Functional correction of adult mdx mouse muscle using gutted adenoviral vectors expressing full-length dystrophin. - Wikidata - wikimedia - TriplyDB

Functional correction of adult mdx mouse muscle using gutted adenoviral vectors expressing full-length dystrophin.

Functional correction of adult mdx mouse muscle using gutted adenoviral vectors expressing full-length dystrophin.

http://www.wikidata.org/entity/Q34190967

about

Current understanding of molecular pathology and treatment of cardiomyopathy in duchenne muscular dystrophy Animal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapy Gutless adenovirus: last-generation adenovirus for gene therapy Enhanced effect of microdystrophin gene transfection by HSV-VP22 mediated intercellular protein transport Dystrophin delivery to muscles of mdx mice using lentiviral vectors leads to myogenic progenitor targeting and stable gene expression.Gene and cell-mediated therapies for muscular dystrophy.Sarcolemmal nNOS anchoring reveals a qualitative difference between dystrophin and utrophin.Gene therapy for muscular dystrophies: progress and challenges Systemic delivery of genes to striated muscles using adeno-associated viral vectors.Physiological characterization of muscle strength with variable levels of dystrophin restoration in mdx mice following local antisense therapy.Body-wide gene therapy of Duchenne muscular dystrophy in the mdx mouse model.Targeting artificial transcription factors to the utrophin A promoter: effects on dystrophic pathology and muscle function.Sarcospan reduces dystrophic pathology: stabilization of the utrophin-glycoprotein complex.The FVB Background Does Not Dramatically Alter the Dystrophic Phenotype of Mdx Mice Gene therapy for muscular dystrophy - a review of promising progress.Gene therapy in orthopaedic surgery: the current status.Soluble miniagrin enhances contractile function of engineered skeletal muscle.How much dystrophin is enough: the physiological consequences of different levels of dystrophin in the mdx mouse The muscular dystrophies: from genes to therapies.Viral-mediated gene therapy for the muscular dystrophies: successes, limitations and recent advances Recombinant adeno-associated viral (rAAV) vectors as therapeutic tools for Duchenne muscular dystrophy (DMD).Duchenne muscular dystrophy and dystrophin: pathogenesis and opportunities for treatment Effect of rapamycin on immunity induced by vector-mediated dystrophin expression in mdx skeletal muscle Mechanisms of stretch-induced muscle damage in normal and dystrophic muscle: role of ionic changes.Therapeutic restoration of dystrophin expression in Duchenne muscular dystrophy.Gene therapy in sport Cell-lineage regulated myogenesis for dystrophin replacement: a novel therapeutic approach for treatment of muscular dystrophy Emerging strategies for cell and gene therapy of the muscular dystrophies Diaphragm muscle strip preparation for evaluation of gene therapies in mdx mice.Systemic microdystrophin gene delivery improves skeletal muscle structure and function in old dystrophic mdx mice.Designing heart performance by gene transfer.Gene therapy in large animal models of muscular dystrophy.Genome Editing Gene Therapy for Duchenne Muscular Dystrophy.Preclinical studies for gene therapy of Duchenne muscular dystrophy.Biologic treatment of mild and moderate intervertebral disc degeneration.Gene transfer to the outflow tract.Nanotherapy for Duchenne muscular dystrophy.Progress toward Gene Therapy for Duchenne Muscular Dystrophy.Strength and muscle specificity of a compact promoter derived from the slow troponin I gene in the context of episomal (gutless adenovirus) and integrating (lentiviral) vectors.Capsid-modified adenoviral vectors for improved muscle-directed gene therapy

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Functional correction of adult mdx mouse muscle using gutted adenoviral vectors expressing full-length dystrophin.

http://www.wikidata.org/entity/Q34190967

description

2002 nî lūn-bûn

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2002 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2002 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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type

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Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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Functional correction of adult ...... essing full-length dystrophin.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Functional correction of adult ...... ressing full-length dystrophin

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P2093

Ann S Robinson

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Catherine Barjot

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Christiana DelloRusso

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Dennis Hartigan-O'Connor

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Giovanni Salvatori

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Jeannine M Scott

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Jeffrey S Chamberlain

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Robert W Crawford

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P2860

The complete sequence of dystrophin predicts a rod-shaped cytoskeletal protein

Developments in gene therapy for muscular dystrophy.

Cellular immunity to viral antigens limits E1-deleted adenoviruses for gene therapy

Persistence in muscle of an adenoviral vector that lacks all viral genes.

Forced expression of dystrophin deletion constructs reveals structure-function correlations.

Dystrophin protects the sarcolemma from stresses developed during muscle contraction

Assembly of the dystrophin-associated protein complex does not require the dystrophin COOH-terminal domain

Specific cloning of DNA fragments absent from the DNA of a male patient with an X chromosome deletion.

A new adenoviral vector: Replacement of all viral coding sequences with 28 kb of DNA independently expressing both full-length dystrophin and beta-galactosidase.

Functional protection of dystrophic mouse (mdx) muscles after adenovirus-mediated transfer of a dystrophin minigene.

P304

12979-12984

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P31

scholarly article

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10.1073/PNAS.202300099

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P407

P433

20

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P478

99

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P577

2002-09-23T00:00:00Z

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10658723

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12271128

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P921

P932

130572

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