Capsule enlargement in Cryptococcus neoformans confers resistance to oxidative stress suggesting a mechanism for intracellular survival.
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The capsule of Porphyromonas gingivalis leads to a reduction in the host inflammatory response, evasion of phagocytosis, and increase in virulenceThe Cryptococcus neoformans capsule: a sword and a shield.Cryptococcus neoformans: Tripping on Acid in the PhagolysosomeCryptococcus and Phagocytes: Complex Interactions that Influence Disease OutcomeFungal cell gigantism during mammalian infectionStructures of Cryptococcus neoformans Protein Farnesyltransferase Reveal Strategies for Developing Inhibitors That Target Fungal PathogensAnti-Immune Strategies of Pathogenic FungiCapsules from pathogenic and non-pathogenic Cryptococcus spp. manifest significant differences in structure and ability to protect against phagocytic cellsFluconazole alters the polysaccharide capsule of Cryptococcus gattii and leads to distinct behaviors in murine CryptococcosisCapsule independent uptake of the fungal pathogen Cryptococcus neoformans into brain microvascular endothelial cellsSpecies in the Cryptococcus gattii Complex Differ in Capsule and Cell Size following Growth under Capsule-Inducing ConditionsCryptococcal interactions with the host immune system.Surfactant protein D facilitates Cryptococcus neoformans infection.Extracellular vesicles from Cryptococcus neoformans modulate macrophage functionsPhagocytosis of Cryptococcus neoformans by, and nonlytic exocytosis from, Acanthamoeba castellanii.The interaction between Candida krusei and murine macrophages results in multiple outcomes, including intracellular survival and escape from killing.Temporal behavior of capsule enlargement by Cryptococcus neoformans.Paradoxical growth of Candida albicans in the presence of caspofungin is associated with multiple cell wall rearrangements and decreased virulence.Phylogenomics of the oxidative phosphorylation in fungi reveals extensive gene duplication followed by functional divergence.Elucidation of the Role of 3-Hydroxy Fatty Acids in Cryptococcus-amoeba Interactions.Paradoxical roles of alveolar macrophages in the host response to Cryptococcus neoformans.Capsule growth in Cryptococcus neoformans is coordinated with cell cycle progression.Phenotypic differences of Cryptococcus molecular types and their implications for virulence in a Drosophila model of infection.Phospholipids trigger Cryptococcus neoformans capsular enlargement during interactions with amoebae and macrophages.Cryptococcus neoformans histone acetyltransferase Gcn5 regulates fungal adaptation to the hostNonlytic exocytosis of Cryptococcus neoformans from macrophages occurs in vivo and is influenced by phagosomal pH.Cryptococcus interactions with macrophages: evasion and manipulation of the phagosome by a fungal pathogen.Mechanisms of microbial escape from phagocyte killing.Evidence for branching in cryptococcal capsular polysaccharides and consequences on its biological activity.Antibiotic modulation of capsular exopolysaccharide and virulence in Acinetobacter baumanniiRole of sphingosine-1-phosphate (S1P) and S1P receptor 2 in the phagocytosis of Cryptococcus neoformans by alveolar macrophagesMultiple Disguises for the Same Party: The Concepts of Morphogenesis and Phenotypic Variations in Cryptococcus neoformansFungal Pathogens: Survival and Replication within MacrophagesHeteroresistance to Itraconazole Alters the Morphology and Increases the Virulence of Cryptococcus gattii.Cryptococcus neoformans induces antimicrobial responses and behaves as a facultative intracellular pathogen in the non mammalian model Galleria mellonellaALL2, a Homologue of ALL1, Has a Distinct Role in Regulating pH Homeostasis in the Pathogen Cryptococcus neoformans.Cryptococcus neoformans Rim101 is associated with cell wall remodeling and evasion of the host immune responses.Magnesium Ion Acts as a Signal for Capsule Induction in Cryptococcus neoformans.The transcriptional response of Cryptococcus neoformans to ingestion by Acanthamoeba castellanii and macrophages provides insights into the evolutionary adaptation to the mammalian host.Redundant catalases detoxify phagocyte reactive oxygen and facilitate Histoplasma capsulatum pathogenesis
P2860
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P2860
Capsule enlargement in Cryptococcus neoformans confers resistance to oxidative stress suggesting a mechanism for intracellular survival.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@ast
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@en
type
label
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@ast
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@en
prefLabel
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@ast
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@en
P2093
P2860
P1476
Capsule enlargement in Cryptoc ...... sm for intracellular survival.
@en
P2093
Cara J Chrisman
Juan Luis Rodríguez-Tudela
Manuel Cuenca-Estrella
Maria Victoria Castelli
Oscar Zaragoza
Susana Frases
P2860
P304
P356
10.1111/J.1462-5822.2008.01186.X
P577
2008-06-28T00:00:00Z