Intracellular signals mediating the food intake-suppressive effects of hindbrain glucagon-like peptide-1 receptor activation - Wikidata - wikimedia - TriplyDB

Intracellular signals mediating the food intake-suppressive effects of hindbrain glucagon-like peptide-1 receptor activation

Intracellular signals mediating the food intake-suppressive effects of hindbrain glucagon-like peptide-1 receptor activation

http://www.wikidata.org/entity/Q35024826

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Potentials of incretin-based therapies in dementia and stroke in type 2 diabetes mellitus Brain regulation of energy balance and body weight.Physiology of proglucagon peptides: role of glucagon and GLP-1 in health and disease Glucagon-Like Peptide-1 and Its Class B G Protein-Coupled Receptors: A Long March to Therapeutic Successes Glucagon-like peptide 1 interacts with ghrelin and leptin to regulate glucose metabolism and food intake through vagal afferent neuron signaling The anorectic effect of GLP-1 in rats is nutrient dependent The arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight loss.Liraglutide and obesity: a review of the data so far.Glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1: Incretin actions beyond the pancreas.Hindbrain nucleus tractus solitarius glucagon-like peptide-1 receptor signaling reduces appetitive and motivational aspects of feeding.The CNS glucagon-like peptide-2 receptor in the control of energy balance and glucose homeostasis Dynamic and extensive metabolic state-dependent regulation of cytokine expression and circulating levels Liraglutide, leptin and their combined effects on feeding: additive intake reduction through common intracellular signalling mechanisms.Genome-wide association and systems genetic analyses of residual feed intake, daily feed consumption, backfat and weight gain in pigs GLP-1 based therapeutics: simultaneously combating T2DM and obesity.Opioidergic consequences of dietary-induced binge eating.Jejunal linoleic acid infusions require GLP-1 receptor signaling to inhibit food intake: implications for the effectiveness of Roux-en-Y gastric bypass.Glucagon-like peptide-1 receptor agonists suppress water intake independent of effects on food intake.Hippocampal GLP-1 receptors influence food intake, meal size, and effort-based responding for food through volume transmission Incretins and amylin: neuroendocrine communication between the gut, pancreas, and brain in control of food intake and blood glucose.GLP-1 neurons in the nucleus of the solitary tract project directly to the ventral tegmental area and nucleus accumbens to control for food intake.Brain GLP-1 and insulin sensitivity.Neuroprotective and neurotrophic actions of glucagon-like peptide-1: an emerging opportunity to treat neurodegenerative and cerebrovascular disorders Endogenous leptin receptor signaling in the medial nucleus tractus solitarius affects meal size and potentiates intestinal satiation signals.Brainstem nutrient sensing in the nucleus of the solitary tract inhibits feeding.Astrocytes Regulate GLP-1 Receptor-Mediated Effects on Energy Balance.Activation of GLP-1 Receptor Promotes Bone Marrow Stromal Cell Osteogenic Differentiation through β-Catenin High-fat diet changes the temporal profile of GLP-1 receptor-mediated hypophagia in rats.GLP-1R agonism enhances adjustable gastric banding in diet-induced obese rats Hindbrain GLP-1 receptor-mediated suppression of food intake requires a PI3K-dependent decrease in phosphorylation of membrane-bound Akt.Glucagon-like peptide-1 receptor agonist administration suppresses both water and saline intake in rats.The anti-hyperglycemic efficacy of a lipid-lowering drug Daming capsule and the underlying signaling mechanisms in a rat model of diabetes mellitus.Leptin signaling in the medial nucleus tractus solitarius reduces food seeking and willingness to work for food.Sugar for the brain: the role of glucose in physiological and pathological brain function C1q/TNF-related protein 4 (CTRP4) is a unique secreted protein with two tandem C1q domains that functions in the hypothalamus to modulate food intake and body weight.Neuronal and intracellular signaling pathways mediating GLP-1 energy balance and glycemic effects.Non-glycaemic effects mediated via GLP-1 receptor agonists and the potential for exploiting these for therapeutic benefit: focus on liraglutide.Current and emerging concepts on the role of peripheral signals in the control of food intake and development of obesity.Update on incretin hormones.Hindbrain neurons as an essential hub in the neuroanatomically distributed control of energy balance.

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Intracellular signals mediating the food intake-suppressive effects of hindbrain glucagon-like peptide-1 receptor activation

http://www.wikidata.org/entity/Q35024826

description

2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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Intracellular signals mediatin ...... peptide-1 receptor activation

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Intracellular signals mediatin ...... peptide-1 receptor activation

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Intracellular signals mediatin ...... peptide-1 receptor activation

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J.CMET.2011.02.001

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Cell Metabolism

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Intracellular signals mediatin ...... peptide-1 receptor activation

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Amy Chowansky

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Bart C De Jonghe

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Derek Zimmer

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Grace S Lee

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Harvey J Grill

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Kendra K Bence

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Matthew R Hayes

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Scott E Kanoski

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Shiru Zhao

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Theresa M Leichner

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P2860

A role for glucagon-like peptide-1 in the central regulation of feeding

The diverse roles of specific GLP-1 receptors in the control of food intake and the response to visceral illness

C75, a fatty acid synthase inhibitor, reduces food intake via hypothalamic AMP-activated protein kinase

AMP-kinase regulates food intake by responding to hormonal and nutrient signals in the hypothalamus

The Physiology of Glucagon-like Peptide 1

Intracellular signalling. Key enzyme in leptin-induced anorexia

cAMP activates MAP kinase and Elk-1 through a B-Raf- and Rap1-dependent pathway

Constitutive activation of Galphas within forebrain neurons causes deficits in sensorimotor gating because of PKA-dependent decreases in cAMP.

The action of leptin in the ventral tegmental area to decrease food intake is dependent on Jak-2 signaling

Endogenous leptin signaling in the caudal nucleus tractus solitarius and area postrema is required for energy balance regulation.

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320-330

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10.1016/J.CMET.2011.02.001

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3

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21356521

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