Brain estrogen deficiency accelerates Abeta plaque formation in an Alzheimer's disease animal model. - Wikidata - wikimedia - TriplyDB

Brain estrogen deficiency accelerates Abeta plaque formation in an Alzheimer's disease animal model.

Brain estrogen deficiency accelerates Abeta plaque formation in an Alzheimer's disease animal model.

http://www.wikidata.org/entity/Q34244907

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Menopause, obesity and inflammation: interactive risk factors for Alzheimer's disease Natural products as aromatase inhibitors Identification of a regulatory loop for the synthesis of neurosteroids: a steroidogenic acute regulatory protein-dependent mechanism involving hypothalamic-pituitary-gonadal axis receptors Sex and Gender Differences in Central Nervous System-Related Disorders The endocrine dyscrasia that accompanies menopause and andropause induces aberrant cell cycle signaling that triggers re-entry of post-mitotic neurons into the cell cycle, neurodysfunction, neurodegeneration and cognitive disease Research progress on flavonoids isolated from traditional Chinese medicine in treatment of Alzheimer's disease Differential contributions of ApoE4 and female sex to BACE1 activity and expression mediate Aβ deposition and learning and memory in mouse models of Alzheimer's disease Effect of Estradiol on Neurotrophin Receptors in Basal Forebrain Cholinergic Neurons: Relevance for Alzheimer's Disease Progesterone receptors: form and function in brain Ligand for translocator protein reverses pathology in a mouse model of Alzheimer's disease Interactions between inflammation, sex steroids, and Alzheimer's disease risk factors Luteinizing hormone receptor mediates neuronal pregnenolone production via up-regulation of steroidogenic acute regulatory protein expression The roles of membrane estrogen receptor subtypes in modulating dopamine transporters in PC-12 cells Overexpression of low-density lipoprotein receptor in the brain markedly inhibits amyloid deposition and increases extracellular A beta clearance The detection of surfactant proteins A, B, C and D in the human brain and their regulation in cerebral infarction, autoimmune conditions and infections of the CNS.Estrogen actions in the brain and the basis for differential action in men and women: a case for sex-specific medicines.Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination.Molecular mechanisms for Alzheimer's disease: implications for neuroimaging and therapeutics.Estradiol prevents olfactory dysfunction induced by A-β 25-35 injection in hippocampus.Gender- and age-dependent gamma-secretase activity in mouse brain and its implication in sporadic Alzheimer disease.Estrogen-Related Factors in the Frontal Lobe of Alzheimer's Disease Patients and Importance of Body Mass Index.Brain sex matters: estrogen in cognition and Alzheimer's disease Non-feminizing estrogens: a novel neuroprotective therapy.Age-dependent regulation of obesity and Alzheimer-related outcomes by hormone therapy in female 3xTg-AD mice.Estrogen therapy and cognition: a review of the cholinergic hypothesis Sex steroid levels and AD-like pathology in 3xTgAD mice.Sex differences in cognitive impairment and Alzheimer's disease.Down-regulation of serum gonadotropins is as effective as estrogen replacement at improving menopause-associated cognitive deficits.17β-Estradiol regulates insulin-degrading enzyme expression via an ERβ/PI3-K pathway in hippocampus: relevance to Alzheimer's prevention Progression from mild cognitive impairment to Alzheimer's disease: effects of sex, butyrylcholinesterase genotype, and rivastigmine treatment.Brain levels of sex steroid hormones in men and women during normal aging and in Alzheimer's disease.Metabolic alteration of neuroactive steroids and protective effect of progesterone in Alzheimer's disease-like rats Development of subtype-selective oestrogen receptor-based therapeutics.Comparative gene expression analysis in mouse models for multiple sclerosis, Alzheimer's disease and stroke for identifying commonly regulated and disease-specific gene changes Estrogen anti-inflammatory activity in brain: a therapeutic opportunity for menopause and neurodegenerative diseases.Reduction of β-amyloid and γ-secretase by calorie restriction in female Tg2576 mice A novel mouse model of Alzheimer's disease with chronic estrogen deficiency leads to glial cell activation and hypertrophy.The perimenopausal aging transition in the female rat brain: decline in bioenergetic systems and synaptic plasticity Effects of aromatase inhibition versus gonadectomy on hippocampal complex amyloid pathology in triple transgenic mice Variants in CYP17 and CYP19 cytochrome P450 genes are associated with onset of Alzheimer's disease in women with down syndrome.

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Brain estrogen deficiency accelerates Abeta plaque formation in an Alzheimer's disease animal model.

http://www.wikidata.org/entity/Q34244907

description

2005 nî lūn-bûn

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2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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type

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Proceedings of the National Academy of Sciences of the United States of America

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Brain estrogen deficiency acce ...... heimer's disease animal model.

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Melissa Lu

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Nobuhiro Harada

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Phillip Cao

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Rena Li

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Shin-Ichiro Honda

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Techie Lancaster

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Xu Yue

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Yong Shen

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Zhenyu Zhong

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P2860

Insulin-degrading enzyme regulates extracellular levels of amyloid beta-protein by degradation

Beta-secretase cleavage of Alzheimer's amyloid precursor protein by the transmembrane aspartic protease BACE

Gender differences in the incidence of AD and vascular dementia: The EURODEM Studies

Amyloid beta peptide load is correlated with increased beta-secretase activity in sporadic Alzheimer's disease patients

Identification of the major Abeta1-42-degrading catabolic pathway in brain parenchyma: suppression leads to biochemical and pathological deposition

Sources of estrogen and their importance

Estrogen regulates neprilysin activity in rat brain

Androgen- and estrogen-dependent regulation of insulin-degrading enzyme in subcellular fractions of rat prostate and uterus

Insulysin hydrolyzes amyloid beta peptides to products that are neither neurotoxic nor deposit on amyloid plaques.

Estrogen reduces neuronal generation of Alzheimer beta-amyloid peptides.

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19198-19203

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10.1073/PNAS.0505203102

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52

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102

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Matthias Staufenbiel

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2005-12-19T00:00:00Z

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7405946

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16365303

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Alzheimer's disease

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1323154

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