Ketone body utilization drives tumor growth and metastasis. - Wikidata - wikimedia - TriplyDB

Ketone body utilization drives tumor growth and metastasis.

Ketone body utilization drives tumor growth and metastasis.

http://www.wikidata.org/entity/Q34307146

about

Oncogenes induce the cancer-associated fibroblast phenotype: metabolic symbiosis and "fibroblast addiction" are new therapeutic targets for drug discovery Evaluation and prognostic significance of ACAT1 as a marker of prostate cancer progression Ethanol exposure induces the cancer-associated fibroblast phenotype and lethal tumor metabolism: implications for breast cancer prevention A gene expression estimator of intramuscular fat percentage for use in both cattle and sheep The nutritional phenome of EMT-induced cancer stem-like cells Deregulation of the EGFR/PI3K/PTEN/Akt/mTORC1 pathway in breast cancer: possibilities for therapeutic intervention.FABP7 and HMGCS2 are novel protein markers for apocrine differentiation categorizing apocrine carcinoma of the breast.The ketogenic diet and hyperbaric oxygen therapy prolong survival in mice with systemic metastatic cancer.Identification of Serum Markers of Esophageal Adenocarcinoma by Global and Targeted Metabolic Profiling Hydroxycarboxylic acid receptors are essential for breast cancer cells to control their lipid/fatty acid metabolism A network-based pathway-expanding approach for pathway analysis.Mitochondria "fuel" breast cancer metabolism: fifteen markers of mitochondrial biogenesis label epithelial cancer cells, but are excluded from adjacent stromal cells Mitochondrial dysfunction in breast cancer cells prevents tumor growth: understanding chemoprevention with metformin.Cigarette smoke metabolically promotes cancer, via autophagy and premature aging in the host stromal microenvironment.Compartment-specific activation of PPARγ governs breast cancer tumor growth, via metabolic reprogramming and symbiosis.Intermittent energy restriction induces changes in breast gene expression and systemic metabolism Autophagy in breast cancer and its implications for therapy Could Intermittent Energy Restriction and Intermittent Fasting Reduce Rates of Cancer in Obese, Overweight, and Normal-Weight Subjects? A Summary of Evidence Oncogenes and inflammation rewire host energy metabolism in the tumor microenvironment: RAS and NFκB target stromal MCT4.Discovery of genes from feces correlated with colorectal cancer progression The action of β-hydroxybutyrate on the growth, metabolism and global histone H3 acetylation of spontaneous mouse mammary tumours: evidence of a β-hydroxybutyrate paradox Molecular and diagnostic features of apocrine breast lesions.Fibroblast activation and senescence in oral cancer.Multi-dimensional Roles of Ketone Bodies in Fuel Metabolism, Signaling, and Therapeutics.Ketone supplementation decreases tumor cell viability and prolongs survival of mice with metastatic cancer Cell-surface G-protein-coupled receptors for tumor-associated metabolites: A direct link to mitochondrial dysfunction in cancer.Metabolic Plasticity of Stem Cells and Macrophages in Cancer.Inactivation of HMGCL promotes proliferation and metastasis of nasopharyngeal carcinoma by suppressing oxidative stress.Age-related changes in the transcriptome of antibody-secreting cells.Vitamin C and Doxycycline: A synthetic lethal combination therapy targeting metabolic flexibility in cancer stem cells (CSCs).Synergy of Dendritic Cell Vaccines and Avasimibe in Treatment of Head and Neck Cancer in Mice.Cancer metabolism: new validated targets for drug discovery.Response to commentary by Champ and Klement: Is a ketogenic diet the solution for the hyperglycemia problem in glioblastoma therapy?Mitoketoscins: Novel mitochondrial inhibitors for targeting ketone metabolism in cancer stem cells (CSCs).Preliminary investigation of blood concentrations of insulin-like growth factor, insulin, lactate and β-hydroxybutyrate in dogs with lymphoma as compared with matched controls.Discovery and validation of urinary metabotypes for the diagnosis of hepatocellular carcinoma in West Africans.Tracking cellular and molecular changes in a species-specific manner during experimental tumor progression in vivo.Cancer stem cells (CSCs): metabolic strategies for their identification and eradication.Insulin promotes progression of colon cancer by upregulation of ACAT1.Beta-hydroxybutyrate (3-OHB) can influence the energetic phenotype of breast cancer cells, but does not impact their proliferation and the response to chemotherapy or radiation.

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P2860

Ketone body utilization drives tumor growth and metastasis.

http://www.wikidata.org/entity/Q34307146

description

2012 nî lūn-bûn

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2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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type

label

Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Ketone body utilization drives tumor growth and metastasis.

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Anthony Howell

http://www.w3.org/2001/XMLSchema#string

Ubaldo E Martinez-Outschoorn

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Zhao Lin

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P2860

Ketones and lactate "fuel" tumor growth and metastasis: Evidence that epithelial cancer cells use oxidative mitochondrial metabolism

An absence of stromal caveolin-1 expression predicts early tumor recurrence and poor clinical outcome in human breast cancers

Ketone bodies: a review of physiology, pathophysiology and application of monitoring to diabetes.

Is there an astrocyte-neuron ketone body shuttle?

Ketone bodies as a therapeutic for Alzheimer's disease.

Stromal-epithelial metabolic coupling in cancer: integrating autophagy and metabolism in the tumor microenvironment.

Ketones and lactate increase cancer cell "stemness," driving recurrence, metastasis and poor clinical outcome in breast cancer: achieving personalized medicine via Metabolo-Genomics

Evidence for a stromal-epithelial "lactate shuttle" in human tumors: MCT4 is a marker of oxidative stress in cancer-associated fibroblasts.

Hyperactivation of oxidative mitochondrial metabolism in epithelial cancer cells in situ: visualizing the therapeutic effects of metformin in tumor tissue.

Using the "reverse Warburg effect" to identify high-risk breast cancer patients: stromal MCT4 predicts poor clinical outcome in triple-negative breast cancers.

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3964-3971

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10.4161/CC.22137

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21

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11

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Michael P. Lisanti

Diana Whitaker-Menezes

Federica Sotgia

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2012-09-19T00:00:00Z

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230884442

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23082722

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3507492

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