Ionizing radiation-induced long-term expression of senescence markers in mice is independent of p53 and immune status. - Wikidata - wikimedia - TriplyDB

Ionizing radiation-induced long-term expression of senescence markers in mice is independent of p53 and immune status.

Ionizing radiation-induced long-term expression of senescence markers in mice is independent of p53 and immune status.

http://www.wikidata.org/entity/Q33949558

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Current Evidence for Developmental, Structural, and Functional Brain Defects following Prenatal Radiation Exposure Therapeutic Implications for Overcoming Radiation Resistance in Cancer Therapy Preventive or potential therapeutic value of nutraceuticals against ionizing radiation-induced oxidative stress in exposed subjects and frequent fliers Ionizing radiation reduces ADAM10 expression in brain microvascular endothelial cells undergoing stress-induced senescence.Senescent cells: a novel therapeutic target for aging and age-related diseases DNA-SCARS: distinct nuclear structures that sustain damage-induced senescence growth arrest and inflammatory cytokine secretion Four faces of cellular senescence.The role of senescent cells in ageing.The Achilles' heel of senescent cells: from transcriptome to senolytic drugs.Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL Cell senescence abrogates the therapeutic potential of human mesenchymal stem cells in the lethal endotoxemia model.Clinical strategies and animal models for developing senolytic agents.An essential role for senescent cells in optimal wound healing through secretion of PDGF-AA Progerin, the protein responsible for the Hutchinson-Gilford progeria syndrome, increases the unrepaired DNA damages following exposure to ionizing radiation.Age-associated increase in heterochromatic marks in murine and primate tissues.Lamin B1 loss is a senescence-associated biomarker.Inhibition of p38 MAPK attenuates ionizing radiation-induced hematopoietic cell senescence and residual bone marrow injury Prolonged expression of senescence markers in mice exposed to gamma-irradiation.Radiation-induced senescence in securin-deficient cancer cells promotes cell invasion involving the IL-6/STAT3 and PDGF-BB/PDGFR pathways p53-dependent release of Alarmin HMGB1 is a central mediator of senescent phenotypes Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation Exacerbation of lung radiation injury by viral infection: the role of Clara cells and Clara cell secretory protein.Mitochondrial DNA damage induces apoptosis in senescent cells The DNA damage response induces inflammation and senescence by inhibiting autophagy of GATA4.Ionizing radiation-mediated premature senescence and paracrine interactions with cancer cells enhance the expression of syndecan 1 in human breast stromal fibroblasts: the role of TGF-β.Expression of the senescence marker p16INK4a in skin biopsies of acute lymphoblastic leukemia survivors: a pilot study.Cellular Senescence - its role in cancer and the response to ionizing radiation.Opportunities and challenges of radiotherapy for treating cancer.Modeling DNA damage-induced pneumopathy in mice: insight from danger signaling cascades.Murine mesenchymal cells that express elevated levels of the CDK inhibitor p16(Ink4a) in vivo are not necessarily senescent.Cellular senescence in renal ageing and disease.Balancing Between Aging and Cancer: Molecular Genetics Meets Traditional Chinese Medicine.Senescence in Health and Disease.Senescent cells: an emerging target for diseases of ageing.How to measure RNA expression in rare senescent cells expressing any specific protein such as p16Ink4a.Innate immune sensing of cytosolic chromatin fragments through cGAS promotes senescence.p16(Ink4a) and senescence-associated β-galactosidase can be induced in macrophages as part of a reversible response to physiological stimuli.Sustained p16INK4a expression is required to prevent IR-induced tumorigenesis in mice Synergestic effect of aqueous purslane (Portulaca oleracea L.) extract and fish oil on radiation-induced damage in rats.

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Ionizing radiation-induced long-term expression of senescence markers in mice is independent of p53 and immune status.

http://www.wikidata.org/entity/Q33949558

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年论文

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Ionizing radiation-induced lon ...... dent of p53 and immune status.

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Ionizing radiation-induced lon ...... dent of p53 and immune status.

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Ionizing radiation-induced lon ...... dent of p53 and immune status.

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Ionizing radiation-induced lon ...... dent of p53 and immune status.

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Caroline Laverdière

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Christian M Beauséjour

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Elie Haddad

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Francis Rodier

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Francois Fontaine

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James DeGregori

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Jean-Philippe Coppe

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Judith Campisi

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Oanh N L Le

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Victor Kokta

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P2860

The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor

Pharmacological manipulation of cell death: clinical applications in sight?

Senescence of activated stellate cells limits liver fibrosis

Expression of p16(INK4a) in peripheral blood T-cells is a biomarker of human aging

Two faces of p53: aging and tumor suppression

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

Aging and genome maintenance: lessons from the mouse?

p16INK4a induces an age-dependent decline in islet regenerative potential

Accumulation of senescent cells in mitotic tissue of aging primates

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