Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
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Therapeutic implications of Epstein-Barr virus infection for the treatment of nasopharyngeal carcinomaRTA promoter demethylation and histone acetylation regulation of murine gammaherpesvirus 68 reactivationNewly emerging therapies targeting viral-related lymphomas.Induction of lytic Epstein-Barr virus (EBV) infection by synergistic action of rituximab and dexamethasone renders EBV-positive lymphoma cells more susceptible to ganciclovir cytotoxicity in vitro and in vivoViral response to chemotherapy in endemic burkitt lymphomaZidovudine-based lytic-inducing chemotherapy for Epstein-Barr virus-related lymphomas.Cellular microRNAs 200b and 429 regulate the Epstein-Barr virus switch between latency and lytic replication.Use of adenovirus vectors expressing Epstein-Barr virus (EBV) immediate-early protein BZLF1 or BRLF1 to treat EBV-positive tumors.Antiviral agents active against human herpesviruses HHV-6, HHV-7 and HHV-8.Theodore E. Woodward Award: development of novel, EBV-targeted therapies for EBV-positive tumorsIdentification of a new class of small molecules that efficiently reactivate latent Epstein-Barr Virus.Icaritin induces lytic cytotoxicity in extranodal NK/T-cell lymphoma.Virally targeted therapies for EBV-associated malignancies.Conserved herpesvirus kinases target the DNA damage response pathway and TIP60 histone acetyltransferase to promote virus replication.Short, discontinuous exposure to butyrate effectively sensitizes latently EBV-infected lymphoma cells to nucleoside analogue antiviral agents.Histone deacetylase inhibitors are potent inducers of gene expression in latent EBV and sensitize lymphoma cells to nucleoside antiviral agentsTargeted therapy for Epstein-Barr virus-associated gastric carcinoma using low-dose gemcitabine-induced lytic activation.Advances in Virus-Directed Therapeutics against Epstein-Barr Virus-Associated MalignanciesAzidothymidine inhibits NF-kappaB and induces Epstein-Barr virus gene expression in Burkitt lymphomaImaging virus-associated cancer.An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Cross talk between EBV and telomerase: the role of TERT and NOTCH2 in the switch of latent/lytic cycle of the virusConserved herpesvirus protein kinases.Epstein-barr virus infected gastric adenocarcinoma expresses latent and lytic viral transcripts and has a distinct human gene expression profileThe Epstein-Barr virus LF2 protein inhibits viral replicationBortezomib-induced enzyme-targeted radiation therapy in herpesvirus-associated tumors.Recent developments in the prevention and treatment of Epstein-Barr virus-associated lymphoproliferative diseases.Genipin as a novel chemical activator of EBV lytic cycle.Arsenic mediated disruption of promyelocytic leukemia protein nuclear bodies induces ganciclovir susceptibility in Epstein-Barr positive epithelial cells.Valproic acid antagonizes the capacity of other histone deacetylase inhibitors to activate the Epstein-barr virus lytic cycle.Epstein-Barr virus LF2 protein regulates viral replication by altering Rta subcellular localization.The Epstein-Barr virus (EBV)-encoded protein kinase, EBV-PK, but not the thymidine kinase (EBV-TK), is required for ganciclovir and acyclovir inhibition of lytic viral productionUpregulation of STAT3 marks Burkitt lymphoma cells refractory to Epstein-Barr virus lytic cycle induction by HDAC inhibitorsMaribavir inhibits epstein-barr virus transcription in addition to viral DNA replicationAlveolar epithelial cell injury with Epstein-Barr virus upregulates TGFbeta1 expression.Reactivation of latent Epstein-Barr virus by methotrexate: a potential contributor to methotrexate-associated lymphomas.Lytic induction therapy for Epstein-Barr virus-positive B-cell lymphomas.Co-treatment with arsenic trioxide and ganciclovir reduces tumor volume in a murine xenograft model of nasopharyngeal carcinoma.SUMO binding by the Epstein-Barr virus protein kinase BGLF4 is crucial for BGLF4 function.Identification and characterization of the conserved nucleoside-binding sites in the Epstein-Barr virus thymidine kinase.
P2860
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P2860
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
description
2001 nî lūn-bûn
@nan
2001 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@ast
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@en
type
label
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@ast
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@en
prefLabel
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@ast
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@en
P2093
P2860
P1476
Induction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analogues
@en
P2093
F M Hamzeh
J S Cannon
R F Ambinder
Y C Tanhehco
P2860
P304
P356
10.1128/AAC.45.7.2082-2091.2001
P407
P577
2001-07-01T00:00:00Z