Woodchuck hepatitis virus infections: very rapid recovery after a prolonged viremia and infection of virtually every hepatocyte.
about
Therapeutic vaccination and immunomodulation in the treatment of chronic hepatitis B: preclinical studies in the woodchuckMolecular biology of hepatitis B virus infectionAntiviral activity of beta-L-2',3'-dideoxy-2',3'-didehydro-5-fluorocytidine in woodchucks chronically infected with woodchuck hepatitis virusEvidence that hepatocyte turnover is required for rapid clearance of duck hepatitis B virus during antiviral therapy of chronically infected ducksIdentification of BMS-200475 as a potent and selective inhibitor of hepatitis B virusHepatitis B virus molecular biology and pathogenesisPharmacodynamics of (-)-beta-2',3'-dideoxy-3'-thiacytidine in chronically virus-infected woodchucks compared to its pharmacodynamics in humans.Clonal expansion of hepatocytes during chronic woodchuck hepatitis virus infection.Immune Tolerant Chronic Hepatitis B: The Unrecognized RisksThe woodchuck as an animal model for pathogenesis and therapy of chronic hepatitis B virus infectionApoptosis and regeneration of hepatocytes during recovery from transient hepadnavirus infectionsCombination therapy with lamivudine and adenovirus causes transient suppression of chronic woodchuck hepatitis virus infections.Kinetics of hepadnavirus loss from the liver during inhibition of viral DNA synthesis.Hepatitis B virus biology.Interferons accelerate decay of replication-competent nucleocapsids of hepatitis B virusDynamics of hepatitis B virus clearance in chimpanzees.Half-life of the duck hepatitis B virus covalently closed circular DNA pool in vivo following inhibition of viral replication.CD8(+) T cells mediate viral clearance and disease pathogenesis during acute hepatitis B virus infection.Targeting Hepatitis B Virus With CRISPR/Cas9.Tumor necrosis factor alpha inhibition of hepatitis B virus replication involves disruption of capsid Integrity through activation of NF-kappaB.Identification and characterization of multiple TRIM proteins that inhibit hepatitis B virus transcription.STING agonists induce an innate antiviral immune response against hepatitis B virus.Animal models and the molecular biology of hepadnavirus infectionRapid production of neutralizing antibody leads to transient hepadnavirus infection.The role of cells refractory to productive infection in acute hepatitis B viral dynamics.The liver of woodchucks chronically infected with the woodchuck hepatitis virus contains foci of virus core antigen-negative hepatocytes with both altered and normal morphology.The level of viral antigen presented by hepatocytes influences CD8 T-cell function.Hepadnavirus Genome Replication and Persistence.Effects of lamivudine on serum albumin levels correlate with pretreatment HBV-DNA levels in cirrhotic patients.Therapeutic Antiviral Effect of the Nucleic Acid Polymer REP 2055 against Persistent Duck Hepatitis B Virus Infection.The covalently closed duplex form of the hepadnavirus genome exists in situ as a heterogeneous population of viral minichromosomes.High-level hepatitis B virus replication in transgenic mice.n-Butyrate, a cell cycle blocker, inhibits early amplification of duck hepatitis B virus covalently closed circular DNA after in vitro infection of duck hepatocytesInterleukin-12 inhibits hepatitis B virus replication in transgenic mice.Tumor necrosis factor activates a conserved innate antiviral response to hepatitis B virus that destabilizes nucleocapsids and reduces nuclear viral DNA.Blockade of NKG2D on NKT cells prevents hepatitis and the acute immune response to hepatitis B virusHepatocyte turnover during resolution of a transient hepadnaviral infection.Single-cell analysis of covalently closed circular DNA copy numbers in a hepadnavirus-infected liverLack of effect of antiviral therapy in nondividing hepatocyte cultures on the closed circular DNA of woodchuck hepatitis virus.Transcriptome-Wide Analysis of Hepatitis B Virus-Mediated Changes to Normal Hepatocyte Gene Expression.
P2860
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P2860
Woodchuck hepatitis virus infections: very rapid recovery after a prolonged viremia and infection of virtually every hepatocyte.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@ast
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@en
type
label
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@ast
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@en
prefLabel
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@ast
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@en
P2093
P2860
P1433
P1476
Woodchuck hepatitis virus infe ...... of virtually every hepatocyte.
@en
P2093
P2860
P304
P407
P577
1994-09-01T00:00:00Z