BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks - Wikidata - wikimedia - TriplyDB

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

http://www.wikidata.org/entity/Q33986975

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Connecting the Dots: From DNA Damage and Repair to Aging Noncanonical views of homology-directed DNA repair RFWD3-Dependent Ubiquitination of RPA Regulates Repair at Stalled Replication Forks.Induction of homologous recombination between sequence repeats by the activation induced cytidine deaminase (AID) protein Attenuation of RNA polymerase II pausing mitigates BRCA1-associated R-loop accumulation and tumorigenesis.Clinical Application of Poly(ADP-Ribose) Polymerase Inhibitors in High-Grade Serous Ovarian Cancer.BRCA1 haploinsufficiency for replication stress suppression in primary cells Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.DNA interstrand cross-link repair requires replication-fork convergence.Influence of aflatoxin B1 on copy number variants in human leukocytes in vitro End of the beginning: elongation and termination features of alternative modes of chromosomal replication initiation in bacteria Deciphering the BRCA1 Tumor Suppressor Network.The progression of replication forks at natural replication barriers in live bacteria Replication fork stability confers chemoresistance in BRCA-deficient cells Complex Breakpoints and Template Switching Associated with Non-canonical Termination of Homologous Recombination in Mammalian Cells.DEK is required for homologous recombination repair of DNA breaks.Clusters of Multiple Mutations: Incidence and Molecular Mechanisms "DNA Binding Region" of BRCA1 Affects Genetic Stability through modulating the Intra-S-Phase Checkpoint MRN, CtIP, and BRCA1 mediate repair of topoisomerase II-DNA adducts.Stop relaxing: How DNA damage-induced chromatin compaction may affect epigenetic integrity and disease.FANCD2 Maintains Fork Stability in BRCA1/2-Deficient Tumors and Promotes Alternative End-Joining DNA Repair.Spatial separation of replisome arrest sites influences homologous recombination quality at a Tus/Ter-mediated replication fork barrier.RECQ5 helicase promotes resolution of conflicts between replication and transcription in human cells Tus-Ter as a tool to study site-specific DNA replication perturbation in eukaryotes.Chromosomal Rearrangements in Cancer: Detection and potential causal mechanisms.Stalled replication forks generate a distinct mutational signature in yeast.Deciphering the Code of the Cancer Genome: Mechanisms of Chromosome Rearrangement.Pancreatic cancer biology and genetics from an evolutionary perspective.Remarkably Long-Tract Gene Conversion Induced by Fragile Site Instability in Saccharomyces cerevisiae.BRCA2 suppresses replication stress-induced mitotic and G1 abnormalities through homologous recombination.FANCJ helicase controls the balance between short- and long-tract gene conversions between sister chromatids.Acute inactivation of the replicative helicase in human cells triggers MCM8-9-dependent DNA synthesis.RADX Promotes Genome Stability and Modulates Chemosensitivity by Regulating RAD51 at Replication Forks.Analysis of DNA topology of EBV minichromosomes in HEK 293 cells.Mechanism of tandem duplication formation in BRCA1-mutant cells.A Molecular Toolbox to Engineer Site-Specific DNA Replication Perturbation.Replication Stress Shapes a Protective Chromatin Environment across Fragile Genomic Regions.Complex repeat structure promotes hyper-amplification and amplicon evolution through rolling-circle replication.Poly(ADP-ribose) polymerase-3 overexpression is associated with poor prognosis in patients with breast cancer following chemotherapy Homology-Directed Repair and the Role of BRCA1, BRCA2, and Related Proteins in Genome Integrity and Cancer

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BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

http://www.wikidata.org/entity/Q33986975

description

2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@ast

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@en

type

label

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@ast

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@en

prefLabel

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@ast

BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@en

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BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks

@en

P2093

Amy Kwok

http://www.w3.org/2001/XMLSchema#string

Bin Huang

http://www.w3.org/2001/XMLSchema#string

Chuxia Deng

http://www.w3.org/2001/XMLSchema#string

Cindy Follonier

http://www.w3.org/2001/XMLSchema#string

Gurushankar Chandramouly

http://www.w3.org/2001/XMLSchema#string

Nicholas A Willis

http://www.w3.org/2001/XMLSchema#string

Ralph Scully

http://www.w3.org/2001/XMLSchema#string

P2860

Human GEN1 and the SLX4-associated nucleases MUS81 and SLX1 are essential for the resolution of replication-induced Holliday junctions

Stabilization of stalled DNA replication forks by the BRCA2 breast cancer susceptibility protein

The DNA Damage Response: Making It Safe to Play with Knives

Migrating bubble during break-induced replication drives conservative DNA synthesis.

Initiation of genetic recombination and recombination-dependent replication

Replication from oriP of Epstein-Barr virus requires human ORC and is inhibited by geminin

Minding the gap: the underground functions of BRCA1 and BRCA2 at stalled replication forks

In search of the tumour-suppressor functions of BRCA1 and BRCA2

The importance of repairing stalled replication forks

A microhomology-mediated break-induced replication model for the origin of human copy number variation.

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556-559

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scholarly article

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10.1038/NATURE13295

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P433

7506

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510

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2014-04-28T00:00:00Z

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261955081

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1048623420

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24776801

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4118467

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