A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming. - Wikidata - wikimedia - TriplyDB

A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming.

A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming.

http://www.wikidata.org/entity/Q33858072

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Psychological and educational interventions for atopic eczema in children Complementary and alternative medicine treatments for atopic eczema Loss of corneodesmosin leads to severe skin barrier defect, pruritus, and atopy: unraveling the peeling skin disease Mutations in the filaggrin gene and food allergy Epidermal permeability barrier defects and barrier repair therapy in atopic dermatitis Filaggrin deficiency promotes the dissemination of cutaneously inoculated vaccinia virus Filaggrin failure - from ichthyosis vulgaris to atopic eczema and beyond Filaggrin in the frontline: role in skin barrier function and disease The human epidermal differentiation complex: cornified envelope precursors, S100 proteins and the 'fused genes' family Tmem79/Matt is the matted mouse gene and is a predisposing gene for atopic dermatitis in human subjects Increased retinoic acid levels through ablation of Cyp26b1 determine the processes of embryonic skin barrier formation and peridermal development Gene loss in keratinization programs accompanies adaptation of cetacean skin to aquatic lifestyle Re-appraisal of current theories for the development and loss of epidermal pigmentation in hominins and modern humans Skin barrier defects in atopic dermatitis Flow cytometry imaging identifies rare T(H)2 cells expressing thymic stromal lymphopoietin receptor in a "proallergic" milieu.Filaggrin deficiency confers a paracellular barrier abnormality that reduces inflammatory thresholds to irritants and haptens Epidermal barrier defects link atopic dermatitis with altered skin cancer susceptibility Mast cells in atopic dermatitis Thymic stromal lymphopoietin and the pathophysiology of atopic disease.Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract.Filaggrin-deficient mice exhibit TH17-dominated skin inflammation and permissiveness to epicutaneous sensitization with protein antigen Effectiveness and cost-effectiveness of daily all-over-body application of emollient during the first year of life for preventing atopic eczema in high-risk children (The BEEP trial): protocol for a randomised controlled trial.Genome-wide association analysis of eosinophilic esophagitis provides insight into the tissue specificity of this allergic disease.The infectious aspects of atopic dermatitis.Allergic asthma: influence of genetic and environmental factors.Emollient enhancement of the skin barrier from birth offers effective atopic dermatitis prevention Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence Peanut allergy: effect of environmental peanut exposure in children with filaggrin loss-of-function mutations.New insights into the epidemiology of childhood atopic dermatitis.The Atopic March: Progression from Atopic Dermatitis to Allergic Rhinitis and Asthma.The allergy gene: how a mutation in a skin protein revealed a link between eczema and asthma.Tight junction defects in patients with atopic dermatitis.Exacerbation of allergen-induced eczema in TLR4- and TRIF-deficient mice.Atopic dermatitis results in intrinsic barrier and immune abnormalities: implications for contact dermatitis.Atopic dermatitis increases the effect of exposure to peanut antigen in dust on peanut sensitization and likely peanut allergy.Loss-of-function variants in the filaggrin gene are a significant risk factor for peanut allergy.Filaggrin genotype in ichthyosis vulgaris predicts abnormalities in epidermal structure and function Atopic dermatitis: a disease of altered skin barrier and immune dysregulation.Genetic dissection of eosinophilic esophagitis provides insight into disease pathogenesis and treatment strategies Matriptase deletion initiates a Sjögren's syndrome-like disease in mice.

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P2860

A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming.

http://www.wikidata.org/entity/Q33858072

description

2009 nî lūn-bûn

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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2009 թվականի ապրիլին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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A homozygous frameshift mutati ...... percutaneous allergen priming.

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A homozygous frameshift mutati ...... percutaneous allergen priming.

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A homozygous frameshift mutati ...... percutaneous allergen priming.

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A homozygous frameshift mutati ...... percutaneous allergen priming.

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A homozygous frameshift mutati ...... percutaneous allergen priming.

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Aiko Shiohama

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Aileen Sandilands

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Hiroshi Kawasaki

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John P Sundberg

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Jun Kudoh

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Linda E Campbell

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Nobuyoshi Shimizu

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Philip Fleckman

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Takashi Sasaki

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P2860

The burden of disease associated with filaggrin mutations: a population-based, longitudinal birth cohort study

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

Functional analysis of the profilaggrin N-terminal peptide: identification of domains that regulate nuclear and cytoplasmic distribution

Toward a major risk factor for atopic eczema: meta-analysis of filaggrin polymorphism data.

Roles of TH1 and TH2 cytokines in a murine model of allergic dermatitis.

The structure of the gene for mouse filaggrin and a comparison of the repeating units.

Loss-of-function mutations in the gene encoding filaggrin cause ichthyosis vulgaris.

How epidemiology has challenged 3 prevailing concepts about atopic dermatitis.

Contribution of IL-18 to atopic-dermatitis-like skin inflammation induced by Staphylococcus aureus product in mice

Atopic dermatitis and the atopic march.

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602-608

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10.1038/NG.358

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5

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Masayuki Amagai

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John J Callanan

Niamh E. Mangan

Sean P. Saunders

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2009-04-06T00:00:00Z

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19349982

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