Control of central nervous system viral persistence by neutralizing antibody.
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Antisense morpholino-oligomers directed against the 5' end of the genome inhibit coronavirus proliferation and growthClearance of virus infection from the CNSThe pathogenesis of murine coronavirus infection of the central nervous system.Progression from IgD+ IgM+ to isotype-switched B cells is site specific during coronavirus-induced encephalomyelitis.FTY720 (fingolimod) modulates the severity of viral-induced encephalomyelitis and demyelination.Enhancement of blood-brain barrier permeability is required for intravenously administered virus neutralizing antibodies to clear an established rabies virus infection from the brain and prevent the development of rabies in mice.Factors supporting intrathecal humoral responses following viral encephalomyelitis.CXCR3-dependent plasma blast migration to the central nervous system during viral encephalomyelitis.Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination.Axonal pathology and demyelination in viral models of multiple sclerosis.T helper cell- and CD40-dependent germline IgM prevents chronic virus-induced demyelinating diseaseMurine coronavirus-induced oligodendrocyte apoptosis is mediated through the activation of the Fas signaling pathway.CD4 T cells promote CD8 T cell immunity at the priming and effector site during viral encephalitisClearance of an immunosuppressive virus from the CNS coincides with immune reanimation and diversification.CXCR2 signaling and host defense following coronavirus-induced encephalomyelitis.Oral administration of live virus protects susceptible mice from developing Theiler's virus-induced demyelinating disease.The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Interleukin-10 is a critical regulator of white matter lesion containment following viral induced demyelinationCXCL13 promotes isotype-switched B cell accumulation to the central nervous system during viral encephalomyelitis.Insertion of the CXC chemokine ligand 9 (CXCL9) into the mouse hepatitis virus genome results in protection from viral-induced encephalitis and hepatitisIL-15 independent maintenance of virus-specific CD8(+) T cells in the CNS during chronic infection.IL-21 optimizes T cell and humoral responses in the central nervous system during viral encephalitis.Mouse hepatitis virus infection of the CNS: a model for defense, disease, and repair.The Biology of Persistent Infection: Inflammation and Demyelination following Murine Coronavirus Infection of the Central Nervous SystemExpression of cellular oncogene Bcl-xL prevents coronavirus-induced cell death and converts acute infection to persistent infection in progenitor rat oligodendrocytesIntrathecal humoral immunity to encephalitic RNA viruses.Immune responses to non-tumor antigens in the central nervous system.Persistent neuroinvasive West Nile virus infection in an immunocompromised patient.Interferon gamma modulation of disease manifestation and the local antibody response to alphavirus encephalomyelitis.Adaptive immune response to viral infections in the central nervous system.Protective Humoral Immunity in the CNS Requires Peripheral CD19-Dependent Germinal Center Formation Following Coronavirus Encephalomyelitis.
P2860
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P2860
Control of central nervous system viral persistence by neutralizing antibody.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
name
Control of central nervous system viral persistence by neutralizing antibody.
@ast
Control of central nervous system viral persistence by neutralizing antibody.
@en
Control of central nervous system viral persistence by neutralizing antibody.
@nl
type
label
Control of central nervous system viral persistence by neutralizing antibody.
@ast
Control of central nervous system viral persistence by neutralizing antibody.
@en
Control of central nervous system viral persistence by neutralizing antibody.
@nl
prefLabel
Control of central nervous system viral persistence by neutralizing antibody.
@ast
Control of central nervous system viral persistence by neutralizing antibody.
@en
Control of central nervous system viral persistence by neutralizing antibody.
@nl
P2093
P2860
P1433
P1476
Control of central nervous system viral persistence by neutralizing antibody.
@en
P2093
Chandran Ramakrishna
Cornelia C Bergmann
Roscoe Atkinson
Stephen A Stohlman
P2860
P304
P356
10.1128/JVI.77.8.4670-4678.2003
P407
P577
2003-04-01T00:00:00Z