A minority of foci or pan-nuclear apoptotic staining of gammaH2AX in the S phase after UV damage contain DNA double-strand breaks. - Wikidata - wikimedia - TriplyDB

A minority of foci or pan-nuclear apoptotic staining of gammaH2AX in the S phase after UV damage contain DNA double-strand breaks.

A minority of foci or pan-nuclear apoptotic staining of gammaH2AX in the S phase after UV damage contain DNA double-strand breaks.

http://www.wikidata.org/entity/Q33859669

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Rho GTPases: Novel Players in the Regulation of the DNA Damage Response?Establishment of a γ-H2AX foci-based assay to determine biological dose of radon to red bone marrow in rats 4-Hydroxynonenal induces G2/M phase cell cycle arrest by activation of the ataxia telangiectasia mutated and Rad3-related protein (ATR)/checkpoint kinase 1 (Chk1) signaling pathway Telmisartan induces growth inhibition, DNA double-strand breaks and apoptosis in human endometrial cancer cells Aberrant DNA damage response pathways may predict the outcome of platinum chemotherapy in ovarian cancer E2F7 represses a network of oscillating cell cycle genes to control S-phase progression Nuclear accumulation of the papillomavirus E1 helicase blocks S-phase progression and triggers an ATM-dependent DNA damage response The role of ROS and subsequent DNA-damage response in PUMA-induced apoptosis of ovarian cancer cells.G1 checkpoint establishment in vivo during embryonic liver development.The nuclear γ-H2AX apoptotic ring: implications for cancers and autoimmune diseases.Nitric oxide induces ataxia telangiectasia mutated (ATM) protein-dependent γH2AX protein formation in pancreatic β cells.The Drosophila Werner exonuclease participates in an exonuclease-independent response to replication stress.DNA damage signaling regulates age-dependent proliferative capacity of quiescent inner ear supporting cells Inhibition of histone deacetylases enhances DNA damage repair in SCNT embryos.Epidermal p65/NF-κB signalling is essential for skin carcinogenesis Ciprofloxacin as a potential radio-sensitizer to tumor cells and a radio-protectant for normal cells: differential effects on γ-H2AX formation, p53 phosphorylation, Bcl-2 production, and cell death Mitochondrial reactive oxygen species are scavenged by Cockayne syndrome B protein in human fibroblasts without nuclear DNA damage UV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cells MDC1 cleavage by caspase-3: a novel mechanism for inactivating the DNA damage response during apoptosis.Comparative transcriptome profiling of an SV40-transformed human fibroblast (MRC5CVI) and its untransformed counterpart (MRC-5) in response to UVB irradiation.Functional relevance of the histone gammaH2Ax in the response to DNA damaging agents.The NR4A2 nuclear receptor is recruited to novel nuclear foci in response to UV irradiation and participates in nucleotide excision repair The rem mutations in the ATP-binding groove of the Rad3/XPD helicase lead to Xeroderma pigmentosum-Cockayne syndrome-like phenotypes.Excision of translesion synthesis errors orchestrates responses to helix-distorting DNA lesions.DNA double-strand breaks induced by high NaCl occur predominantly in gene deserts Distinct kinetics of DNA repair protein accumulation at DNA lesions and cell cycle-dependent formation of γH2AX- and NBS1-positive repair foci.Streptococcus pneumoniae secretes hydrogen peroxide leading to DNA damage and apoptosis in lung cells.Deficiency in mammalian histone H2B ubiquitin ligase Bre1 (Rnf20/Rnf40) leads to replication stress and chromosomal instability.Low Dose Iron Treatments Induce a DNA Damage Response in Human Endothelial Cells within Minutes EZH2 suppresses the nucleotide excision repair in nasopharyngeal carcinoma by silencing XPA gene.DNA polymerase zeta is required for proliferation of normal mammalian cells.Triplex structures induce DNA double strand breaks via replication fork collapse in NER deficient cells.DNA damage induces a meiotic arrest in mouse oocytes mediated by the spindle assembly checkpoint.Linking hypoxia, DNA damage and proliferation in multicellular tumor spheroids Cellular Recognition and Repair of Monofunctional-Intercalative Platinum--DNA Adducts Oncogenes induce genotoxic stress by mitotic processing of unusual replication intermediates.The shape of the radiation dose response for DNA double-strand break induction and repair.In vitro and in vivo radiosensitization induced by hydroxyapatite nanoparticles.Clustered DNA damage induces pan-nuclear H2AX phosphorylation mediated by ATM and DNA-PK Effect of gadolinium-based nanoparticles on nuclear DNA damage and repair in glioblastoma tumor cells

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A minority of foci or pan-nuclear apoptotic staining of gammaH2AX in the S phase after UV damage contain DNA double-strand breaks.

http://www.wikidata.org/entity/Q33859669

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

@ast

A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

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type

label

A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

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A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

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prefLabel

A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

@ast

A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

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Proceedings of the National Academy of Sciences of the United States of America

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A minority of foci or pan-nucl ...... tain DNA double-strand breaks.

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Ingrid Revet

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James E Cleaver

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Luzviminda Feeney

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Sebastien de Feraudy

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Vladimir Bezrookove

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P2860

hRAD30 mutations in the variant form of xeroderma pigmentosum

MDC1 is a mediator of the mammalian DNA damage checkpoint

ATM activation by DNA double-strand breaks through the Mre11-Rad50-Nbs1 complex

UV-induced replication arrest in the xeroderma pigmentosum variant leads to DNA double-strand breaks, gamma -H2AX formation, and Mre11 relocalization

Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses

Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes

The XPV (xeroderma pigmentosum variant) gene encodes human DNA polymerase eta

ATM phosphorylates histone H2AX in response to DNA double-strand breaks

Accumulation of checkpoint protein 53BP1 at DNA breaks involves its binding to phosphorylated histone H2AX

Histone H2AX is phosphorylated in an ATR-dependent manner in response to replicational stress

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107

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42639183

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20351298

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apoptotic process

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2872460

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