Participation of target Fas protein in apoptosis pathway induced by CD4+ Th1 and CD8+ cytotoxic T cells.
about
The role of CD95 and CD95 ligand in cancerBystander target cell lysis and cytokine production by dengue virus-specific human CD4(+) cytotoxic T-lymphocyte clonesFas- and tumor necrosis factor-induced apoptosis is inhibited by the poxvirus crmA gene productA recombinant scFv-FasLext as a targeting cytotoxic agent against human Jurkat-Ras cancerA novel role of IL-2 in organ-specific autoimmune inflammation beyond regulatory T cell checkpoint: both IL-2 knockout and Fas mutation prolong lifespan of Scurfy mice but by different mechanismsCa2+ signaling modulates cytolytic T lymphocyte effector functions.Distinct T cell receptor signaling requirements for perforin- or FasL-mediated cytotoxicity.Fas (CD95)-dependent cell-mediated immunity to Listeria monocytogenes.Th1 CD4+ lymphocytes delete activated macrophages through the Fas/APO-1 antigen pathway.Cyclosporin A-sensitive transcription factor Egr-3 regulates Fas ligand expression.The relative role of lymphocyte granule exocytosis versus death receptor-mediated cytotoxicity in viral pathophysiologyThe immunomodulatory role of CD4-positive cytotoxic T-lymphocytes in health and disease.Molecular and cellular mechanisms regulating T and B cell apoptosis through Fas/FasL interaction.Involvement of Fas-Fas ligand interactions in graft rejection.IL-11 separates graft-versus-leukemia effects from graft-versus-host disease after bone marrow transplantation.Fas ligand expression on T cells is sufficient to prevent prolonged airway inflammation in a murine model of asthma.Acute cardiac allograft rejection by directly cytotoxic CD4 T cells: parallel requirements for Fas and perforin.Inducible resistance to Fas-mediated apoptosis in B cells.The course of Mycobacterium tuberculosis infection in the lungs of mice lacking expression of either perforin- or granzyme-mediated cytolytic mechanismsBoth the Fas ligand and inducible nitric oxide synthase are needed for control of parasite replication within lesions in mice infected with Leishmania major whereas the contribution of tumor necrosis factor is minimal.Induction of degenerative brain lesions after adoptive transfer of brain lymphocytes from Borna disease virus-infected rats: presence of CD8+ T cells and perforin mRNAThe adenovirus E3-10.4K/14.5K complex mediates loss of cell surface Fas (CD95) and resistance to Fas-induced apoptosisIncreased protection from vaccinia virus infection in mice genetically prone to lymphoproliferative disorders.Differential ability of T cell subsets to undergo activation-induced cell death.CD40 ligation induces Apo-1/Fas expression on human B lymphocytes and facilitates apoptosis through the Apo-1/Fas pathway.Involvement of the CD95 (APO-1/Fas) receptor and ligand in liver damage.The Fas counterattack: Fas-mediated T cell killing by colon cancer cells expressing Fas ligandOpposing effects of transmembrane and soluble Fas ligand expression on inflammation and tumor cell survival.Essential lymphocyte function associated 1 (LFA-1): intercellular adhesion molecule interactions for T cell-mediated B cell apoptosis by Fas/APO-1/CD95.Tolerant B lymphocytes acquire resistance to Fas-mediated apoptosis after treatment with interleukin 4 but not after treatment with specific antigen unless a surface immunoglobulin threshold is exceeded.The Fas counterattack: a molecular mechanism of tumor immune privilege.Quantitation of Fas and Fas ligand gene expression in human ovarian, cervical and endometrial carcinomas using real-time quantitative RT-PCR.Th1 and Th2 cells form morphologically distinct immunological synapses.Infiltration of CD4+ lymphocytes into the brain contributes to neurodegeneration in a mouse model of Parkinson disease.Fas-dependent CD4+ cytotoxic T-cell-mediated pathogenesis during virus infectionThe dual functions of fas ligand in the regulation of peripheral CD8+ and CD4+ T cells.Linomide prevents the lethal effect of anti-Fas antibody and reduces Fas-mediated ceramide production in mouse hepatocytes.Perforin/granzyme-dependent and independent mechanisms are both important for the development of graft-versus-host disease after murine bone marrow transplantationRegression of basal cell carcinoma by intralesional interferon-alpha treatment is mediated by CD95 (Apo-1/Fas)-CD95 ligand-induced suicideDifferential effects of staphylococcal toxic shock syndrome toxin-1 on B cell apoptosis.
