Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells. - Wikidata - wikimedia - TriplyDB

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

http://www.wikidata.org/entity/Q34927437

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Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci GARP: a surface molecule of regulatory T cells that is involved in the regulatory function and TGF-β releasing Cellular and molecular determinants for the development of natural and induced regulatory T cells Skewed Helper T-Cell Responses to IL-12 Family Cytokines Produced by Antigen-Presenting Cells and the Genetic Background in Behcet's Disease The development and in vivo function of T helper 9 cells MicroRNA-21 promotes Th17 differentiation and mediates experimental autoimmune encephalomyelitis Identification of possible pathogenic pathways in Behçet's disease using genome-wide association study data from two different populations Prioritizing biological pathways by recognizing context in time-series gene expression data TGF-β activation and function in immunity.Therapeutic potential of TGF-β-induced CD4(+) Foxp3(+) regulatory T cells in autoimmune diseases.Counter-regulation of T cell effector function by differentially activated p38.The function of BAFF on T helper cells in autoimmunity Transforming growth factor β1 promotes invasion of human JEG-3 trophoblast cells via TGF-β/Smad3 signaling pathway.Role of the adjacent stroma cells in prostate cancer development and progression: synergy between TGF-β and IGF signaling.Crosstalk between the p38 and TGF-β signaling pathways through TβRI, TβRII and Smad3 expression in plancental choriocarcinoma JEG-3 cells.Isolation of purified and live Foxp3+ regulatory T cells using FACS sorting on scatter plot.BAFF promotes Th17 cells and aggravates experimental autoimmune encephalomyelitis All-trans retinoic acid promotes TGF-β-induced Tregs via histone modification but not DNA demethylation on Foxp3 gene locus.Critical role of all-trans retinoic acid in stabilizing human natural regulatory T cells under inflammatory conditions SMAD2 is essential for TGF beta-mediated Th17 cell generation.IL-1β-mediated signals preferentially drive conversion of regulatory T cells but not conventional T cells into IL-17-producing cells.Secretion of immunoregulatory cytokines by mesenchymal stem cells.Aberrant Activation of Heat Shock Protein 60/65 Reactive T Cells in Patients with Behcet's Disease.Advances in distinguishing natural from induced Foxp3(+) regulatory T cells Cutting edge: all-trans retinoic acid sustains the stability and function of natural regulatory T cells in an inflammatory milieu.Generation of human regulatory T cells de novo with suppressive function prevent xenogeneic graft versus host disease.Dichotomous response to transforming growth factor β after T cell receptor activation by naive CD4+ T cells from DBA/1 mice: enhanced retinoic acid receptor-related orphan nuclear receptor γt expression yet reduced FoxP3 expression.The histone peptide H4 71-94 alone is more effective than a cocktail of peptide epitopes in controlling lupus: immunoregulatory mechanisms.FOXP3+ regulatory T cell development and function require histone/protein deacetylase 3.Antigen-specific transforming growth factor β-induced Treg cells, but not natural Treg cells, ameliorate autoimmune arthritis in mice by shifting the Th17/Treg cell balance from Th17 predominance to Treg cell predominance.Activation of p38 MAPK in CD4 T cells controls IL-17 production and autoimmune encephalomyelitis The Active Form of Vitamin D Transcriptionally Represses Smad7 Signaling and Activates Extracellular Signal-regulated Kinase (ERK) to Inhibit the Differentiation of a Inflammatory T Helper Cell Subset and Suppress Experimental Autoimmune Encephalomy Exome analysis of patients with concurrent pediatric inflammatory bowel disease and autoimmune disease.Signaling via the kinase p38α programs dendritic cells to drive TH17 differentiation and autoimmune inflammation Requirements of transcription factor Smad-dependent and -independent TGF-β signaling to control discrete T-cell functions.Phosphorylation status determines the opposing functions of Smad2/Smad3 as STAT3 cofactors in TH17 differentiation Gastric Helicobacter pylori Infection Affects Local and Distant Microbial Populations and Host Responses.PSG9 Stimulates Increase in FoxP3+ Regulatory T-Cells through the TGF-β1 Pathway Selective Impairment of TH17-Differentiation and Protection against Autoimmune Arthritis after Overexpression of BCL2A1 in T Lymphocytes The role of all-trans retinoic acid in the biology of Foxp3+ regulatory T cells

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Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

http://www.wikidata.org/entity/Q34927437

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@ast

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@en

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@nl

type

label

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@ast

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@en

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@nl

prefLabel

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@ast

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@en

Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

@nl

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JIMMUNOL.0903418

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Journal of Immunology

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Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cells.

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P2093

David A Horwitz

http://www.w3.org/2001/XMLSchema#string

David Brand

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Feng Zhang

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Ling Lu

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Song Guo Zheng

http://www.w3.org/2001/XMLSchema#string

Wei Shi

http://www.w3.org/2001/XMLSchema#string

Xuehao Wang

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Yang Chai

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P2860

Disruption of a new forkhead/winged-helix protein, scurfin, results in the fatal lymphoproliferative disorder of the scurfy mouse

TGF beta signals through a heteromeric protein kinase receptor complex

IL-21 is produced by Th17 cells and drives IL-17 production in a STAT3-dependent manner

Conversion of peripheral CD4+CD25- naive T cells to CD4+CD25+ regulatory T cells by TGF-beta induction of transcription factor Foxp3

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Foxp3 programs the development and function of CD4+CD25+ regulatory T cells

Transforming growth factor-beta induces development of the T(H)17 lineage

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

Cutting edge: TGF-beta induces a regulatory phenotype in CD4+CD25- T cells through Foxp3 induction and down-regulation of Smad7

IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

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4295-4306

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10.4049/JIMMUNOL.0903418

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2010-03-19T00:00:00Z

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