Infection-stimulated fibrin deposition controls hemorrhage and limits hepatic bacterial growth during listeriosis - Wikidata - wikimedia - TriplyDB

Infection-stimulated fibrin deposition controls hemorrhage and limits hepatic bacterial growth during listeriosis

Infection-stimulated fibrin deposition controls hemorrhage and limits hepatic bacterial growth during listeriosis

http://www.wikidata.org/entity/Q33883280

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The omptins of Yersinia pestis and Salmonella enterica cleave the reactive center loop of plasminogen activator inhibitor 1 Activation of mannan-binding lectin-associated serine proteases leads to generation of a fibrin clot Causes and consequences of coagulation activation in sepsis: an evolutionary medicine perspective Crossroads of coagulation and innate immunity: the case of deep vein thrombosis Simultaneous activation of complement and coagulation by MBL-associated serine protease 2 Factor XI-deficient mice display reduced inflammation, coagulopathy, and bacterial growth during listeriosis Gammaherpesvirus latency induces antibody-associated thrombocytopenia in mice.Fibrinogen regulates the cytotoxicity of mycobacterial trehalose dimycolate but is not required for cell recruitment, cytokine response, or control of mycobacterial infection.Dengue-2 structural proteins associate with human proteins to produce a coagulation and innate immune response biased interactome.Tumor necrosis factor alpha and gamma interferon, but not hemorrhage or pathogen burden, dictate levels of protective fibrin deposition during infection Mycobacterium tuberculosis infection and tissue factor expression in macrophages.Role of tissue factor in Mycobacterium tuberculosis-induced inflammation and disease pathogenesis.CYLD enhances severe listeriosis by impairing IL-6/STAT3-dependent fibrin production Factor XIII Transglutaminase Supports the Resolution of Mucosal Damage in Experimental Colitis.Inhibition of factor XI activation attenuates inflammation and coagulopathy while improving the survival of mouse polymicrobial sepsis Mice expressing a mutant form of fibrinogen that cannot support fibrin formation exhibit compromised antimicrobial host defense.Genetic elimination of the binding motif on fibrinogen for the S. aureus virulence factor ClfA improves host survival in septicemia.Spatial localization of bacteria controls coagulation of human blood by 'quorum acting'Neither fibrin nor plasminogen activator inhibitor-1 deficiency protects lung function in a mouse model of acute lung injury Systemic but not local infections elicit immunosuppressive IL-10 production by natural killer cells.Bacteria under stress by complement and coagulation.Fibrinogen Is at the Interface of Host Defense and Pathogen Virulence in Staphylococcus aureus Infection.Innate immunity, hemostasis and matrix remodeling: PTX3 as a link.Fibrin facilitates both innate and T cell-mediated defense against Yersinia pestis.Inflammation and the coagulation system in tuberculosis: Tissue Factor leads the dance.Protective roles for fibrin, tissue factor, plasminogen activator inhibitor-1, and thrombin activatable fibrinolysis inhibitor, but not factor XI, during defense against the gram-negative bacterium Yersinia enterocolitica.In situ assays demonstrate that interferon-gamma suppresses infection-stimulated hepatic fibrin deposition by promoting fibrinolysis.Interleukin-1 promotes coagulation, which is necessary for protective immunity in the lung against Streptococcus pneumoniae infection.Distinct behavior of myelomonocytic cells and CD8 T cells underlies the hepatic response to Listeria monocytogenes.

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P2860

Infection-stimulated fibrin deposition controls hemorrhage and limits hepatic bacterial growth during listeriosis

http://www.wikidata.org/entity/Q33883280

description

2005 nî lūn-bûn

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2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2005 թվականի հուլիսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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Infection-stimulated fibrin de ...... rial growth during listeriosis

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Infection-stimulated fibrin de ...... rial growth during listeriosis

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IAI.73.7.3888-3895.2005

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Infection and Immunity

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Infection-stimulated fibrin de ...... rial growth during listeriosis

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Frank M Szaba

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Isis K Mullarky

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Kiera N Berggren

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Lawrence L Johnson

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Lawrence W Kummer

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Michelle A Parent

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Stephen T Smiley

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Wangxue Chen

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P2860

Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

Universal changes in biomarkers of coagulation and inflammation occur in patients with severe sepsis, regardless of causative micro-organism [ISRCTN74215569]

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

Efficacy and safety of recombinant human activated protein C for severe sepsis

Leukocyte engagement of fibrin(ogen) via the integrin receptor alphaMbeta2/Mac-1 is critical for host inflammatory response in vivo

The plasminogen activation system enhances brain and heart invasion in murine relapsing fever borreliosis.

Surface protein adhesins of Staphylococcus aureus.

Neutrophils are involved in acute, nonspecific resistance to Listeria monocytogenes in mice.

Plasminogen is required for efficient dissemination of B. burgdorferi in ticks and for enhancement of spirochetemia in mice.

Reticulocyte count using thiazole orange. A flow cytometry method.

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