P2860
Q26995847-07A3287F-B3C0-4216-8B03-AFF376A1D6F9Q27469356-897DDF33-267E-47D2-B387-C557666F9FDBQ28639862-42D2549D-666E-4F09-AC7F-0F98206CE06CQ30421880-5C925BF3-B442-4244-BAC2-391CE4A854CFQ30423284-2C1B7746-D6A0-40E9-A0F1-4F3DF8E74B04Q30442666-FFCFC90D-8FB3-434E-B36C-936D21F28C3AQ30442845-9BFD6826-24E3-4CEE-B4B8-BD819E6A345CQ33761193-5D35F51D-3EAD-49CC-8753-808DF4FB4670Q33766466-03E82129-7D01-48FA-94F3-A20D755B2EAEQ33774541-DC8D6F92-D9DD-441B-98A6-0CA4F80105DFQ33781851-5B2EB638-4DAA-485E-8162-D6F2F48AF963Q33836317-868844BA-C5FA-4A3B-9565-66F49A407179Q33836328-B3E685C0-A1E9-4E4C-A808-E4B5C1AF09C8Q33836342-76966779-A5BF-4D3A-8A84-3431B2685CC8Q33853636-592C768E-9ED7-4042-9E2F-16B5D860E5FAQ34104832-C776066B-47C3-4BA9-8637-CFCFA5CD5D5BQ34128024-7B59662E-0613-4165-91F5-566018F3EEBFQ34146806-650C6289-AE03-4E99-A751-CD6EEE175EF3Q35545357-8140A10F-E4B9-494B-A379-B77C939B3901Q35801828-8B7AA225-DE0A-47E3-AD11-8E642E19505AQ35879561-9D16E3F8-B12B-48B4-B01A-1D5C01B1B9E1Q35898131-1E5826DE-5B12-45EB-8B5F-473CB4C5C20DQ36023204-A501C2FB-41C8-498E-92EF-CD866263DFE8Q36158748-316EC82F-98BB-4986-823D-91FF4E7618D9Q36365403-01EFDE0A-6B5D-4F93-949B-A7380B1E90BEQ36365423-CDC80AFC-A9F6-4A8E-9D94-20D51F8243D2Q36367323-F09574C7-231C-458A-B0BC-2C2B50283B63Q36368504-72B41DA5-1088-440C-99AE-272FE9364886Q36380686-3AED2637-CFAB-47C4-AC1A-2C129079AFF6Q36400473-8D6C7E21-2D58-420E-862D-92150A6BE1BBQ36437768-214E0712-C9BF-4C72-9056-468BABBFEBB5Q36641753-1C442F48-321A-46F6-B0D3-472793CB9B11Q36740495-79FF6274-0F0A-402C-BBD1-4E3B8F37FE0DQ37036090-182F1290-94A7-445A-AF6C-61435D35805CQ37050470-5F6A9F9A-4533-46FC-BEA5-EA6DEDD661D1Q37114374-A92FF317-4403-4F3D-AC89-C3CC8FD23DECQ37359497-98D60F8A-9D3D-4437-A3E2-F6BA4DD924A6Q37371294-6B25D766-7DB1-492E-A27B-E781E3C09BBDQ37375409-5217EF34-9B1C-481B-84C9-DADA5ED86AADQ37588015-E948A875-625F-4A91-8F08-D6F68B290547
P2860
Participation of target Fas protein in apoptosis pathway induced by CD4+ Th1 and CD8+ cytotoxic T cells.
description
1994 nî lūn-bûn
@nan
1994 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@ast
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@en
type
label
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@ast
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@en
prefLabel
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@ast
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@en
P2093
P2860
P356
P1476
Participation of target Fas pr ...... h1 and CD8+ cytotoxic T cells.
@en
P2093
A Marshak-Rothstein
R Ettinger
P2860
P304
P356
10.1073/PNAS.91.10.4185
P407
P577
1994-05-01T00:00:00Z