Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
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Effect of anti-inflammatory agents on transforming growth factor beta over-expressing mouse brains: a model revisedACE overexpression in myelomonocytic cells: effect on a mouse model of Alzheimer's diseaseBlood platelets in the progression of Alzheimer's diseaseProtein misfolding and aggregation in Alzheimer's disease and type 2 diabetes mellitusAβ40 oligomers identified as a potential biomarker for the diagnosis of Alzheimer's diseaseNeuroprotective and neurotoxic properties of glial cells in the pathogenesis of Alzheimer's diseaseAlzheimer disease models and human neuropathology: similarities and differencesA longitudinal study of cognition, proton MR spectroscopy and synaptic and neuronal pathology in aging wild-type and AβPPswe-PS1dE9 miceAlzheimer's disease: pathophysiology and applications of magnetic nanoparticles as MRI theranostic agents.Diverse roles of the vasculature within the neural stem cell niche.Retinal ultrastructure of murine models of dry age-related macular degeneration (AMD).Why AMD is a disease of ageing and not of development: mechanisms and insights.Apolipoprotein E allele-dependent pathogenesis: a model for age-related retinal degeneration.Targeting vascular amyloid in arterioles of Alzheimer disease transgenic mice with amyloid β protein antibody-coated nanoparticles.Solutes, but not cells, drain from the brain parenchyma along basement membranes of capillaries and arteries: significance for cerebral amyloid angiopathy and neuroimmunology.Neuronal or glial expression of human apolipoprotein e4 affects parenchymal and vascular amyloid pathology differentially in different brain regions of double- and triple-transgenic micebeta-site amyloid precursor protein cleaving enzyme 1 increases amyloid deposition in brain parenchyma but reduces cerebrovascular amyloid angiopathy in aging BACE x APP[V717I] double-transgenic miceThe nature and effects of cortical microvascular pathology in aging and Alzheimer's disease.The neurovascular unit and its growth factors: coordinated response in the vascular and nervous systems.Mechanism of cerebral beta-amyloid angiopathy: murine and cellular models.Regional cerebral blood flow reductions, heart failure and Alzheimer's disease.Population studies of sporadic cerebral amyloid angiopathy and dementia: a systematic review.Cholesterol retention in Alzheimer's brain is responsible for high beta- and gamma-secretase activities and Abeta production.ABCG2 is upregulated in Alzheimer's brain with cerebral amyloid angiopathy and may act as a gatekeeper at the blood-brain barrier for Abeta(1-40) peptides.ABC transporters and cytochromes P450 in the human central nervous system: influence on brain pharmacokinetics and contribution to neurodegenerative disorders.Is Vasomotion in Cerebral Arteries Impaired in Alzheimer's Disease?Thiazine Red(+) platelet inclusions in Cerebral Blood Vessels are first signs in an Alzheimer's Disease mouse model.Low levels of amyloid-beta and its transporters in neonatal rats with and without hydrocephalus.Alzheimer disease macrophages shuttle amyloid-beta from neurons to vessels, contributing to amyloid angiopathy.Gene expression profiles of peripheral and cord blood mononuclear cells altered by thymic stromal lymphopoietin.Passive immunotherapy targeting amyloid-β reduces cerebral amyloid angiopathy and improves vascular reactivity.Attenuation of Abeta deposition in the entorhinal cortex of normal elderly individuals associated with tobacco smoking.Cerebral amyloid angiopathy in streptozotocin rat model of sporadic Alzheimer's disease: a long-term follow up study.Beta-amyloid and oxidative stress jointly induce neuronal death, amyloid deposits, gliosis, and memory impairment in the rat brain.Amyloid-β-dependent compromise of microvascular structure and function in a model of Alzheimer's disease.Platelets are responsible for the accumulation of β-amyloid in blood clots inside and around blood vessels in mouse brain after thrombosis.Clinical assessment of cerebrospinal fluid dynamics in hydrocephalus. Guide to interpretation based on observational study.Proteomics analysis identifies new markers associated with capillary cerebral amyloid angiopathy in Alzheimer's disease.Platelets isolated from an Alzheimer mouse damage healthy cortical vessels and cause inflammation in an organotypic ex vivo brain slice modelAngiotensin-converting enzyme (ACE) levels and activity in Alzheimer's disease, and relationship of perivascular ACE-1 to cerebral amyloid angiopathy
P2860
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P2860
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
description
2003 nî lūn-bûn
@nan
2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@ast
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@en
type
label
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@ast
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@en
prefLabel
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@ast
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@en
P2860
P1476
Cerebral amyloid angiopathy: pathogenesis and effects on the ageing and Alzheimer brain.
@en
P2093
James A Nicoll
Roy O Weller
P2860
P304
P356
10.1179/016164103101202057
P577
2003-09-01T00:00:00Z