Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice. - Wikidata - wikimedia - TriplyDB

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

http://www.wikidata.org/entity/Q34333682

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Modulation of Runx2 activity by estrogen receptor-alpha: implications for osteoporosis and breast cancer Kif3a deficiency reverses the skeletal abnormalities in Pkd1 deficient mice by restoring the balance between osteogenesis and adipogenesis Myocyte enhancer factor 2c, an osteoblast transcription factor identified by dimethyl sulfoxide (DMSO)-enhanced mineralization Synaptotagmin VII regulates bone remodeling by modulating osteoclast and osteoblast secretion Osteoblast-specific deletion of Pkd2 leads to low-turnover osteopenia and reduced bone marrow adiposity RAGE supports parathyroid hormone-induced gains in femoral trabecular bone Conditional disruption of Pkd1 in osteoblasts results in osteopenia due to direct impairment of bone formation.Role of the polycytin-primary cilia complex in bone development and mechanosensing Disruption of the Fgf2 gene activates the adipogenic and suppresses the osteogenic program in mesenchymal marrow stromal stem cells.Glutamine repeat variants in human RUNX2 associated with decreased femoral neck BMD, broadband ultrasound attenuation and target gene transactivation Conditional mesenchymal disruption of pkd1 results in osteopenia and polycystic kidney disease.Polyalanine repeat polymorphism in RUNX2 is associated with site-specific fracture in post-menopausal females Cilia involvement in patterning and maintenance of the skeleton Deletion of Mecom in mouse results in early-onset spinal deformity and osteopenia.Runx2 protein expression utilizes the Runx2 P1 promoter to establish osteoprogenitor cell number for normal bone formation Conditional deletion of Pkd1 in osteocytes disrupts skeletal mechanosensing in mice.Cilia-like structures and polycystin-1 in osteoblasts/osteocytes and associated abnormalities in skeletogenesis and Runx2 expression.Central Role of Core Binding Factor β2 in Mucosa-Associated Lymphoid Tissue Organogenesis in Mouse Ectopic expression of SOX9 in osteoblasts alters bone mechanical properties.Disruption of Kif3a in osteoblasts results in defective bone formation and osteopenia.Prevention of premature fusion of calvarial suture in GLI-Kruppel family member 3 (Gli3)-deficient mice by removing one allele of Runt-related transcription factor 2 (Runx2).Longitudinal evaluation of FGF23 changes and mineral metabolism abnormalities in a mouse model of chronic kidney disease.Polycystin-1 regulates skeletogenesis through stimulation of the osteoblast-specific transcription factor RUNX2-II.FGF2 stimulation of the pyrophosphate-generating enzyme, PC-1, in pre-osteoblast cells is mediated by RUNX2.Overexpression of bone sialoprotein leads to an uncoupling of bone formation and bone resorption in mice.RUNX2 mutations in Chinese patients with cleidocranial dysplasia.Inducible expression of Runx2 results in multiorgan abnormalities in mice.Dose-dependent effects of Runx2 on bone development.Dlx5 specifically regulates Runx2 type II expression by binding to homeodomain-response elements in the Runx2 distal promoter.Runx2 promotes both osteoblastogenesis and novel osteoclastogenic signals in ST2 mesenchymal progenitor cells.Mitotic Inheritance of mRNA Facilitates Translational Activation of the Osteogenic-Lineage Commitment Factor Runx2 in Progeny of Osteoblastic Cells.Inhibition of osteogenic differentiation of human mesenchymal stem cells Haploinsufficiency of Runx2 results in bone formation decrease and different BSP expression pattern changes in two transgenic mouse models.Over-expression of Runx1 transcription factor impairs the development of thymocytes from the double-negative to double-positive stages.Redefining the activity of a bone-specific transcription factor: novel insights for understanding bone formation.Transcription factor Runx2 in the low bone mineral density of girls with adolescent idiopathic scoliosis.Germinated soy germ with increased soyasaponin Ab improves BMP-2-induced bone formation and protects against in vivo bone loss in osteoporosis

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P2860

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

http://www.wikidata.org/entity/Q34333682

description

2005 nî lūn-bûn

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2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2005 թվականի հուլիսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@ast

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@en

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@nl

type

label

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@ast

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@en

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@nl

prefLabel

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@ast

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@en

Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

@nl

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J.YDBIO.2005.04.028

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Developmental Biology

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Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.

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Farshid Guilak

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Hani A Awad

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Josh Mahlios

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Leigh Darryl Quarles

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Matthew S Mayo

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Shiguang Liu

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Shiqin Zhang

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Zhousheng Xiao

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P2860

The novel zinc finger-containing transcription factor osterix is required for osteoblast differentiation and bone formation

Mutations involving the transcription factor CBFA1 cause cleidocranial dysplasia

Indian hedgehog signaling regulates proliferation and differentiation of chondrocytes and is essential for bone formation

MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

Osf2/Cbfa1: a transcriptional activator of osteoblast differentiation

Critical roles for collagenase-3 (Mmp13) in development of growth plate cartilage and in endochondral ossification

Maturational disturbance of chondrocytes in Cbfa1-deficient mice

Ihh signaling is directly required for the osteoblast lineage in the endochondral skeleton

Cbfbeta interacts with Runx2 and has a critical role in bone development

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10.1016/J.YDBIO.2005.04.028

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7806329

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1360182

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