A Tlr7 translocation accelerates systemic autoimmunity in murine lupus. - Wikidata - wikimedia - TriplyDB

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

http://www.wikidata.org/entity/Q34773105

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Dysregulation of antiviral helicase pathways in systemic lupus erythematosus Caspase-activated DNase is required for maintenance of tolerance to lupus nuclear autoantigens Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)Trex1 prevents cell-intrinsic initiation of autoimmunity Predominant Role of Plasmacytoid Dendritic Cells in Stimulating Systemic Autoimmunity Defects in Germinal Center Selection in SLE Contribution of Toll-like receptor signaling to germinal center antibody responses Toll-like receptors in systemic lupus erythematosus: potential targets for therapeutic intervention Intracellular nucleic acid sensors and autoimmunity Leukocyte Beta-Catenin Expression Is Disturbed in Systemic Lupus Erythematosus Modelling clinical systemic lupus erythematosus: similarities, differences and success stories Sexual dimorphism in autoimmunity Nucleic Acid-Sensing Receptors: Rheostats of Autoimmunity and Autoinflammation Mechanisms of autoantibody production in systemic lupus erythematosus miRNAs in the Pathogenesis of Systemic Lupus Erythematosus The receptor TREML4 amplifies TLR7-mediated signaling during antiviral responses and autoimmunity The lupus-related Lmb3 locus contains a disease-suppressing Coronin-1A gene mutation.Guanine-modified inhibitory oligonucleotides efficiently impair TLR7- and TLR9-mediated immune responses of human immune cells Estrogen modulation of endosome-associated toll-like receptor 8: an IFNα-independent mechanism of sex-bias in systemic lupus erythematosus.The lupus-prone NZM2410/NZW strain-derived Sle1b sublocus alters the germinal center checkpoint in female mice in a B cell-intrinsic manner.The largely normal response to Toll-like receptor 7 and 9 stimulation and the enhanced expression of SIGIRR by B cells in systemic lupus erythematosus.Lupus pathobiology based on genomics.Dosage of X-linked Toll-like receptor 8 determines gender differences in the development of systemic lupus erythematosus.A genomic approach to human autoimmune diseases.IFN regulatory factor 5 is required for disease development in the FcgammaRIIB-/-Yaa and FcgammaRIIB-/- mouse models of systemic lupus erythematosus ICOS expression by effector T cells influences the ability of regulatory T cells to inhibit anti-chromatin B cell responses in recipient mice.Type I interferons produced by resident renal cells may promote end-organ disease in autoantibody-mediated glomerulonephritis.Requirement for MyD88 signaling in B cells and dendritic cells for germinal center anti-nuclear antibody production in Lyn-deficient mice Autoimmunity and Klinefelter's syndrome: when men have two X chromosomes.The unexplained female predominance of systemic lupus erythematosus: clues from genetic and cytokine studies.Pivotal Functions of Plasmacytoid Dendritic Cells in Systemic Autoimmune Pathogenesis.Interferon alpha in systemic lupus erythematosus Recent advances in the genetics of systemic lupus erythematosus.Germinal center B cell depletion diminishes CD4+ follicular T helper cells in autoimmune mice Nucleic acid sensing receptors in systemic lupus erythematosus: development of novel DNA- and/or RNA-like analogues for treating lupus.Monocyte surface expression of Fcgamma receptor RI (CD64), a biomarker reflecting type-I interferon levels in systemic lupus erythematosus.Therapeutic strategies for SLE involving cytokines: mechanism-oriented therapies especially IFN-gamma targeting gene therapy.Treatment with a toll-like receptor inhibitory GpG oligonucleotide delays and attenuates lupus nephritis in NZB/W mice.Decreased frequencies of circulating follicular helper T cell counterparts and plasmablasts in ankylosing spondylitis patients Naïve for TNF blockers.CXCR4/CXCL12 hyperexpression plays a pivotal role in the pathogenesis of lupus.

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A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

http://www.wikidata.org/entity/Q34773105

description

2006 nî lūn-bûn

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2006 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2006 թվականի հունիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@ast

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@en

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@nl

type

label

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@ast

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@en

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@nl

prefLabel

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@ast

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@en

A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

@nl

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PNAS.0603912103

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Proceedings of the National Academy of Sciences of the United States of America

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A Tlr7 translocation accelerates systemic autoimmunity in murine lupus.

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P2093

Andrew Wang

http://www.w3.org/2001/XMLSchema#string

Chaoying Liang

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Edward K Wakeland

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Guy Bartov

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Jinchun Zhou

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Katalin Tus

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Lisa D McDaniel

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Quan-Zhen Li

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Roger A Schultz

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Srividya Subramanian

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P2860

Human TLR7 or TLR8 independently confer responsiveness to the antiviral compound R-848

Species-specific recognition of single-stranded RNA via toll-like receptor 7 and 8

Recognition of single-stranded RNA viruses by Toll-like receptor 7

Small anti-viral compounds activate immune cells via the TLR7 MyD88-dependent signaling pathway

A RING-type ubiquitin ligase family member required to repress follicular helper T cells and autoimmunity

Innate antiviral responses by means of TLR7-mediated recognition of single-stranded RNA

Chromatin-IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors

Genetic dissection of lupus pathogenesis: a recipe for nephrophilic autoantibodies.

The major murine systemic lupus erythematosus susceptibility locus, Sle1, is a cluster of functionally related genes.

Identification of autoantibody clusters that best predict lupus disease activity using glomerular proteome arrays.

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