Neuronal activity regulates the regional vulnerability to amyloid-β deposition. - Wikidata - wikimedia - TriplyDB

Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

http://www.wikidata.org/entity/Q35009626

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Alzheimer mechanisms and therapeutic strategies Lysosomal sorting of amyloid-β by the SORLA receptor is impaired by a familial Alzheimer's disease mutation Amyloid-β oligomers induce synaptic damage via Tau-dependent microtubule severing by TTLL6 and spastin Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injury Inhibitory interneuron deficit links altered network activity and cognitive dysfunction in Alzheimer model.Meditation experience is associated with differences in default mode network activity and connectivity Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model.A paravascular pathway facilitates CSF flow through the brain parenchyma and the clearance of interstitial solutes, including amyloid β.Calcium-Sensing Receptors of Human Neural Cells Play Crucial Roles in Alzheimer's Disease Candidate mechanisms underlying the association between sleep-wake disruptions and Alzheimer's disease Connectivity of Pathology: The Olfactory System as a Model for Network-Driven Mechanisms of Alzheimer's Disease Pathogenesis Gene expression parallels synaptic excitability and plasticity changes in Alzheimer's disease A genetic cause of Alzheimer disease: mechanistic insights from Down syndrome Clearance systems in the brain-implications for Alzheimer disease Shift in brain metabolism in late onset Alzheimer's disease: implications for biomarkers and therapeutic interventions Explaining the encoding/retrieval flip: memory-related deactivations and activations in the posteromedial cortex Network dysfunction in Alzheimer's disease: refining the disconnection hypothesis Self-propagation of pathogenic protein aggregates in neurodegenerative diseases A brief history of the resting state: the Washington University perspective Neurodegenerative diseases: expanding the prion concept Sleep, circadian rhythms, and the pathogenesis of Alzheimer disease The multifaceted nature of amyloid precursor protein and its proteolytic fragments: friends and foes Simulated cytoskeletal collapse via tau degradation Genome-wide analysis of miRNA signature in the APPswe/PS1ΔE9 mouse model of alzheimer's disease Interictal spikes during sleep are an early defect in the Tg2576 mouse model of β-amyloid neuropathology Three dimensions of the amyloid hypothesis: time, space and 'wingmen'On the central role of brain connectivity in neurodegenerative disease progression The Glymphatic System: A Beginner's Guide Stem cell therapy for Alzheimer's disease and related disorders: current status and future perspectives Preventing Alzheimer's disease by means of natural selection Identification of the novel activity-driven interaction between synaptotagmin 1 and presenilin 1 links calcium, synapse, and amyloid beta Brain pyroglutamate amyloid-β is produced by cathepsin B and is reduced by the cysteine protease inhibitor E64d, representing a potential Alzheimer's disease therapeutic Apolipoprotein E and Alzheimer disease: risk, mechanisms and therapy Molecular mechanisms of cognitive dysfunction following traumatic brain injury Tracking pathophysiological processes in Alzheimer's disease: an updated hypothetical model of dynamic biomarkers Effects of CD2-associated protein deficiency on amyloid-β in neuroblastoma cells and in an APP transgenic mouse model.The effect of PN-1, a Traditional Chinese Prescription, on the Learning and Memory in a Transgenic Mouse Model of Alzheimer's Disease.Self-reported sleep and β-amyloid deposition in community-dwelling older adults.Origins of Alzheimer's disease: reconciling cerebrospinal fluid biomarker and neuropathology data regarding the temporal sequence of amyloid-beta and tau involvement.Cognitive decline and brain pathology in aging--need for a dimensional, lifespan and systems vulnerability view.

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年论文

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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Neuronal activity regulates the regional vulnerability to amyloid-β deposition.

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P2093

Adam W Bero

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Floy R Stewart

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Jee Hoon Roh

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Jin-Moo Lee

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John R Cirrito

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Ping Yan

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P2860

Beta-secretase cleavage of Alzheimer's amyloid precursor protein by the transmembrane aspartic protease BACE

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

A default mode of brain function

Spatial correlation between brain aerobic glycolysis and amyloid-β (Aβ ) deposition

Regional aerobic glycolysis in the human brain

Amyloid-beta dynamics are regulated by orexin and the sleep-wake cycle

The brain's default network: anatomy, function, and relevance to disease

Alzheimer's disease: genes, proteins, and therapy

Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase

Exogenous induction of cerebral beta-amyloidogenesis is governed by agent and host

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10.1038/NN.2801

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6

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14

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David M. Holtzman

Marcus E. Raichle

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2011-05-01T00:00:00Z

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51088786

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3102784

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