c-myc as a mediator of accelerated apoptosis and involution in mammary glands lacking Socs3.
about
Deaf-1 regulates epithelial cell proliferation and side-branching in the mammary glandSOCS3: An essential physiological inhibitor of signaling by interleukin-6 and G-CSF family cytokinesc-Myc affects mRNA translation, cell proliferation and progenitor cell function in the mammary gland.Novel IL-21 signaling pathway up-regulates c-Myc and induces apoptosis of diffuse large B-cell lymphomas.Murine models of colorectal cancer.Expression of metabolic, tissue remodeling, oxidative stress, and inflammatory pathways in mammary tissue during involution in lactating dairy cowsArsenic trioxide-mediated growth inhibition of myeloma cells is associated with an extrinsic or intrinsic signaling pathway through activation of TRAIL or TRAIL receptor 2.Replacement of E-cadherin by N-cadherin in the mammary gland leads to fibrocystic changes and tumor formation.Direct and immune-mediated cytotoxicity of interleukin-21 contributes to antitumor effects in mantle cell lymphoma.Deletion of the SOCS box of suppressor of cytokine signaling 3 (SOCS3) in embryonic stem cells reveals SOCS box-dependent regulation of JAK but not STAT phosphorylation.The BH3-only protein BIM contributes to late-stage involution in the mouse mammary gland.Hunk negatively regulates c-myc to promote Akt-mediated cell survival and mammary tumorigenesis induced by loss of Pten.Forced involution of the functionally differentiated mammary gland by overexpression of the pro-apoptotic protein baxImmune cell regulators in mouse mammary development and involution.Key signalling nodes in mammary gland development and cancer: Myc.The relationship between total and phosphorylated STAT1 and STAT3 tumour cell expression, components of tumour microenvironment and survival in patients with invasive ductal breast cancer.c-Myc induction of programmed cell death may contribute to carcinogenesis: a perspective inspired by several concepts of chemical carcinogenesis.Stat3 accelerates Myc induced tumor formation while reducing growth rate in a mouse model of breast cancer.Protective properties of inhaled IL-22 in a model of ventilator-induced lung injury.Tissue microarray-based study of patients with lymph node-positive breast cancer shows tyrosine phosphorylation of signal transducer and activator of transcription 3 (tyrosine705-STAT3) is a marker of good prognosis.Th-POK regulates mammary gland lactation through mTOR-SREBP pathway.
P2860
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P2860
c-myc as a mediator of accelerated apoptosis and involution in mammary glands lacking Socs3.
description
2006 nî lūn-bûn
@nan
2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
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2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@ast
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@en
type
label
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@ast
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@en
prefLabel
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@ast
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@en
P2093
P2860
P356
P1433
P1476
c-myc as a mediator of acceler ...... mammary glands lacking Socs3.
@en
P2093
Christine J Watson
Edward J McManus
François Vaillant
Geoffrey J Lindeman
Gunther Schutz
Jane E Visvader
Kate D Sutherland
Lewis A Chodosh
Natasha C Forrest
Sheridan L Holroyd
P2860
P304
P356
10.1038/SJ.EMBOJ.7601455
P407
P577
2006-11-30T00:00:00Z