U(S)3 protein kinase of herpes simplex virus 1 blocks caspase 3 activation induced by the products of U(S)1.5 and U(L)13 genes and modulates expression of transduced U(S)1.5 open reading frame in a cell type-specific manner.
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Proteolytic cleavage of VP1-2 is required for release of herpes simplex virus 1 DNA into the nucleusMapping of key functions of the herpes simplex virus 1 U(S)3 protein kinase: the U(S)3 protein can form functional heteromultimeric structures derived from overlapping truncated polypeptidesBovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting ApoptosisTowards an understanding of the herpes simplex virus type 1 latency-reactivation cycleThe suppression of apoptosis by α-herpesvirus.Herpes simplex virus 1 ICP22 regulates the accumulation of a shorter mRNA and of a truncated US3 protein kinase that exhibits altered functionsU(S)3 and U(S)3.5 protein kinases of herpes simplex virus 1 differ with respect to their functions in blocking apoptosis and in virion maturation and egress.ICP0 and the US3 protein kinase of herpes simplex virus 1 independently block histone deacetylation to enable gene expression.Herpes simplex virus type 1 and bovine herpesvirus 1 latency.Sequence variation in the herpes simplex virus U(S)1 ocular virulence determinant.Herpes simplex virus infection and apoptosis.The UL13 and US3 Protein Kinases of Herpes Simplex Virus 1 Cooperate to Promote the Assembly and Release of Mature, Infectious Virions.Origin of expression of the herpes simplex virus type 1 protein U(S)1.5.Constitutive and Inducible Innate Responses in Cells Infected by HSV-1-Derived Amplicon Vectors.Manipulation of apoptosis and necroptosis signaling by herpesvirusesIdentification of sequences in herpes simplex virus type 1 ICP22 that influence RNA polymerase II modification and viral late gene expression.Varicella-zoster virus-infected human sensory neurons are resistant to apoptosis, yet human foreskin fibroblasts are susceptible: evidence for a cell-type-specific apoptotic response.Herpes simplex virus type 1 immediate-early gene expression is required for the induction of apoptosis in human epithelial HEp-2 cellsHerpes simplex virus UL14 protein blocks apoptosis.Cultured vestibular ganglion neurons demonstrate latent HSV1 reactivation.Elevated PrPC expression predisposes to increased HSV-1 pathogenicity.VZV ORF63 protects human neuronal and keratinocyte cell lines from apoptosis and changes its localization upon apoptosis induction.
P2860
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P2860
U(S)3 protein kinase of herpes simplex virus 1 blocks caspase 3 activation induced by the products of U(S)1.5 and U(L)13 genes and modulates expression of transduced U(S)1.5 open reading frame in a cell type-specific manner.
description
2002 nî lūn-bûn
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2002 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2002年の論文
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2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@ast
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@en
type
label
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@ast
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@en
prefLabel
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@ast
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@en
P2093
P2860
P1433
P1476
U(S)3 protein kinase of herpes ...... n a cell type-specific manner.
@en
P2093
Alice P W Poon
Joshua Munger
Ryan Hagglund
P2860
P304
P356
10.1128/JVI.76.2.743-754.2002
P407
P577
2002-01-01T00:00:00Z