Transient expression of herpes simplex virus type 1 ICP22 represses viral promoter activity and complements the replication of an ICP22 null virus. - Wikidata - wikimedia - TriplyDB

Transient expression of herpes simplex virus type 1 ICP22 represses viral promoter activity and complements the replication of an ICP22 null virus.

Transient expression of herpes simplex virus type 1 ICP22 represses viral promoter activity and complements the replication of an ICP22 null virus.

http://www.wikidata.org/entity/Q37333707

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Herpes simplex virus 1 ICP22 inhibits the transcription of viral gene promoters by binding to and blocking the recruitment of P-TEFb Comprehensive characterization of interaction complexes of herpes simplex virus type 1 ICP22, UL3, UL4, and UL20.5.The ICP22 protein selectively modifies the transcription of different kinetic classes of pseudorabies virus genes.Proteomic characterization of pseudorabies virus extracellular virions Sequence variation in the herpes simplex virus U(S)1 ocular virulence determinant.Herpes Simplex Virus 1 (HSV-1) ICP22 protein directly interacts with cyclin-dependent kinase (CDK)9 to inhibit RNA polymerase II transcription elongation Probing the nuclear import signal and nuclear transport molecular determinants of PRV ICP22 Origin of expression of the herpes simplex virus type 1 protein U(S)1.5.Herpes simplex virus 1 ICP22 but not US 1.5 is required for efficient acute replication in mice and VICE domain formation.HSV-1 ICP22: hijacking host nuclear functions to enhance viral infection.Characterization of synonymous codon usage bias in the pseudorabies virus US1 gene.

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Transient expression of herpes simplex virus type 1 ICP22 represses viral promoter activity and complements the replication of an ICP22 null virus.

http://www.wikidata.org/entity/Q37333707

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 17 June 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Transient expression of herpes ...... cation of an ICP22 null virus.

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Transient expression of herpes ...... cation of an ICP22 null virus.

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type

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Transient expression of herpes ...... cation of an ICP22 null virus.

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Transient expression of herpes ...... cation of an ICP22 null virus.

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prefLabel

Transient expression of herpes ...... cation of an ICP22 null virus.

@en

Transient expression of herpes ...... cation of an ICP22 null virus.

@nl

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Journal of Virology

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Transient expression of herpes ...... cation of an ICP22 null virus.

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Anna S Kushnir

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David J Davido

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J Jason Bowman

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Joseph S Orlando

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Priscilla A Schaffer

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P2860

Herpes simplex virus 1 mutant deleted in the alpha 22 gene: growth and gene expression in permissive and restrictive cells and establishment of latency in mice

ICP22 and the UL13 protein kinase are both required for herpes simplex virus-induced modification of the large subunit of RNA polymerase II.

Site-directed mutagenesis of large DNA palindromes: construction and in vitro characterization of herpes simplex virus type 1 mutants containing point mutations that eliminate the oriL or oriS initiation function

A generalized technique for deletion of specific genes in large genomes: alpha gene 22 of herpes simplex virus 1 is not essential for growth.

ICP22 is required for wild-type composition and infectivity of herpes simplex virus type 1 virions.

Herpes simplex virus immediate-early protein ICP22 is required for viral modification of host RNA polymerase II and establishment of the normal viral transcription program.

Herpes simplex virus immediate-early protein ICP22 triggers loss of serine 2-phosphorylated RNA polymerase II

Processing of the herpes simplex virus regulatory protein alpha 22 mediated by the UL13 protein kinase determines the accumulation of a subset of alpha and gamma mRNAs and proteins in infected cells.

Establishment of latency in mice by herpes simplex virus 1 recombinants that carry insertions affecting regulation of the thymidine kinase gene.

Activation of the thymidine kinase promoter by herpes simplex virus type 1 immediate early proteins.

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8733-8743

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scholarly article

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10.1128/JVI.00810-09

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17

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83

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2009-06-17T00:00:00Z

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26300434

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19535441

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2738139

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