FLIP switches Fas-mediated glucose signaling in human pancreatic beta cells from apoptosis to cell replication. - Wikidata - wikimedia - TriplyDB

FLIP switches Fas-mediated glucose signaling in human pancreatic beta cells from apoptosis to cell replication.

FLIP switches Fas-mediated glucose signaling in human pancreatic beta cells from apoptosis to cell replication.

http://www.wikidata.org/entity/Q34032136

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Interleukin-6 regulates pancreatic alpha-cell mass expansion What is the impact of n-3 PUFAs on inflammation markers in Type 2 diabetic mellitus populations?: a systematic review and meta-analysis of randomized controlled trials The required beta cell research for improving treatment of type 2 diabetes FLIP and the death effector domain family Fas Ag-FasL coupling leads to ERK1/2-mediated proliferation of gastric mucosal cells A 3D map of the islet routes throughout the healthy human pancreas.An islet in distress: β cell failure in type 2 diabetes.c-FLIPL enhances anti-apoptotic Akt functions by modulation of Gsk3β activity.Leptin modulates beta cell expression of IL-1 receptor antagonist and release of IL-1beta in human islets Interleukin-1 as a common denominator from autoinflammatory to autoimmune disorders: premises, perils, and perspectives Fas-mediated apoptosis in cholangiocarcinoma cells is enhanced by 3,3'-diindolylmethane through inhibition of AKT signaling and FLICE-like inhibitory protein.Decreased beta-cell mass in diabetes: significance, mechanisms and therapeutic implications.The Fas pathway is involved in pancreatic beta cell secretory function.c-FLIP maintains tissue homeostasis by preventing apoptosis and programmed necrosis Beta-cells in type 2 diabetes: a loss of function and mass.Nodal induces apoptosis through activation of the ALK7 signaling pathway in pancreatic INS-1 β-cells.IFN-γ-mediated hematopoietic cell destruction in murine models of immune-mediated bone marrow failure.Dual modulation of Ras-Mnk and PI3K-AKT-mTOR pathways: A Novel c-FLIP inhibitory mechanism of 3-AWA mediated translational attenuation through dephosphorylation of eIF4E.On the role of IRS2 in the regulation of functional beta-cell mass.Beta cells in type 2 diabetes - a crucial contribution to pathogenesis.The vicious cycle of apoptotic beta-cell death in type 1 diabetes.The role of beta-cell dysfunction in the cardiometabolic syndrome.TOSO promotes β-cell proliferation and protects from apoptosis.The antiinflammatory cytokine interleukin-1 receptor antagonist protects from high-fat diet-induced hyperglycemia.Histopathology of type 1 diabetes: old paradigms and new insights.Cellular FLIP inhibits beta-catenin ubiquitylation and enhances Wnt signaling.Glucose-induced β cell production of IL-1β contributes to glucotoxicity in human pancreatic islets.Genetics of new-onset diabetes after transplantation.β-Cell dysfunction in chronic pancreatitis.Type 2 diabetes mellitus: From a metabolic disorder to an inflammatory condition.Interorgan Crosstalk Contributing to β-Cell Dysfunction.Inhibitory effect of unsaturated fatty acids on saturated fatty acid-induced apoptosis in human pancreatic β-cells: activation of caspases and ER stress induction.Lipofection of insulin-producing RINm5F cells: methodological improvements.Indoleamine-2,3-Dioxygenase/Kynurenine Pathway as a Potential Pharmacological Target to Treat Depression Associated with Diabetes.Hyperglycemia induces apoptosis of human pancreatic islet endothelial cells: effects of pravastatin on the Akt survival pathway.Overexpression of E2F3 promotes proliferation of functional human β cells without induction of apoptosis.Fas/Fas-Ligand Interaction As a Mechanism of Immune Homeostasis and β-Cell Cytotoxicity: Enforcement Rather Than Neutralization for Treatment of Type 1 Diabetes.Caspase-3-dependent beta-cell apoptosis in the initiation of autoimmune diabetes mellitus.Protection of human pancreatic islets using a lentiviral vector expressing two genes: cFLIP and GFP.Transgenic overexpression of the Caspase-8 inhibitor FLIP(short) leads to impaired T cell proliferation and an increased memory T cell pool after staphylococcal enterotoxin B injection.

P2860

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P2860

FLIP switches Fas-mediated glucose signaling in human pancreatic beta cells from apoptosis to cell replication.

http://www.wikidata.org/entity/Q34032136

description

2002 nî lūn-bûn

@nan

2002 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2002年の論文

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2002年論文

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2002年論文

@zh-hant

2002年論文

@zh-hk

2002年論文

@zh-mo

2002年論文

@zh-tw

2002年论文

@wuu

name

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@ast

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@en

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@nl

type

label

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@ast

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@en

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

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prefLabel

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@ast

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@en

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

FLIP switches Fas-mediated glu ...... apoptosis to cell replication.

@en

P2093

Adriano Fontana

http://www.w3.org/2001/XMLSchema#string

Andrea Tasinato

http://www.w3.org/2001/XMLSchema#string

Christian Toso

http://www.w3.org/2001/XMLSchema#string

Felix Bachmann

http://www.w3.org/2001/XMLSchema#string

Frédéric Ris

http://www.w3.org/2001/XMLSchema#string

Giatgen A Spinas

http://www.w3.org/2001/XMLSchema#string

José Oberholzer

http://www.w3.org/2001/XMLSchema#string

Kathrin Maedler

http://www.w3.org/2001/XMLSchema#string

Marc Y Donath

http://www.w3.org/2001/XMLSchema#string

Pavel Sergeev

http://www.w3.org/2001/XMLSchema#string

P2860

Inhibition of death receptor signals by cellular FLIP

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways

High glucose causes apoptosis in cultured human pancreatic islets of Langerhans: a potential role for regulation of specific Bcl family genes toward an apoptotic cell death program

Islet growth and development in the adult.

Insulin resistance is not necessarily an essential component of type 2 diabetes.

Interactions between insulin resistance and insulin secretion in the development of glucose intolerance.

Proliferation and differentiation in the human fetal endocrine pancreas.

Expression of cellular FLICE-inhibitory protein in human coronary arteries and in a rat vascular injury model.

Glucose and tolbutamide induce apoptosis in pancreatic beta-cells. A process dependent on intracellular Ca2+ concentration.

P304

8236-8241

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scholarly article

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10.1073/PNAS.122686299

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P407

P433

12

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P478

99

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P577

2002-06-01T00:00:00Z

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11313159

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12060768

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123051

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