Apoptosis inhibitor of macrophage (AIM) is required for obesity-associated recruitment of inflammatory macrophages into adipose tissue. - Wikidata - wikimedia - TriplyDB

Apoptosis inhibitor of macrophage (AIM) is required for obesity-associated recruitment of inflammatory macrophages into adipose tissue.

Apoptosis inhibitor of macrophage (AIM) is required for obesity-associated recruitment of inflammatory macrophages into adipose tissue.

http://www.wikidata.org/entity/Q35123208

about

The immune system's involvement in obesity-driven type 2 diabetes Macrophage plasticity and polarization: in vivo veritas Cellular and molecular players in adipose tissue inflammation in the development of obesity-induced insulin resistance The scavenger protein apoptosis inhibitor of macrophages (AIM) potentiates the antimicrobial response against Mycobacterium tuberculosis by enhancing autophagy.Stabilization and augmentation of circulating AIM in mice by synthesized IgM-Fc.What causes the insulin resistance underlying obesity?Inflammatory and anti-inflammatory states of adipose tissue in transgenic mice bearing a single T-cell receptor.Verification of B-lymphocyte activating factor's involvement in the exacerbation of insulin resistance as well as an autoimmune response in patients with nonalcoholic steatohepatitis and patients with HCV-related chronic liver disease Adipose tissue macrophages in the development of obesity-induced inflammation, insulin resistance and type 2 diabetes.Relationship of gene expression in the injured human meniscus to body mass index: a biologic connection between obesity and osteoarthritis.The human CD5L/AIM-CD36 axis: A novel autophagy inducer in macrophages that modulates inflammatory responses.Inflammation and oxidative stress in obesity-related glomerulopathy Macrophage-mediated inflammation in metabolic disease Making many from few: IL-12p40 as a model for the combinatorial assembly of heterodimeric cytokines.Molecular targets related to inflammation and insulin resistance and potential interventions The utility of apoptosis inhibitor of macrophages as a possible diagnostic marker in patients with Crohn's disease.'Metabolically healthy obesity': origins and implications.LXR activation by GW3965 alters fat tissue distribution and adipose tissue inflammation in ob/ob female mice.Alternatively activated macrophages determine repair of the infarcted adult murine heart.MafB deficiency accelerates the development of obesity in mice Stress in obesity and associated metabolic and cardiovascular disorders.Impact of feline AIM on the susceptibility of cats to renal disease.A proteolytic modification of AIM promotes its renal excretion.Serum levels of apoptosis inhibitor of macrophage are associated with hepatic fibrosis in patients with chronic hepatitis C.Biological roles of liver X receptors in immune cells.Impacts of the apoptosis inhibitor of macrophage (AIM) on obesity-associated inflammatory diseases.AIM/CD5L: a key protein in the control of immune homeostasis and inflammatory disease.Hemodynamics as Measured With Color Doppler Sonography in Early-Stage Obesity-Related Nephropathy in Children.Apoptosis inhibitor of macrophage protein enhances intraluminal debris clearance and ameliorates acute kidney injury in mice.Deletion of Apoptosis Inhibitor of Macrophage (AIM)/CD5L Attenuates the Inflammatory Response and Infarct Size in Acute Myocardial Infarction.Molecular cloning and gene expression of canine apoptosis inhibitor of macrophage.Apoptosis inhibitor of macrophage depletion decreased M1 macrophage accumulation and the incidence of cardiac rupture after myocardial infarction in mice.Macrophage Lamin A/C Regulates Inflammation and the Development of Obesity-Induced Insulin Resistance.Independent modes of disease repair by AIM protein distinguished in AIM-felinized mice

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Apoptosis inhibitor of macrophage (AIM) is required for obesity-associated recruitment of inflammatory macrophages into adipose tissue.

http://www.wikidata.org/entity/Q35123208

description

2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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PNAS.1101841108

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Proceedings of the National Academy of Sciences of the United States of America

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Apoptosis inhibitor of macroph ...... crophages into adipose tissue.

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Hiromichi Nagano

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Jun Kurokawa

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Naoto Kubota

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Satoko Arai

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Takashi Kadowaki

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Toru Miyazaki

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P2860

Inflammation and insulin resistance

Molecular cloning, mapping to human chromosome 1 q21-q23, and cell binding characteristics of Spalpha, a new member of the scavenger receptor cysteine-rich (SRCR) family of proteins

Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance

Obesity is associated with macrophage accumulation in adipose tissue

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

JNK1 in hematopoietically derived cells contributes to diet-induced inflammation and insulin resistance without affecting obesity

Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes

Monocyte and macrophage heterogeneity

Obesity induces a phenotypic switch in adipose tissue macrophage polarization

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10.1073/PNAS.1101841108

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29

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108

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51468213

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21730133

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apoptotic process

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3141977

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