00:00:00Z
P5875
P6179
1040757147