Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming. - Wikidata - wikimedia - TriplyDB

Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming.

Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming.

http://www.wikidata.org/entity/Q34998243

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Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis Regulation of Bim in Health and Disease Epstein-Barr virus latent genes The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth EBNA2 Drives Formation of New Chromosome Binding Sites and Target Genes for B-Cell Master Regulatory Transcription Factors RBP-jκ and EBF1 An ensemble of regulatory elements controls Runx3 spatiotemporal expression in subsets of dorsal root ganglia proprioceptive neurons Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection Epstein-Barr virus nuclear antigen 3A protein regulates CDKN2B transcription via interaction with MIZ-1 Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.Epigenetic remodeling in B-cell acute lymphoblastic leukemia occurs in two tracks and employs embryonic stem cell-like signatures.Epstein-Barr virus transcription factor Zta acts through distal regulatory elements to directly control cellular gene expression.EBNA2 binds to genomic intervals associated with multiple sclerosis and overlaps with vitamin D receptor occupancy.Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs.Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.Epstein-Barr virus latency: current and future perspectives.EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases.Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression RUNX super-enhancer control through the Notch pathway by Epstein-Barr virus transcription factors regulates B cell growth.A single amino acid in EBNA-2 determines superior B lymphoblastoid cell line growth maintenance by Epstein-Barr virus type 1 EBNA-2.Regulation of tumour related genes by dynamic epigenetic alteration at enhancer regions in gastric epithelial cells infected by Epstein-Barr virus.Roles of RUNX in B Cell Immortalisation.The Use of Chromatin Precipitation Coupled to DNA Sequencing (ChIP-Seq) for the Analysis of Zta Binding to the Human and EBV Genome.EBF1 binds to EBNA2 and promotes the assembly of EBNA2 chromatin complexes in B cells.EBV and Apoptosis: The Viral Master Regulator of Cell Fate?CRISPR-Cas9 Genetic Analysis of Virus-Host Interactions.Pumilio directs deadenylation-associated translational repression of the cyclin-dependent kinase 1 activator RGC-32.Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.Current Progress in EBV-Associated B-Cell Lymphomas.Epstein-Barr virus nuclear antigen EBNA-LP is essential for transforming naïve B cells, and facilitates recruitment of transcription factors to the viral genome.The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of and the Locus via Interaction with the Histone Lysine Demethylase KDM2B Enhancer control of miR-155 expression in Epstein-Barr virus infected B cells Epstein-Barr Virus-Associated Malignancies: Roles of Viral Oncoproteins in Carcinogenesis

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Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming.

http://www.wikidata.org/entity/Q34998243

description

2013 nî lūn-bûn

@nan

2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

@wuu

name

Modulation of enhancer looping ...... irects cellular reprogramming.

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Modulation of enhancer looping ...... irects cellular reprogramming.

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Modulation of enhancer looping ...... irects cellular reprogramming.

@nl

type

label

Modulation of enhancer looping ...... irects cellular reprogramming.

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Modulation of enhancer looping ...... irects cellular reprogramming.

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Modulation of enhancer looping ...... irects cellular reprogramming.

@nl

prefLabel

Modulation of enhancer looping ...... irects cellular reprogramming.

@ast

Modulation of enhancer looping ...... irects cellular reprogramming.

@en

Modulation of enhancer looping ...... irects cellular reprogramming.

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JOURNAL.PPAT.1003636

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Modulation of enhancer looping ...... irects cellular reprogramming.

@en

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Aaron Arvey

http://www.w3.org/2001/XMLSchema#string

Aditi Kanhere

http://www.w3.org/2001/XMLSchema#string

Bettina Kempkes

http://www.w3.org/2001/XMLSchema#string

C David Wood

http://www.w3.org/2001/XMLSchema#string

Helen M Webb

http://www.w3.org/2001/XMLSchema#string

Marie L Harth-Hertle

http://www.w3.org/2001/XMLSchema#string

Michael J McClellan

http://www.w3.org/2001/XMLSchema#string

Michelle J West

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Opeoluwa Ojeniyi

http://www.w3.org/2001/XMLSchema#string

Richard D Palermo

http://www.w3.org/2001/XMLSchema#string

P2860

Model-based analysis of ChIP-Seq (MACS)

Molecular cloning and characterization of a cellular phosphoprotein that interacts with a conserved C-terminal domain of adenovirus E1A involved in negative modulation of oncogenic transformation

A region in the C-terminus of adenovirus 2/5 E1a protein is required for association with a cellular phosphoprotein and important for the negative modulation of T24-ras mediated transformation, tumorigenesis and metastasis

The Epstein-Barr virus nuclear protein 2 acidic domain forms a complex with a novel cellular coactivator that can interact with TFIIE

Epstein-Barr virus nuclear antigen 3C and prothymosin alpha interact with the p300 transcriptional coactivator at the CH1 and CH3/HAT domains and cooperate in regulation of transcription and histone acetylation.

The 62- and 80-kDa subunits of transcription factor IIH mediate the interaction with Epstein-Barr virus nuclear protein 2

Histone H3K27ac separates active from poised enhancers and predicts developmental state

Epstein-Barr virus nuclear protein 2 (EBNA2) binds to a component of the human SNF-SWI complex, hSNF5/Ini1

Histone modifications at human enhancers reflect global cell-type-specific gene expression

Two nonconsensus sites in the Epstein-Barr virus oncoprotein EBNA3A cooperate to bind the co-repressor carboxyl-terminal-binding protein (CtBP)

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e1003636

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scholarly article

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10.1371/JOURNAL.PPAT.1003636

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9

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9

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Richard G. Jenner

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2013-09-12T00:00:00Z

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257075103

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24068937

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Epstein–Barr virus

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3771879

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