Astrocyte response to IFN-γ limits IL-6-mediated microglia activation and progressive autoimmune encephalomyelitis. - Wikidata - wikimedia - TriplyDB

Astrocyte response to IFN-γ limits IL-6-mediated microglia activation and progressive autoimmune encephalomyelitis.

Astrocyte response to IFN-γ limits IL-6-mediated microglia activation and progressive autoimmune encephalomyelitis.

http://www.wikidata.org/entity/Q35540927

about

Nutritional and Nanotechnological Modulators of Microglia Neuroprotection by IFN-γ via astrocyte-secreted IL-6 in acute neuroinflammation.Protective effect of carnosine after chronic cerebral hypoperfusion possibly through suppressing astrocyte activation.Sustained TNF production by central nervous system infiltrating macrophages promotes progressive autoimmune encephalomyelitis.Opposing Roles of Interferon-Gamma on Cells of the Central Nervous System in Autoimmune Neuroinflammation.Microglia: Housekeeper of the Central Nervous System.Priming astrocytes with TNF enhances their susceptibility to Trypanosoma cruzi infection and creates a self-sustaining inflammatory milieu.IL-17-triggered downregulation of miR-497 results in high HIF-1α expression and consequent IL-1β and IL-6 production by astrocytes in EAE mice.Sphingosine 1-phosphate receptor modulation suppresses pathogenic astrocyte activation and chronic progressive CNS inflammation.Effectors of Th1 and Th17 cells act on astrocytes and augment their neuroinflammatory properties.dOCRL maintains immune cell quiescence by regulating endosomal traffic.Microglia and Aging: The Role of the TREM2-DAP12 and CX3CL1-CX3CR1 Axes.Blockade of sustained tumor necrosis factor in a transgenic model of progressive autoimmune encephalomyelitis limits oligodendrocyte apoptosis and promotes oligodendrocyte maturation.

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Astrocyte response to IFN-γ limits IL-6-mediated microglia activation and progressive autoimmune encephalomyelitis.

http://www.wikidata.org/entity/Q35540927

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

@wuu

name

Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

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Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

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type

label

Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

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Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

@en

prefLabel

Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

@ast

Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

@en

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Journal of Neuroinflammation

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Astrocyte response to IFN-γ li ...... autoimmune encephalomyelitis.

@en

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Alice Valentin-Torres

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Bruce D Trapp

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Carine Savarin

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David R Hinton

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Stephen A Stohlman

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Zhihong Chen

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Astrocytes: biology and pathology

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Interleukin-6 family of cytokines and gp130

Immune privilege as an intrinsic CNS property: astrocytes protect the CNS against T-cell-mediated neuroinflammation

Conditional ablation of astroglial CCL2 suppresses CNS accumulation of M1 macrophages and preserves axons in mice with MOG peptide EAE.

IFN-γ signaling to astrocytes protects from autoimmune mediated neurological disability

Cytokines and chemokines as mediators of protection and injury in the central nervous system assessed in transgenic mice.

RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation.

Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation

Lipopolysaccharide-induced microglial activation and neuroprotection against experimental brain injury is independent of hematogenous TLR4

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Cornelia C. Bergmann

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