Polyclonal B cell activation in lupus-prone mice precedes and predicts the development of autoimmune disease.
about
Autoimmunity, polyclonal B-cell activation and infectionAdjuvant activity of CpG oligodeoxynucleotidesCD40 gene silencing reduces the progression of experimental lupus nephritis modulating local milieu and systemic mechanismsPleuropulmonary manifestations of systemic lupus erythematosus.Self DNA from lymphocytes that have undergone activation-induced cell death enhances murine B cell proliferation and antibody production.CpG DNA as a vaccine adjuvant.Hypergammaglobulinaemia and autoimmune rheumatic diseases.Use of CpG oligodeoxynucleotides as immunoprotective agents.Lupus susceptibility loci in New Zealand mice.Shared signaling networks active in B cells isolated from genetically distinct mouse models of lupus.Host-microflora interaction in systemic lupus erythematosus (SLE): circulating antibodies to the indigenous bacteria of the intestinal tract.Host-microflora interaction in systemic lupus erythematosus (SLE): colonization resistance of the indigenous bacteria of the intestinal tractRepertoire cloning of lupus anti-DNA autoantibodies.Regulatory B cells (B10 cells) have a suppressive role in murine lupus: CD19 and B10 cell deficiency exacerbates systemic autoimmunity.Evidence for multiple mechanisms of polyclonal T cell activation in murine lupus.Intrinsic autoimmune capacities of hematopoietic cells from female New Zealand hybrid mice.Breaching peripheral tolerance promotes the production of HIV-1-neutralizing antibodies.The genetic basis of autoimmune disease in MRL-lpr/lpr mice.Autoimmunity and tolerance in Ig-transgenic mice: murine SLE as a model to study B cell tolerance.Abnormalities in the regulation of variable region genes that encode for antibodies to DNA may be a central factor in the pathogenesis of systemic lupus erythematosus.Impaired cytotoxic T lymphocyte activity in systemic lupus erythematosus following in vitro polyclonal T cell stimulation: a contributory role for non-T cells.The Role of Hyaluronan and CD44 in the Pathogenesis of Lupus Nephritis.Fatty Acid Amide Hydrolase Regulates Peripheral B Cell Receptor Revision, Polyreactivity, and B1 Cells in Lupus.Expansion of marginal zone B cells is not sufficient for the development of renal disease in NZBxNZW F1 mice.
P2860
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P2860
Polyclonal B cell activation in lupus-prone mice precedes and predicts the development of autoimmune disease.
description
1990 nî lūn-bûn
@nan
1990 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@ast
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@en
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@nl
type
label
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@ast
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@en
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@nl
prefLabel
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@ast
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@en
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@nl
P2860
P356
P1476
Polyclonal B cell activation i ...... lopment of autoimmune disease.
@en
P2093
Klinman DM
P2860
P304
P356
10.1172/JCI114831
P407
P577
1990-10-01T00:00:00Z