Neuroinflammation and α-synuclein dysfunction potentiate each other, driving chronic progression of neurodegeneration in a mouse model of Parkinson's disease.
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The role of oxidative stress in Parkinson's diseaseBrain-gut-microbiota axis in Parkinson's diseaseInflammation and α-synuclein's prion-like behavior in Parkinson's disease--is there a link?Neuroimmunological processes in Parkinson's disease and their relation to α-synuclein: microglia as the referee between neuronal processes and peripheral immunityPyrrolidine Dithiocarbamate Prevents Neuroinflammation and Cognitive Dysfunction after Endotoxemia in Rats.Gene-environment interactions: key to unraveling the mystery of Parkinson's diseaseGDF15/MIC1 and MMP9 Cerebrospinal Fluid Levels in Parkinson's Disease and Lewy Body DementiaTNFα and IL-1β are mediated by both TLR4 and Nod1 pathways in the cultured HAPI cells stimulated by LPSConsiderations on the role of environmental toxins in idiopathic Parkinson's disease pathophysiologyAdulthood Exposure to Lipopolysaccharide Exacerbates the Neurotoxic and Inflammatory Effects of Rotenone in the Substantia NigraTargeting Wnt signaling at the neuroimmune interface for dopaminergic neuroprotection/repair in Parkinson's disease.Sigma-1 receptor chaperones in neurodegenerative and psychiatric disordersIncreased intestinal permeability correlates with sigmoid mucosa alpha-synuclein staining and endotoxin exposure markers in early Parkinson's disease.Substance P exacerbates dopaminergic neurodegeneration through neurokinin-1 receptor-independent activation of microglial NADPH oxidaseRole of oxidative stress in Parkinson's disease.Inflammatory dysregulation of blood monocytes in Parkinson's disease patients.Imaging neuroinflammation in gray and white matter in schizophrenia: an in-vivo PET study with [18F]-FEPPA.Dopaminergic neurotoxicant 6-OHDA induces oxidative damage through proteolytic activation of PKCδ in cell culture and animal models of Parkinson's diseaseA novel role of microglial NADPH oxidase in mediating extra-synaptic function of norepinephrine in regulating brain immune homeostasis.Post-treatment with an ultra-low dose of NADPH oxidase inhibitor diphenyleneiodonium attenuates disease progression in multiple Parkinson's disease modelsThe acute inflammatory response to intranigral α-synuclein differs significantly from intranigral lipopolysaccharide and is exacerbated by peripheral inflammation.Systemic inflammation and microglial activation: systematic review of animal experimentsNeuroinflammation in Parkinson's Disease and Related Disorders: A Lesson from Genetically Manipulated Mouse Models of α-Synucleinopathies.Studies of lipopolysaccharide effects on the induction of α-synuclein pathology by exogenous fibrils in transgenic mice.Reactive oxygen species and inhibitors of inflammatory enzymes, NADPH oxidase, and iNOS in experimental models of Parkinson's disease.Distinct clinical and neuropathological features of G51D SNCA mutation cases compared with SNCA duplication and H50Q mutationGut Microbiota Regulate Motor Deficits and Neuroinflammation in a Model of Parkinson's Disease.NADPH oxidases: novel therapeutic targets for neurodegenerative diseasesHypoestoxide reduces neuroinflammation and α-synuclein accumulation in a mouse model of Parkinson's diseaseUpregulation of α-synuclein during localized radiation therapy signals the association of cancer-related fatigue with the activation of inflammatory and neuroprotective pathways.Altered Expression Patterns of Inflammation-Associated and Trophic Molecules in Substantia Nigra and Striatum Brain Samples from Parkinson's Disease, Incidental Lewy Body Disease and Normal Control Cases.Assessment of Radiation Induced Therapeutic Effect and Cytotoxicity in Cancer Patients Based on Transcriptomic Profiling.Thinking laterally about neurodegenerative proteinopathies.Celecoxib reduces brain dopaminergic neuronaldysfunction, and improves sensorimotor behavioral performance in neonatal rats exposed to systemic lipopolysaccharide.Interplay of endoplasmic reticulum stress and autophagy in neurodegenerative disorders.Amyloidogenic α-synuclein seeds do not invariably induce rapid, widespread pathology in mice.Neonatal exposure to lipopolysaccharide enhances accumulation of α-synuclein aggregation and dopamine transporter protein expression in the substantia nigra in responses to rotenone challenge in later lifeMaternal exposure to mercury chloride during pregnancy and lactation affects the immunity and social behavior of offspringParkinson's disease and enhanced inflammatory response.Direct detection of alpha synuclein oligomers in vivo
P2860
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P2860
Neuroinflammation and α-synuclein dysfunction potentiate each other, driving chronic progression of neurodegeneration in a mouse model of Parkinson's disease.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Neuroinflammation and α-synucl ...... model of Parkinson's disease.
@en
type
label
Neuroinflammation and α-synucl ...... model of Parkinson's disease.
@en
prefLabel
Neuroinflammation and α-synucl ...... model of Parkinson's disease.
@en
P2093
P2860
P921
P356
P1476
Neuroinflammation and α-synucl ...... e model of Parkinson's disease
@en
P2093
Belinda Wilson
Feng Zhang
Hui-Ming Gao
Wayneho Kam
P2860
P304
P356
10.1289/EHP.1003013
P577
2011-01-18T00:00:00Z