Spinal cord injury causes a wide-spread, persistent loss of Kir4.1 and glutamate transporter 1: benefit of 17 beta-oestradiol treatment
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Age-related changes in brain support cells: Implications for stroke severityAstroglia in Thick Tissue with Super Resolution and Cellular ReconstructionKIR4.1: K+ Channel Illusion or Reality in the Autoimmune Pathogenesis of Multiple SclerosisLinolenic acid provides multi-cellular protective effects after photothrombotic cerebral ischemia in ratsEstrogen Attenuates Local Inflammasome Expression and Activation after Spinal Cord Injury.Postinjury administration of 17β-estradiol induces protection in the gray and white matter with associated functional recovery after cervical spinal cord injury in male rats.Overexpression of the astrocyte glutamate transporter GLT1 exacerbates phrenic motor neuron degeneration, diaphragm compromise, and forelimb motor dysfunction following cervical contusion spinal cord injury.Chronic at-level thermal hyperalgesia following rat cervical contusion spinal cord injury is accompanied by neuronal and astrocyte activation and loss of the astrocyte glutamate transporter, GLT1, in superficial dorsal horn.Persistent at-level thermal hyperalgesia and tactile allodynia accompany chronic neuronal and astrocyte activation in superficial dorsal horn following mouse cervical contusion spinal cord injuryEnhanced GLT-1 mediated glutamate uptake and migration of primary astrocytes directed by fibronectin-coated electrospun poly-L-lactic acid fibers.Recent advances in the pharmacologic treatment of spinal cord injuryRegulation of neurovascular coupling in autoimmunity to water and ion channelsUnidirectional photoreceptor-to-Müller glia coupling and unique K+ channel expression in Caiman retina.Human iPS cell-derived astrocyte transplants preserve respiratory function after spinal cord injury.Elevated GFAP induces astrocyte dysfunction in caudal brain regions: A potential mechanism for hindbrain involved symptoms in type II Alexander disease.Correlating Gene-specific DNA Methylation Changes with Expression and Transcriptional Activity of Astrocytic KCNJ10 (Kir4.1)GLT1 overexpression reverses established neuropathic pain-related behavior and attenuates chronic dorsal horn neuron activation following cervical spinal cord injury.Regulation of Kir4.1 expression in astrocytes and astrocytic tumors: a role for interleukin-1 βDNA methylation functions as a critical regulator of Kir4.1 expression during CNS development.GABA and central neuropathic pain following spinal cord injurySpatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats.Astrocyte dysfunction in temporal lobe epilepsy: K+ channels and gap junction coupling.The role of glial-specific Kir4.1 in normal and pathological states of the CNS.Astroglial Scarring and Seizures: A Cell Biological Perspective on Epilepsy.Reduction in expression of the astrocyte glutamate transporter, GLT1, worsens functional and histological outcomes following traumatic spinal cord injuryHormonal therapy in traumatic spinal cord injury.Effects of estrogen on functional and neurological recovery after spinal cord injury: An experimental study with rats.Neuronal release and successful astrocyte uptake of aminoacidergic neurotransmitters after spinal cord injury in lampreys.In vivo microdialysis of glutamate in ventroposterolateral nucleus of thalamus following electrolytic lesion of spinothalamic tract in rats.Early down regulation of the glial Kir4.1 and GLT-1 expression in pericontusional cortex of the old male mice subjected to traumatic brain injury.Astroglial transcriptome dysregulation in early disease of an ALS mutant SOD1 mouse model.Transcriptional regulation of the GLAST/EAAT-1 gene in rat and man.Estradiol attenuates spinal cord injury-related central pain by decreasing glutamate levels in thalamic VPL nucleus in male rats.Spatial and temporal changes in promoter activity of the astrocyte glutamate transporter GLT1 following traumatic spinal cord injury.Kir4.1-Dependent Astrocyte-Fast Motor Neuron Interactions Are Required for Peak Strength.A toll-like receptor 9 antagonist restores below-level glial glutamate transporter expression in the dorsal horn following spinal cord injury.Differential loss of KIR4.1 immunoreactivity in multiple sclerosis lesions
P2860
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P2860
Spinal cord injury causes a wide-spread, persistent loss of Kir4.1 and glutamate transporter 1: benefit of 17 beta-oestradiol treatment
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@ast
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@en
type
label
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@ast
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@en
prefLabel
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@ast
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@en
P2093
P2860
P356
P1433
P1476
Spinal cord injury causes a wi ...... f 17 beta-oestradiol treatment
@en
P2093
Candace L Floyd
Michael B McFerrin
Susan C Campbell
P2860
P304
P356
10.1093/BRAIN/AWQ049
P407
P577
2010-04-01T00:00:00Z