A new class of mutations reveals a novel function for the original phosphatidylinositol 3-kinase binding site.
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PRL-3 activates mTORC1 in Cancer Progression.Lessons from polyoma middle T antigen on signaling and transformation: A DNA tumor virus contribution to the war on cancer.Polyomavirus middle T antigen induces the transcription of osteopontin, a gene important for the migration of transformed cells.The p110alpha isoform of phosphatidylinositol 3-kinase is essential for polyomavirus middle T antigen-mediated transformation.
P2860
A new class of mutations reveals a novel function for the original phosphatidylinositol 3-kinase binding site.
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2003 nî lūn-bûn
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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած
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2003 թվականի հուլիսին հրատարակված գիտական հոդված
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2003年の論文
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2003年論文
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2003年論文
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2003年論文
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2003年論文
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2003年論文
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2003年论文
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name
A new class of mutations revea ...... nositol 3-kinase binding site.
@ast
A new class of mutations revea ...... nositol 3-kinase binding site.
@en
type
label
A new class of mutations revea ...... nositol 3-kinase binding site.
@ast
A new class of mutations revea ...... nositol 3-kinase binding site.
@en
prefLabel
A new class of mutations revea ...... nositol 3-kinase binding site.
@ast
A new class of mutations revea ...... nositol 3-kinase binding site.
@en
P2093
P2860
P356
P1476
A new class of mutations revea ...... nositol 3-kinase binding site.
@en
P2093
Aki Mikami
Brian Schaffhausen
Thomas M Roberts
Y Kate Hong
P2860
P304
P356
10.1073/PNAS.1432964100
P407
P577
2003-07-24T00:00:00Z