The host response to West Nile Virus infection limits viral spread through the activation of the interferon regulatory factor 3 pathway.
about
Rotavirus nonstructural protein 1 subverts innate immune response by inducing degradation of IFN regulatory factor 3Regulating intracellular antiviral defense and permissiveness to hepatitis C virus RNA replication through a cellular RNA helicase, RIG-IWest Nile Virus: biology, transmission, and human infectionAssembly and release of infectious hepatitis C virus involving unusual organization of the secretory pathwayThe Src Family Kinase c-Yes Is Required for Maturation of West Nile Virus ParticlesAlpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survivalA Single Amino Acid Substitution in the West Nile Virus Nonstructural Protein NS2A Disables Its Ability To Inhibit Alpha/Beta Interferon Induction and Attenuates Virus Virulence in MiceRNase L plays a role in the antiviral response to West Nile virus.West Nile virus evades activation of interferon regulatory factor 3 through RIG-I-dependent and -independent pathways without antagonizing host defense signaling.PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in NeuronsResistance to Alpha/Beta Interferon Is a Determinant of West Nile Virus Replication Fitness and VirulencePathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral EvasionInhibition of apoptosis prevents West Nile virus induced cell deathCell-Specific IRF-3 Responses Protect against West Nile Virus Infection by Interferon-Dependent and -Independent MechanismsTranscriptional Activation of Interferon-Stimulated Genes but Not of Cytokine Genes after Primary Infection of Rhesus Macaques with Dengue Virus Type 1West Nile Virus-Induced Interferon Production Is Mediated by the Double-Stranded RNA-Dependent Protein Kinase PKRIdentification of Novel Small-Molecule Inhibitors of West Nile Virus InfectionDifferential Expression of Interferon (IFN) Regulatory Factors and IFN-Stimulated Genes at Early Times after West Nile Virus Infection of Mouse Embryo FibroblastsDifferential Effects of Mutations in NS4B on West Nile Virus Replication and Inhibition of Interferon SignalingDifferential Activation of Human Monocyte-Derived and Plasmacytoid Dendritic Cells by West Nile Virus Generated in Different Host CellsEstablishment and Maintenance of the Innate Antiviral Response to West Nile Virus Involves both RIG-I and MDA5 Signaling through IPS-1Interferon Regulatory Factor IRF-7 Induces the Antiviral Alpha Interferon Response and Protects against Lethal West Nile Virus InfectionThe structural immunology of antibody protection against West Nile virusInnate immune evasion by hepatitis C virus and West Nile virusProgress on the development of therapeutics against West Nile virusIPS-1 Is Essential for the Control of West Nile Virus Infection and ImmunityThe 2',5'-oligoadenylate synthetase 1b is a potent inhibitor of West Nile virus replication inside infected cellsRNA interference screen for human genes associated with West Nile virus infectionInhibition of interferon signaling by the New York 99 strain and Kunjin subtype of West Nile virus involves blockage of STAT1 and STAT2 activation by nonstructural proteinsWest Nile virus inhibits the signal transduction pathway of alpha interferon.Modulation of innate immune signaling by the secreted form of the West Nile virus NS1 glycoproteinA new model to produce infectious hepatitis C virus without the replication requirement.Host genetic risk factors for West Nile virus infection and disease progression.Environmental and biological factors influencing Culex pipiens quinquefasciatus (Diptera: Culicidae) vector competence for West Nile Virus.West Nile virus and its emergence in the United States of America.Identification of five interferon-induced cellular proteins that inhibit west nile virus and dengue virus infections.Herpes simplex virus 1 counteracts viperin via its virion host shutoff protein UL412'-O methylation of the viral mRNA cap by West Nile virus evades ifit1-dependent and -independent mechanisms of host restriction in vivoMultiplexed digital mRNA profiling of the inflammatory response in the West Nile Swiss Webster mouse modelIRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling
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The host response to West Nile Virus infection limits viral spread through the activation of the interferon regulatory factor 3 pathway.
description
2004 nî lūn-bûn
@nan
2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
The host response to West Nile ...... n regulatory factor 3 pathway.
@ast
The host response to West Nile ...... n regulatory factor 3 pathway.
@en
type
label
The host response to West Nile ...... n regulatory factor 3 pathway.
@ast
The host response to West Nile ...... n regulatory factor 3 pathway.
@en
prefLabel
The host response to West Nile ...... n regulatory factor 3 pathway.
@ast
The host response to West Nile ...... n regulatory factor 3 pathway.
@en
P2860
P50
P1433
P1476
The host response to West Nile ...... n regulatory factor 3 pathway.
@en
P2093
Brenda L Fredericksen
P2860
P304
P356
10.1128/JVI.78.14.7737-7747.2004
P407
P577
2004-07-01T00:00:00Z