The oxidation states of DJ-1 dictate the cell fate in response to oxidative stress triggered by 4-hpr: autophagy or apoptosis?
about
Oxidative stress induces autophagy in response to multiple noxious stimuli in retinal ganglion cellsUse of cysteine-reactive cross-linkers to probe conformational flexibility of human DJ-1 demonstrates that Glu18 mutations are dimersEuropean contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Oxidant-induced Interprotein Disulfide Formation in Cardiac Protein DJ-1 Occurs via an Interaction with Peroxiredoxin 2.Autophagy in Diabetic Retinopathy.β-Ecdysterone Protects SH-SY5Y Cells Against 6-Hydroxydopamine-Induced Apoptosis via Mitochondria-Dependent Mechanism: Involvement of p38(MAPK)-p53 Signaling Pathway.Tanshinone IIA Protects Hippocampal Neuronal Cells from Reactive Oxygen Species Through Changes in Autophagy and Activation of Phosphatidylinositol 3-Kinase, Protein Kinas B, and Mechanistic Target of Rapamycin Pathways.Cytoprotective mechanisms of DJ-1 against oxidative stress through modulating ERK1/2 and ASK1 signal transduction.Cardioprotective effects of Cu(II)ATSM in human vascular smooth muscle cells and cardiomyocytes mediated by Nrf2 and DJ-1.Hypoxia inducible factor-1α regulates autophagy via the p27-E2F1 signaling pathway.An Isoquinolin-1(2H)-Imine Derivative Induces Cell Death via Generation of Reactive Oxygen Species and Activation of JNK in Human A549 Cancer Cells.Metal chelator TPEN selectively induces apoptosis in K562 cells through reactive oxygen species signaling mechanism: implications for chronic myeloid leukemia.DJ-1 is a redox sensitive adapter protein for high molecular weight complexes involved in regulation of catecholamine homeostasis.Regulation of Signal Transduction by DJ-1.Structural Biology of the DJ-1 Superfamily.DJ-1/PARK7, But Not Its L166P Mutant Linked to Autosomal Recessive Parkinsonism, Modulates the Transcriptional Activity of the Orphan Nuclear Receptor Nurr1 In Vitro and In Vivo.DJ-1 as a Therapeutic Target Against Cancer.The role of DJ-1 complexes and catecholamine metabolism: relevance for familial and idiopathic Parkinson's disease.Overexpression of DJ-1 correlates with aggressive clinicopathological characteristics and poor prognosis in malignant tumors: a meta-analysis.
P2860
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P2860
The oxidation states of DJ-1 dictate the cell fate in response to oxidative stress triggered by 4-hpr: autophagy or apoptosis?
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@ast
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@en
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@nl
type
label
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@ast
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@en
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@nl
prefLabel
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@ast
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@en
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@nl
P2093
P2860
P356
P1476
The oxidation states of DJ-1 d ...... 4-hpr: autophagy or apoptosis?
@en
P2093
Guanyu Lin
Hailin Yan
Meidan Ying
Qiaojun He
Xiaochun Yang
P2860
P304
P356
10.1089/ARS.2013.5446
P577
2014-02-19T00:00:00Z