ICP22 is required for wild-type composition and infectivity of herpes simplex virus type 1 virions. - Wikidata - wikimedia - TriplyDB

ICP22 is required for wild-type composition and infectivity of herpes simplex virus type 1 virions.

ICP22 is required for wild-type composition and infectivity of herpes simplex virus type 1 virions.

http://www.wikidata.org/entity/Q35101439

about

Herpes simplex virus type 1 immediate-early protein ICP27 is required for efficient incorporation of ICP0 and ICP4 into virions Herpes simplex virus type 1 immediate-early protein ICP22 is required for VICE domain formation during productive viral infection.Genome-wide screen of three herpesviruses for protein subcellular localization and alteration of PML nuclear bodies Role of nuclear factor Y in stress-induced activation of the herpes simplex virus type 1 ICP0 promoter Herpes simplex virus tegument ICP0 is capsid associated, and its E3 ubiquitin ligase domain is important for incorporation into virions.Role of herpes simplex virus 1 immediate early protein ICP22 in viral nuclear egress.Comprehensive characterization of interaction complexes of herpes simplex virus type 1 ICP22, UL3, UL4, and UL20.5.Proteomic characterization of pseudorabies virus extracellular virions Sequence variation in the herpes simplex virus U(S)1 ocular virulence determinant.Herpes Simplex Virus 1 (HSV-1) ICP22 protein directly interacts with cyclin-dependent kinase (CDK)9 to inhibit RNA polymerase II transcription elongation Herpes simplex virus immediate-early protein ICP22 triggers loss of serine 2-phosphorylated RNA polymerase II Probing the nuclear import signal and nuclear transport molecular determinants of PRV ICP22 The IR4 auxiliary regulatory protein expands the in vitro host range of equine herpesvirus 1 and is essential for pathogenesis in the murine model.Transient expression of herpes simplex virus type 1 ICP22 represses viral promoter activity and complements the replication of an ICP22 null virus.Origin of expression of the herpes simplex virus type 1 protein U(S)1.5.Herpes simplex virus 1 ICP22 but not US 1.5 is required for efficient acute replication in mice and VICE domain formation.HSV-1 ICP22: hijacking host nuclear functions to enhance viral infection.Herpes simplex virus type 1 virion-derived US11 inhibits type 1 interferon-induced protein kinase R phosphorylation.Identification of sequences in herpes simplex virus type 1 ICP22 that influence RNA polymerase II modification and viral late gene expression.A Herpesviral Immediate Early Protein Promotes Transcription Elongation of Viral Transcripts.Characterization of synonymous codon usage bias in the pseudorabies virus US1 gene.

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P2860

ICP22 is required for wild-type composition and infectivity of herpes simplex virus type 1 virions.

http://www.wikidata.org/entity/Q35101439

description

2006 nî lūn-bûn

@nan

2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2006年の論文

@ja

2006年論文

@yue

2006年論文

@zh-hant

2006年論文

@zh-hk

2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

name

ICP22 is required for wild-typ ...... simplex virus type 1 virions.

@ast

ICP22 is required for wild-typ ...... simplex virus type 1 virions.

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type

label

ICP22 is required for wild-typ ...... simplex virus type 1 virions.

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ICP22 is required for wild-typ ...... simplex virus type 1 virions.

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prefLabel

ICP22 is required for wild-typ ...... simplex virus type 1 virions.

@ast

ICP22 is required for wild-typ ...... simplex virus type 1 virions.

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P1433

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Journal of Virology

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ICP22 is required for wild-typ ...... simplex virus type 1 virions.

@en

P2093

Anna S Kushnir

http://www.w3.org/2001/XMLSchema#string

David M Knipe

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John W Balliet

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Joseph S Orlando

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Magdalena Kosz-Vnenchak

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Priscilla A Schaffer

http://www.w3.org/2001/XMLSchema#string

Stephen A Rice

http://www.w3.org/2001/XMLSchema#string

Todd L Astor

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P2860

Herpes simplex virus 1 regulatory protein ICP22 interacts with a new cell cycle-regulated factor and accumulates in a cell cycle-dependent fashion in infected cells.

A novel cellular protein, p60, interacting with both herpes simplex virus 1 regulatory proteins ICP22 and ICP0 is modified in a cell-type-specific manner and Is recruited to the nucleus after infection

Functional anatomy of herpes simplex virus 1 overlapping genes encoding infected-cell protein 22 and US1.5 protein

Herpes simplex virus 1 mutant deleted in the alpha 22 gene: growth and gene expression in permissive and restrictive cells and establishment of latency in mice

Distinct monoclonal antibodies separately label the hexons or the pentons of herpes simplex virus capsid.

The latent herpes simplex virus type 1 genome copy number in individual neurons is virus strain specific and correlates with reactivation

Herpes simplex virus type 1 glycoprotein gC mediates immune evasion in vivo

The disappearance of cyclins A and B and the increase in activity of the G(2)/M-phase cellular kinase cdc2 in herpes simplex virus 1-infected cells require expression of the alpha22/U(S)1.5 and U(L)13 viral genes.

Optimized viral dose and transient immunosuppression enable herpes simplex virus ICP0-null mutants To establish wild-type levels of latency in vivo.

ICP0 is required for efficient reactivation of herpes simplex virus type 1 from neuronal latency.

P304

9381-9390

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scholarly article

P356

10.1128/JVI.01061-06

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19

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80

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2006-10-01T00:00:00Z

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16973544

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1617265

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