Neutrophils are critical for host defense against primary infection with the facultative intracellular bacterium Francisella tularensis in mice and participate in defense against reinfection
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TularemiaImmunotherapy for tularemiaSubversion of host recognition and defense systems by Francisella sppLow doses of imatinib induce myelopoiesis and enhance host anti-microbial immunityA novel receptor - ligand pathway for entry of Francisella tularensis in monocyte-like THP-1 cells: interaction between surface nucleolin and bacterial elongation factor Tu.Molecular immune responses to aerosol challenge with Francisella tularensis in mice inoculated with live vaccine candidates of varying efficacy.Toll-like receptor 3 agonist protection against experimental Francisella tularensis respiratory tract infectionNucleolin, a shuttle protein promoting infection of human monocytes by Francisella tularensis.Role of polymorphonuclear neutrophils in a murine model of Chlamydia psittaci-induced abortion.Pyrin-only protein 2 limits inflammation but improves protection against bacteria.A galU mutant of Francisella tularensis is attenuated for virulence in a murine pulmonary model of tularemiaRapid local expression of interleukin-12, tumor necrosis factor alpha, and gamma interferon after cutaneous Francisella tularensis infection in tularemia-immune mice.Neutrophils play a protective nonphagocytic role in systemic Mycobacterium tuberculosis infection of miceHost-pathogen interactions and immune evasion strategies in Francisella tularensis pathogenicityIKKβ in myeloid cells controls the host response to lethal and sublethal Francisella tularensis LVS infection.Characterization of lethal inhalational infection with Francisella tularensis in the common marmoset (Callithrix jacchus).Administration of a synthetic TLR4 agonist protects mice from pneumonic tularemia.CD14 signaling reciprocally controls collagen deposition and turnover to regulate the development of lyme arthritis.Interleukin-17 is required for T helper 1 cell immunity and host resistance to the intracellular pathogen Francisella tularensis.Mac-1+ cells are the predominant subset in the early hepatic lesions of mice infected with Francisella tularensis.A MyD88-dependent IFNγR-CCR2 signaling circuit is required for mobilization of monocytes and host defense against systemic bacterial challenge.Cytokine expression in the liver during the early phase of murine tularemia.Interleukin-6 is required for a protective immune response to systemic Escherichia coli infectionMinimal requirements for murine resistance to infection with Francisella tularensis LVS.Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.Effects of neutrophil, natural killer cell, and macrophage depletion on murine Clostridium piliforme infection.Critical roles of neutrophils in host defense against experimental systemic infections of mice by Listeria monocytogenes, Salmonella typhimurium, and Yersinia enterocoliticaResistance of Francisella tularensis strains against reactive nitrogen and oxygen species with special reference to the role of KatG.Francisella tularensis: taxonomy, genetics, and Immunopathogenesis of a potential agent of biowarfare.Role of primary human alveolar epithelial cells in host defense against Francisella tularensis infection.An Immature Myeloid/Myeloid-Suppressor Cell Response Associated with Necrotizing Inflammation Mediates Lethal Pulmonary Tularemia.Transcriptome analysis of human immune responses following live vaccine strain (LVS) Francisella tularensis vaccinationPhenotypic, morphological, and functional heterogeneity of splenic immature myeloid cells in the host response to tularemia.Inflammasome-Independent NLRP3 Restriction of a Protective Early Neutrophil Response to Pulmonary Tularemia.Type I interferon signaling is required for activation of the inflammasome during Francisella infection.Repression of bacterial lipoprotein production by Francisella novicida facilitates evasion of innate immune recognitionNeutrophils play an important role in host resistance to respiratory infection with Acinetobacter baumannii in mice.Murine salmonellosis studied by confocal microscopy: Salmonella typhimurium resides intracellularly inside macrophages and exerts a cytotoxic effect on phagocytes in vivo.Specific depletion reveals a novel role for neutrophil-mediated protection in the liver during Listeria monocytogenes infectionB1a cells enhance susceptibility to infection with virulent Francisella tularensis via modulation of NK/NKT cell responses.
P2860
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P2860
Neutrophils are critical for host defense against primary infection with the facultative intracellular bacterium Francisella tularensis in mice and participate in defense against reinfection
description
1994 nî lūn-bûn
@nan
1994 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Neutrophils are critical for h ...... in defense against reinfection
@ast
Neutrophils are critical for h ...... in defense against reinfection
@en
Neutrophils are critical for h ...... in defense against reinfection
@nl
type
label
Neutrophils are critical for h ...... in defense against reinfection
@ast
Neutrophils are critical for h ...... in defense against reinfection
@en
Neutrophils are critical for h ...... in defense against reinfection
@nl
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Neutrophils are critical for h ...... in defense against reinfection
@ast
Neutrophils are critical for h ...... in defense against reinfection
@en
Neutrophils are critical for h ...... in defense against reinfection
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P2860
P1476
Neutrophils are critical for h ...... in defense against reinfection
@en
P2093
P2860
P304
P407
P577
1994-07-01T00:00:00Z