GATA6 levels modulate primitive endoderm cell fate choice and timing in the mouse blastocyst.
about
From blastocyst to gastrula: gene regulatory networks of embryonic stem cells and early mouse embryogenesisHeterogeneities in Nanog Expression Drive Stable Commitment to Pluripotency in the Mouse BlastocystGenetically engineering self-organization of human pluripotent stem cells into a liver bud-like tissue using Gata6.Single cells get together: High-resolution approaches to study the dynamics of early mouse developmentPrimate embryogenesis predicts the hallmarks of human naïve pluripotency.Four simple rules that are sufficient to generate the mammalian blastocystHIPPO pathway members restrict SOX2 to the inner cell mass where it promotes ICM fates in the mouse blastocyst.Epigenetic regulation of histone modifications and Gata6 gene expression induced by maternal diet in mouse embryoid bodies in a model of developmental programming.A mRNA landscape of bovine embryos after standard and MAPK-inhibited culture conditions: a comparative analysis.Sox7 is dispensable for primitive endoderm differentiation from mouse ES cellsCell signaling and transcription factors regulating cell fate during formation of the mouse blastocyst.A loss-of-function and H2B-Venus transcriptional reporter allele for Gata6 in miceGata6 potently initiates reprograming of pluripotent and differentiated cells to extraembryonic endoderm stem cells.Quantitative Analysis of Protein Expression to Study Lineage Specification in Mouse Preimplantation Embryos.Venus trap in the mouse embryo reveals distinct molecular dynamics underlying specification of first embryonic lineages.Lineage-Specific Profiling Delineates the Emergence and Progression of Naive Pluripotency in Mammalian EmbryogenesisCell Fate Specification Based on Tristability in the Inner Cell Mass of Mouse Blastocysts.Asynchronous fate decisions by single cells collectively ensure consistent lineage composition in the mouse blastocyst.PDGFRα+ Cells in Embryonic Stem Cell Cultures Represent the In Vitro Equivalent of the Pre-implantation Primitive Endoderm Precursors.Primitive endoderm differentiation: from specification to epithelium formation.Cell fate in animal and human blastocysts and the determination of viability.The mammalian blastocyst.Lineage Establishment and Progression within the Inner Cell Mass of the Mouse Blastocyst Requires FGFR1 and FGFR2.Signaling pathways in mammalian preimplantation development: Linking cellular phenotypes to lineage decisions.p38 (Mapk14/11) occupies a regulatory node governing entry into primitive endoderm differentiation during preimplantation mouse embryo development.Dedifferentiation into blastomere-like cancer stem cells via formation of polyploid giant cancer cellsThe chromatin modifier Satb1 regulates cell fate through Fgf signalling in the early mouse embryoA multiscale model of early cell lineage specification including cell divisionICM conversion to epiblast by FGF/ERK inhibition is limited in time and requires transcription and protein degradation.FGF/MAPK signaling sets the switching threshold of a bistable circuit controlling cell fate decisions in embryonic stem cells.Plasticity of the inner cell mass in mouse blastocyst is restricted by the activity of FGF/MAPK pathway.Self-organization of the in vitro attached human embryo.Suppression of ERK signalling abolishes primitive endoderm formation but does not promote pluripotency in rabbit embryo.Klf5 maintains the balance of primitive endoderm versus epiblast specification during mouse embryonic development by suppression of Fgf4.The Role of Maternal Nutrition During the Periconceptional Period and Its Effect on Offspring Phenotype.Integrated analysis of single-cell embryo data yields a unified transcriptome signature for the human preimplantation epiblast.Pten facilitates epiblast epithelial polarization and proamniotic lumen formation in early mouse embryos.Argonaute 2 Is Required for Extra-embryonic Endoderm Differentiation of Mouse Embryonic Stem Cells.CHD1 Controls Cell Lineage Specification Through Zygotic Genome Activation.GATA6 phosphorylation by Erk1/2 propels exit from pluripotency and commitment to primitive endoderm.
P2860
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P2860
GATA6 levels modulate primitive endoderm cell fate choice and timing in the mouse blastocyst.
description
2014 nî lūn-bûn
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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
GATA6 levels modulate primitiv ...... iming in the mouse blastocyst.
@ast
GATA6 levels modulate primitiv ...... iming in the mouse blastocyst.
@en
type
label
GATA6 levels modulate primitiv ...... iming in the mouse blastocyst.
@ast
GATA6 levels modulate primitiv ...... iming in the mouse blastocyst.
@en
prefLabel
GATA6 levels modulate primitiv ...... iming in the mouse blastocyst.
@ast
GATA6 levels modulate primitiv ...... iming in the mouse blastocyst.
@en
P2093
P2860
P50
P1433
P1476
GATA6 levels modulate primitiv ...... timing in the mouse blastocyst
@en
P2093
Anna-Katerina Hadjantonakis
Nadine Schrode
Néstor Saiz
Stefano Di Talia
P2860
P304
P356
10.1016/J.DEVCEL.2014.04.011
P407
P577
2014-05-15T00:00:00Z