Induction of the skeletal alpha-actin gene in alpha 1-adrenoceptor-mediated hypertrophy of rat cardiac myocytes
about
Expression cloning of cardiotrophin 1, a cytokine that induces cardiac myocyte hypertrophyInhibition of norepinephrine-induced cardiac hypertrophy in s100beta transgenic mice.Repression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophyDeep mRNA sequencing for in vivo functional analysis of cardiac transcriptional regulators: application to Galphaq.A Ras-dependent pathway regulates RNA polymerase II phosphorylation in cardiac myocytes: implications for cardiac hypertrophy.Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.FGF23 induces left ventricular hypertrophyThe vascular smooth muscle alpha-actin gene is reactivated during cardiac hypertrophy provoked by load.Skeletal actin mRNA increases in the human heart during ontogenic development and is the major isoform of control and failing adult hearts.Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes.The cardiac beta-myosin heavy chain isogene is induced selectively in alpha 1-adrenergic receptor-stimulated hypertrophy of cultured rat heart myocytesNoncoordinate regulation of alpha-1 adrenoreceptor coupling and reexpression of alpha skeletal actin in myocardial infarction-induced left ventricular failure in ratsAlpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload.Maternal diet-induced obesity programs cardiovascular dysfunction in adult male mouse offspring independent of current body weightAlpha-1-adrenergic receptors: targets for agonist drugs to treat heart failure.Chronic hemodynamic unloading regulates the morphologic development of newborn mouse hearts transplanted into the ear of isogeneic adult miceDifferentiation of cardiac myocytes after mitogen withdrawal exhibits three sequential states of the ventricular growth responseTransgenic Galphaq overexpression induces cardiac contractile failure in miceThe c-myc proto-oncogene regulates cardiac development in transgenic mice.Transcriptional activation of the cardiac myosin light chain 2 and atrial natriuretic factor genes by protein kinase C in neonatal rat ventricular myocytes.Re-expression of alpha skeletal actin as a marker for dedifferentiation in cardiac pathologies.Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.Adrenergic regulation of the skeletal alpha-actin gene promoter during myocardial cell hypertrophy.Cardiac alpha1-adrenergic receptors: novel aspects of expression, signaling mechanisms, physiologic function, and clinical importanceSegregation of atrial-specific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy.Alpha-1-adrenergic receptors in heart failure: the adaptive arm of the cardiac response to chronic catecholamine stimulation.Effect of thyroid hormone on the accumulation of mRNA for skeletal and cardiac alpha-actin in hearts from normal and hypophysectomized rats.Adrenergic signaling polymorphisms and their impact on cardiovascular disease.β-myosin heavy chain is induced by pressure overload in a minor subpopulation of smaller mouse cardiac myocytes.Endothelin-3 induces hypertrophy of cardiomyocytes by the endogenous endothelin-1-mediated mechanism.The water extract of Jagamchotang protects the ischemia/reperfusion-induced cytotoxicity of rat neonatal myocardial cells via generation of nitric oxide.Nuclear accumulation of myocyte muscle LIM protein is regulated by heme oxygenase 1 and correlates with cardiac function in the transition to failure.Alpha 1-adrenergic receptor stimulation of sarcomeric actin isogene transcription in hypertrophy of cultured rat heart muscle cells.Some thoughts on pathogenesis, therapy and prevention of hypertension.Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.Directed inhibition of nuclear import in cellular hypertrophy.Low-dose ACE with alpha- or beta-adrenergic receptor inhibitors have beneficial SHR cardiovascular effects.M1 muscarinic receptors heterologously expressed in cardiac myocytes mediate Ras-dependent changes in gene expression.Interleukin-1beta is a negative transcriptional regulator of alpha1-adrenergic induced gene expression in cultured cardiac myocytes.S100beta inhibits alpha1-adrenergic induction of the hypertrophic phenotype in cardiac myocytes.
P2860
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P2860
Induction of the skeletal alpha-actin gene in alpha 1-adrenoceptor-mediated hypertrophy of rat cardiac myocytes
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@ast
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@en
type
label
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@ast
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@en
prefLabel
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@ast
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@en
P2093
P2860
P356
P1476
Induction of the skeletal alph ...... trophy of rat cardiac myocytes
@en
P2093
C P Ordahl
N H Bishopric
P C Simpson
P2860
P304
P356
10.1172/JCI113179
P407
P577
1987-10-01T00:00:00Z