Cholesterol is not an essential source of nutrition for Mycobacterium tuberculosis during infection
about
High-resolution phenotypic profiling defines genes essential for mycobacterial growth and cholesterol catabolismPathogen roid rage: cholesterol utilization by Mycobacterium tuberculosisMetabolic Perspectives on PersistenceCatabolism and biotechnological applications of cholesterol degrading bacteria.Linking the transcriptional profiles and the physiological states of Mycobacterium tuberculosis during an extended intracellular infectionMycobacterium tuberculosis cholesterol catabolism requires a new class of acyl coenzyme A dehydrogenase.The role of 3-ketosteroid 1(2)-dehydrogenase in the pathogenicity of Mycobacterium tuberculosis.Cholesterol oxidase is indispensable in the pathogenesis of Mycobacterium tuberculosisCholesterol catabolism as a therapeutic target in Mycobacterium tuberculosis.The tuberculosis drug discovery and development pipeline and emerging drug targets.The Essential Role of Cholesterol Metabolism in the Intracellular Survival of Mycobacterium leprae Is Not Coupled to Central Carbon Metabolism and Energy Production.Oxadiazoles Have Butyrate-Specific Conditional Activity against Mycobacterium tuberculosisPlasma Membrane Profiling Reveals Upregulation of ABCA1 by Infected Macrophages Leading to Restriction of Mycobacterial Growth.A GMC oxidoreductase homologue is required for acetylation of glycopeptidolipid in Mycobacterium smegmatis.Virulence factors of the Mycobacterium tuberculosis complexUpdating and curating metabolic pathways of TB.3-Ketosteroid 9α-hydroxylase enzymes: Rieske non-heme monooxygenases essential for bacterial steroid degradation.Tuberculosis drug discovery and emerging targets.Cholesterol oxidase binds TLR2 and modulates functional responses of human macrophages.Cholesterol metabolism: a potential therapeutic target in Mycobacteria.The Role of fadD19 and echA19 in Sterol Side Chain Degradation by Mycobacterium smegmatis.CD36-Mediated Uptake of Surfactant Lipids by Human Macrophages Promotes Intracellular Growth of Mycobacterium tuberculosis.α-Methyl Acyl CoA Racemase Provides Mycobacterium tuberculosis Catabolic Access to Cholesterol Esters.Regulation of the KstR2 regulon of Mycobacterium tuberculosis by a cholesterol catabolite.
P2860
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P2860
Cholesterol is not an essential source of nutrition for Mycobacterium tuberculosis during infection
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年学术文章
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2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
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2011年学术文章
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2011年學術文章
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name
Cholesterol is not an essentia ...... tuberculosis during infection
@ast
Cholesterol is not an essentia ...... tuberculosis during infection
@en
Cholesterol is not an essentia ...... tuberculosis during infection
@nl
type
label
Cholesterol is not an essentia ...... tuberculosis during infection
@ast
Cholesterol is not an essentia ...... tuberculosis during infection
@en
Cholesterol is not an essentia ...... tuberculosis during infection
@nl
prefLabel
Cholesterol is not an essentia ...... tuberculosis during infection
@ast
Cholesterol is not an essentia ...... tuberculosis during infection
@en
Cholesterol is not an essentia ...... tuberculosis during infection
@nl
P2093
P2860
P356
P1476
Cholesterol is not an essentia ...... tuberculosis during infection
@en
P2093
Eugenie Dubnau
Issar Smith
Xinxin Yang
P2860
P304
P356
10.1128/JB.01210-10
P407
P577
2011-01-21T00:00:00Z