Differential transformation capacity of Src family kinases during the initiation of prostate cancer.
about
Androgen receptor activation in castration-recurrent prostate cancer: the role of Src-family and Ack1 tyrosine kinasesSRC: marker or actor in prostate cancer aggressivenessCCR5 receptor antagonists block metastasis to bone of v-Src oncogene-transformed metastatic prostate cancer cell lines.Engineered kinase activation reveals unique morphodynamic phenotypes and associated trafficking for Src family isoforms.Targeting protein lipidation in diseaseSaracatinib as a metastasis inhibitor in metastatic castration-resistant prostate cancer: A University of Chicago Phase 2 Consortium and DOD/PCF Prostate Cancer Clinical Trials Consortium Study.Co-expression network analysis identifies Spleen Tyrosine Kinase (SYK) as a candidate oncogenic driver in a subset of small-cell lung cancerC-Src and c-Yes are two unlikely partners of spermatogenesis and their roles in blood-testis barrier dynamics.Differential requirement for Src family tyrosine kinases in the initiation, progression, and metastasis of prostate cancerMolecular pathways and targets in prostate cancerLyn facilitates glioblastoma cell survival under conditions of nutrient deprivation by promoting autophagy.Oncogene-specific activation of tyrosine kinase networks during prostate cancer progression.Yes-mediated phosphorylation of focal adhesion kinase at tyrosine 861 increases metastatic potential of prostate cancer cells.Oncogene-mediated alterations in chromatin conformationFunctions of the Lyn tyrosine kinase in health and diseaseSrc, p130Cas, and Mechanotransduction in Cancer CellsInvestigation of the molecular mechanisms underlying metastasis in prostate cancer by gene expression profilingNovel roles of Src in cancer cell epithelial-to-mesenchymal transition, vascular permeability, microinvasion and metastasis.The residue at position 5 of the N-terminal region of Src and Fyn modulates their myristoylation, palmitoylation, and membrane interactions.LYN expression predicts the response to dasatinib in a subpopulation of lung adenocarcinoma patients.Targeting Src-mediated Tyr216 phosphorylation and activation of GSK-3 in prostate cancer cells inhibit prostate cancer progression in vitro and in vivo.Small-molecule protein tyrosine kinase inhibitors for the treatment of metastatic prostate cancer.Non-receptor protein tyrosine kinases signaling pathways in normal and cancer cells.Src signaling pathways in prostate cancer.Tyrosine kinase inhibitors in the treatment of prostate cancer: taking the next step in clinical development.The prohibitin-repressive interaction with E2F1 is rapidly inhibited by androgen signalling in prostate cancer cells.Lyn kinase mediates cell motility and tumor growth in EGFRvIII-expressing head and neck cancer.Exosomal sorting of the viral oncoprotein LMP1 is restrained by TRAF2 association at signalling endosomes.Lyn tyrosine kinase regulates androgen receptor expression and activity in castrate-resistant prostate cancer.MicroRNA miR-125a-3p modulates molecular pathway of motility and migration in prostate cancer cells.Androgen-sensitive microsomal signaling networks coupled to the proliferation and differentiation of human prostate cancer cells.Smooth muscle contraction and growth of stromal cells in the human prostate are both inhibited by the Src family kinase inhibitors, AZM475271 and PP2.Blocking Myristoylation of Src Inhibits Its Kinase Activity and Suppresses Prostate Cancer Progression.Myristoylation of Src kinase mediates Src-induced and high-fat diet-accelerated prostate tumor progression in mice.Inhibition of SRC family kinases reduces myeloid-derived suppressor cells in head and neck cancer.Paracrine Fibroblast Growth Factor Initiates Oncogenic Synergy with Epithelial FGFR/Src Transformation in Prostate Tumor Progression.CSF-1-induced Src signaling can instruct monocytic lineage choice.Pharmacologically targeting the myristoylation of the scaffold protein FRS2α inhibits FGF/FGFR-mediated oncogenic signaling and tumor progression.Targeting autophagy sensitises lung cancer cells to Src family kinase inhibitors.Emerging Roles of Fibroblast Growth Factor 10 in Cancer
P2860
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P2860
Differential transformation capacity of Src family kinases during the initiation of prostate cancer.
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Differential transformation ca ...... initiation of prostate cancer.
@ast
Differential transformation ca ...... initiation of prostate cancer.
@en
Differential transformation ca ...... initiation of prostate cancer.
@nl
type
label
Differential transformation ca ...... initiation of prostate cancer.
@ast
Differential transformation ca ...... initiation of prostate cancer.
@en
Differential transformation ca ...... initiation of prostate cancer.
@nl
prefLabel
Differential transformation ca ...... initiation of prostate cancer.
@ast
Differential transformation ca ...... initiation of prostate cancer.
@en
Differential transformation ca ...... initiation of prostate cancer.
@nl
P2093
P2860
P356
P1476
Differential transformation ca ...... initiation of prostate cancer.
@en
P2093
Clifford A Lowell
Daniel A Smith
Houjian Cai
Jonathan A Cooper
Owen N Witte
Sanaz Memarzadeh
P2860
P304
P356
10.1073/PNAS.1103904108
P407
P577
2011-04-04T00:00:00Z