Mammalian target of rapamycin complex 2 regulates invariant NKT cell development and function independent of promyelocytic leukemia zinc-finger. - Wikidata - wikimedia - TriplyDB

Mammalian target of rapamycin complex 2 regulates invariant NKT cell development and function independent of promyelocytic leukemia zinc-finger.

Mammalian target of rapamycin complex 2 regulates invariant NKT cell development and function independent of promyelocytic leukemia zinc-finger.

http://www.wikidata.org/entity/Q35043677

about

mTOR Regulation of Lymphoid Cells in Immunity to Pathogens Antitumor Responses of Invariant Natural Killer T Cells Diacylglycerol Kinases in T Cell Tolerance and Effector Function Development of invariant natural killer T cells.mTOR Links Environmental Signals to T Cell Fate Decisions.mTOR and metabolic regulation of conventional and regulatory T cells.Pak2 Controls Acquisition of NKT Cell Fate by Regulating Expression of the Transcription Factors PLZF and Egr2.mTORC2 in Thymic Epithelial Cells Controls Thymopoiesis and T Cell Development.mTOR is critical for intestinal T-cell homeostasis and resistance to Citrobacter rodentium.Critical roles of mTOR Complex 1 and 2 for T follicular helper cell differentiation and germinal center responses.Redundant and specialized roles for diacylglycerol kinases α and ζ in the control of T cell functions.Regulation of NKT Cell Localization in Homeostasis and Infection mTOR and its tight regulation for iNKT cell development and effector function.Lactic acid in tumor microenvironments causes dysfunction of NKT cells by interfering with mTOR signaling.Roles of mTOR complexes in the kidney: implications for renal disease and transplantation.mTORC2 regulates multiple aspects of NKT-cell development and function.MenTORing Immunity: mTOR Signaling in the Development and Function of Tissue-Resident Immune Cells.The differentiation of ROR-γt expressing iNKT17 cells is orchestrated by Runx1.NKAP Regulates Invariant NKT Cell Proliferation and Differentiation into ROR-γt-Expressing NKT17 Cells.Reactive Oxygen Species Regulate the Inflammatory Function of NKT Cells through Promyelocytic Leukemia Zinc Finger.Unraveling Natural Killer T-Cells Development.Applying the TOR(C)QUE in iNKT cells: A new twist in an old tale.Temporal regulation of Wnt/β-catenin signaling is important for invariant NKT cell development and terminal maturation.The role of mTOR-mediated signals during haemopoiesis and lineage commitment

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P2860

Mammalian target of rapamycin complex 2 regulates invariant NKT cell development and function independent of promyelocytic leukemia zinc-finger.

http://www.wikidata.org/entity/Q35043677

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Mammalian target of rapamycin ...... elocytic leukemia zinc-finger.

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type

label

Mammalian target of rapamycin ...... elocytic leukemia zinc-finger.

@en

prefLabel

Mammalian target of rapamycin ...... elocytic leukemia zinc-finger.

@en

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P356

JIMMUNOL.1401985

P1433

Journal of Immunology

P1476

Mammalian target of rapamycin ...... elocytic leukemia zinc-finger.

@en

P2093

Cheong-Hee Chang

http://www.w3.org/2001/XMLSchema#string

Evan Bischoff

http://www.w3.org/2001/XMLSchema#string

Jonathan D Powell

http://www.w3.org/2001/XMLSchema#string

Jyoti Misra Sen

http://www.w3.org/2001/XMLSchema#string

Kalyani Pyaram

http://www.w3.org/2001/XMLSchema#string

Nicolas Prevot

http://www.w3.org/2001/XMLSchema#string

P2860

Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive

The BTB-zinc finger transcriptional regulator PLZF controls the development of invariant natural killer T cell effector functions

The transcription factor PLZF directs the effector program of the NKT cell lineage

The biology of NKT cells

ICOS/ICOSL interaction is required for CD4+ invariant NKT cell function and homeostatic survival.

Temporal differences in the dependency on phosphoinositide-dependent kinase 1 distinguish the development of invariant Valpha14 NKT cells and conventional T cells

The kinase mTOR regulates the differentiation of helper T cells through the selective activation of signaling by mTORC1 and mTORC2.

A central role for mTOR kinase in homeostatic proliferation induced CD8+ T cell memory and tumor immunity

The tumor suppressor Tsc1 enforces quiescence of naive T cells to promote immune homeostasis and function.

Regulation and function of mTOR signalling in T cell fate decisions.

P304

223-230

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scholarly article

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10.4049/JIMMUNOL.1401985

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1

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194

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2014-11-17T00:00:00Z

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25404366

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P932

4315517

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