Anti-nerve growth factor Ab abrogates macrophage-mediated HIV-1 infection and depletion of CD4+ T lymphocytes in hu-SCID mice - Wikidata - wikimedia - TriplyDB

Anti-nerve growth factor Ab abrogates macrophage-mediated HIV-1 infection and depletion of CD4+ T lymphocytes in hu-SCID mice

Anti-nerve growth factor Ab abrogates macrophage-mediated HIV-1 infection and depletion of CD4+ T lymphocytes in hu-SCID mice

http://www.wikidata.org/entity/Q35171217

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The engagement of activating FcgammaRs inhibits primate lentivirus replication in human macrophages Single cycle structure-based humanization of an anti-nerve growth factor therapeutic antibody CD4 ligation on human blood monocytes triggers macrophage differentiation and enhances HIV infection Potent anti-R5 human immunodeficiency virus type 1 effects of a CCR5 antagonist, AK602/ONO4128/GW873140, in a novel human peripheral blood mononuclear cell nonobese diabetic-SCID, interleukin-2 receptor gamma-chain-knocked-out AIDS mouse model.Interactions between HIV-1 and mucosal cells in the female reproductive tract The contribution of peroxynitrite generation in HIV replication in human primary macrophages.Pharmacovirological impact of an integrase inhibitor on human immunodeficiency virus type 1 cDNA species in vivo Role of macrophages in HIV infection and persistence.Inhibition of human immunodeficiency virus (HIV-1) infection in human peripheral blood leucocytes-SCID reconstituted mice by rapamycin.Transforming growth factor-beta induces nerve growth factor expression in pancreatic stellate cells by activation of the ALK-5 pathway.Profound anti-HIV-1 activity of DAPTA in monocytes/macrophages and inhibition of CCR5-mediated apoptosis in neuronal cells.Hitting HIV where it hides.Limited development and progression of resistance of HIV-1 to the nucleoside analogue reverse transcriptase inhibitor lamivudine in human primary macrophages.Inhibition of HIV-1 replication in macrophages by a heterodinucleotide of lamivudine and tenofovir.

P2860

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P2860

Anti-nerve growth factor Ab abrogates macrophage-mediated HIV-1 infection and depletion of CD4+ T lymphocytes in hu-SCID mice

http://www.wikidata.org/entity/Q35171217

description

2003 nî lūn-bûn

@nan

2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի հուլիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年学术文章

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2003年学术文章

@zh-cn

2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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name

Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

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Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

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type

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Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

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Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

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prefLabel

Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

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Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

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PNAS.1332627100

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Proceedings of the National Academy of Sciences of the United States of America

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Anti-nerve growth factor Ab ab ...... T lymphocytes in hu-SCID mice

@en

P2093

Alessandra Amendola

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Carlo-Federico Perno

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Caterina Lapenta

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Enrico Garaci

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Filippo Belardelli

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Massimo Spada

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Sonia Covaceuszach

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Stefano Aquaro

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P2860

Macrophage are the principal reservoir and sustain high virus loads in rhesus macaques after the depletion of CD4+ T cells by a highly pathogenic simian immunodeficiency virus/HIV type 1 chimera (SHIV): Implications for HIV-1 infections of humans

Selective killing of human immunodeficiency virus-infected cells by targeted gene transfer and inducible gene expression using a recombinant human immunodeficiency virus vector.

An analysis of HIV-1-associated inflammatory products in brain tissue of humans and SCID mice with HIV-1 encephalitis.

Phenotypic knockout of nerve growth factor in adult transgenic mice reveals severe deficits in basal forebrain cholinergic neurons, cell death in the spleen, and skeletal muscle dystrophy.

Persistent CCR5 utilization and enhanced macrophage tropism by primary blood human immunodeficiency virus type 1 isolates from advanced stages of disease and comparison to tissue-derived isolates

Identification of shared populations of human immunodeficiency virus type 1 infecting microglia and tissue macrophages outside the central nervous system.

Elimination of infectious human immunodeficiency virus from human T-cell cultures by synergistic action of CD4-Pseudomonas exotoxin and reverse transcriptase inhibitors.

Nerve growth factor inhibits apoptosis in memory B lymphocytes via inactivation of p38 MAPK, prevention of Bcl-2 phosphorylation, and cytochrome c release.

Amino acid deletions are introduced into the V2 region of gp120 during independent pathogenic simian immunodeficiency virus/HIV chimeric virus (SHIV) infections of rhesus monkeys generating variants that are macrophage tropic.

Macrophage activation and HIV infection: can the Trojan horse turn into a fortress?

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10.1073/PNAS.1332627100

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