Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C. - Wikidata - wikimedia - TriplyDB

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

http://www.wikidata.org/entity/Q34425169

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HIV-1 induced bystander apoptosis Longitudinal Analysis of CCR5 and CXCR4 Usage in a Cohort of Antiretroviral Therapy-Naïve Subjects with Progressive HIV-1 Subtype C Infection.Alterations in HIV-1 gp120 V3 region are necessary but not sufficient for coreceptor switching in CRF07_BC in China.Covariance of charged amino acids at positions 322 and 440 of HIV-1 Env contributes to coreceptor specificity of subtype B viruses, and can be used to improve the performance of V3 sequence-based coreceptor usage prediction algorithms.HIV-1 pathogenesis: the virus.CoRSeqV3-C: a novel HIV-1 subtype C specific V3 sequence based coreceptor usage prediction algorithm Utilization of HIV-1 envelope V3 to identify X4- and R5-specific Tat and LTR sequence signatures Mechanisms of HIV-1 subtype C resistance to GRFT, CV-N and SVN.Linkages between HIV-1 specificity for CCR5 or CXCR4 and in vitro usage of alternative coreceptors during progressive HIV-1 subtype C infection.Frequency and Env determinants of HIV-1 subtype C strains from antiretroviral therapy-naive subjects that display incomplete inhibition by maraviroc.Evolution of HIV-1 coreceptor usage and coreceptor switching during pregnancy.Escape from human immunodeficiency virus type 1 (HIV-1) entry inhibitors.Effect of HIV-1 subtype and tropism on treatment with chemokine coreceptor entry inhibitors; overview of viral entry inhibition.Human immunodeficiency virus type 1 biological variation and coreceptor use: from concept to clinical significance.Effect of Amino Acid Substitutions Within the V3 Region of HIV-1 CRF01_AE on Interaction with CCR5-Coreceptor.High-Sequence Diversity and Rapid Virus Turnover Contribute to Higher Rates of Coreceptor Switching in Treatment-Experienced Subjects with HIV-1 Viremia.

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Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

http://www.wikidata.org/entity/Q34425169

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@ast

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@en

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

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type

label

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@ast

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

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Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@nl

prefLabel

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@ast

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@en

Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@nl

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Journal of Acquired Immune Deficiency Syndromes

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Extreme genetic divergence is required for coreceptor switching in HIV-1 subtype C.

@en

P2093

Donald E Mosier

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Mia Coetzer

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Rebecca Nedellec

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Tammy Meyers

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Tonie Cilliers

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P2860

Extensive recombination among human immunodeficiency virus type 1 quasispecies makes an important contribution to viral diversity in individual patients.

Frequent intrapatient recombination between human immunodeficiency virus type 1 R5 and X4 envelopes: implications for coreceptor switch

Use of alternate coreceptors on primary cells by two HIV-1 isolates

Tracking global patterns of N-linked glycosylation site variation in highly variable viral glycoproteins: HIV, SIV, and HCV envelopes and influenza hemagglutinin

The impact of the syncytium-inducing phenotype of human immunodeficiency virus on disease progression

Vpr is required for efficient replication of human immunodeficiency virus type-1 in mononuclear phagocytes

Molecular cloning and analysis of functional envelope genes from human immunodeficiency virus type 1 sequence subtypes A through G. The WHO and NIAID Networks for HIV Isolation and Characterization.

Phylodynamics of HIV-1 in lymphoid and non-lymphoid tissues reveals a central role for the thymus in emergence of CXCR4-using quasispecies

Limited neutralizing antibody specificities drive neutralization escape in early HIV-1 subtype C infection.

V3 loop sequence space analysis suggests different evolutionary patterns of CCR5- and CXCR4-tropic HIV.

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9-15

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