Changes in the organization of excitation-contraction coupling structures in failing human heart. - Wikidata - wikimedia - TriplyDB

Changes in the organization of excitation-contraction coupling structures in failing human heart.

Changes in the organization of excitation-contraction coupling structures in failing human heart.

http://www.wikidata.org/entity/Q33847166

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Revealing T-Tubules in Striated Muscle with New Optical Super-Resolution Microscopy Techniquess Calcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approaches Alterations in T-tubule and dyad structure in heart disease: challenges and opportunities for computational analyses Cardiomyocyte ATP production, metabolic flexibility, and survival require calcium flux through cardiac ryanodine receptors in vivo Sildenafil ameliorates left ventricular T-tubule remodeling in a pressure overload-induced murine heart failure model Analysis of tubular membrane networks in cardiac myocytes from atria and ventricles.Modeling effects of L-type ca(2+) current and na(+)-ca(2+) exchanger on ca(2+) trigger flux in rabbit myocytes with realistic T-tubule geometries Stimulated emission depletion live-cell super-resolution imaging shows proliferative remodeling of T-tubule membrane structures after myocardial infarction.Subcellular heterogeneity of ryanodine receptor properties in ventricular myocytes with low T-tubule density β-Adrenergic receptor antagonists ameliorate myocyte T-tubule remodeling following myocardial infarction.Increased expression of fatty-acid and calcium metabolism genes in failing human heart.T-tubule disease: Relationship between t-tubule organization and regional contractile performance in human dilated cardiomyopathy.Cardiac T-Tubule Microanatomy and Function.Sheet-Like Remodeling of the Transverse Tubular System in Human Heart Failure Impairs Excitation-Contraction Coupling and Functional Recovery by Mechanical Unloading.Calpain-dependent cleavage of junctophilin-2 and T-tubule remodeling in a mouse model of reversible heart failure Next-generation endomyocardial biopsy: the potential of confocal and super-resolution microscopy.Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signaling.In situ single photon confocal imaging of cardiomyocyte T-tubule system from Langendorff-perfused hearts Heterogeneity of T-Tubules in Pig Hearts.Aberrant Glycosylation in the Left Ventricle and Plasma of Rats with Cardiac Hypertrophy and Heart Failure.Mechanical modulation of the transverse tubular system of ventricular cardiomyocytes.Heterogeneous upregulation of apamin-sensitive potassium currents in failing human ventricles Caveolin and caveolae in age associated cardiovascular disease Functional integrity of the T-tubular system in cardiomyocytes depends on p21-activated kinase 1 Emerging mechanisms of T-tubule remodelling in heart failure.Remodeling of calcium handling in human heart failure.Critical roles of junctophilin-2 in T-tubule and excitation-contraction coupling maturation during postnatal development Differential calcium signaling mediated by voltage-gated calcium channels in rat retinal ganglion cells and their unmyelinated axons.Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.Ischemia/Reperfusion Injury following Acute Myocardial Infarction: A Critical Issue for Clinicians and Forensic Pathologists.Ca handling during excitation-contraction coupling in heart failure.Multilayered regulation of cardiac ion channels.Do t-tubules play a role in arrhythmogenesis in cardiac ventricular myocytes?Emerging roles of junctophilin-2 in the heart and implications for cardiac diseases.Mechanisms underlying the impaired contractility of diabetic cardiomyopathy.Calcium in the heart: from physiology to disease.Hold or fold--proteins in advanced heart failure and myocardial recovery.Manipulation of sarcoplasmic reticulum Ca(2+) release in heart failure through mechanical intervention.RNA-sequencing analysis reveals new alterations in cardiomyocyte cytoskeletal genes in patients with heart failure.MG53 is dispensable for T-tubule maturation but critical for maintaining T-tubule integrity following cardiac stress.

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P2860

Changes in the organization of excitation-contraction coupling structures in failing human heart.

http://www.wikidata.org/entity/Q33847166

description

2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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David J Crossman

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P2860

Spatial non-uniformities in [Ca2+]i during excitation-contraction coupling in cardiac myocytes.

Quantitative colocalization analysis of multicolor confocal immunofluorescence microscopy images: pushing pixels to explore biological phenomena

Cardiac excitation-contraction coupling

Membrane systems of guinea pig myocardium: ultrastructure and morphometric studies.

Analysis of ryanodine receptor clusters in rat and human cardiac myocytes

Reduction in density of transverse tubules and L-type Ca(2+) channels in canine tachycardia-induced heart failure.

Loss of T-tubules and other changes to surface topography in ventricular myocytes from failing human and rat heart.

Two-photon laser scanning microscopy of the transverse-axial tubule system in ventricular cardiomyocytes from failing and non-failing human hearts

Increased expression of cytoskeletal, linkage, and extracellular proteins in failing human myocardium.

Orphaned ryanodine receptors in the failing heart.

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