Diverse inflammatory responses in transgenic mouse models of Alzheimer's disease and the effect of immunotherapy on these responses.
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Neuroinflammation in Alzheimer's disease; A source of heterogeneity and target for personalized therapyOral TNFα Modulation Alters Neutrophil Infiltration, Improves Cognition and Diminishes Tau and Amyloid Pathology in the 3xTgAD Mouse ModelNeuroinflammation in Alzheimer's diseaseArginine deprivation and immune suppression in a mouse model of Alzheimer's diseaseSelective Brain-Targeted Antagonism of p38 MAPKα Reduces Hippocampal IL-1β Levels and Improves Morris Water Maze Performance in Aged RatsInduction of hyperhomocysteinemia models vascular dementia by induction of cerebral microhemorrhages and neuroinflammation.β-amyloid deposition is shifted to the vasculature and memory impairment is exacerbated when hyperhomocysteinemia is induced in APP/PS1 transgenic mice.Transition from an M1 to a mixed neuroinflammatory phenotype increases amyloid deposition in APP/PS1 transgenic mice.Lithium treatment of APPSwDI/NOS2-/- mice leads to reduced hyperphosphorylated tau, increased amyloid deposition and altered inflammatory phenotype.Single chain variable fragment against aβ expressed in baculovirus inhibits abeta fibril elongation and promotes its disaggregation.Comparing the efficacy and neuroinflammatory potential of three anti-abeta antibodiesCerebrovascular contributions to aging and Alzheimer's disease in Down syndrome.Intracranial injection of Gammagard, a human IVIg, modulates the inflammatory response of the brain and lowers Aβ in APP/PS1 mice along a different time course than anti-Aβ antibodies.Review: experimental manipulations of microglia in mouse models of Alzheimer's pathology: activation reduces amyloid but hastens tau pathology.The impact of neuroimmune changes on development of amyloid pathology; relevance to Alzheimer's disease.New roles for Fc receptors in neurodegeneration-the impact on Immunotherapy for Alzheimer's Disease.The role of the immunoproteasome in interferon-γ-mediated microglial activation.Neuroprotective effects of the anticancer drug NVP-BEZ235 (dactolisib) on amyloid-β 1-42 induced neurotoxicity and memory impairment.A changing perspective on the role of neuroinflammation in Alzheimer's disease.C5a Increases the Injury to Primary Neurons Elicited by Fibrillar Amyloid Beta.Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization.Atherosclerosis is exacerbated by chitinase-3-like-1 in amyloid precursor protein transgenic mice.Functional alterations of myeloid cells during the course of Alzheimer's disease
P2860
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P2860
Diverse inflammatory responses in transgenic mouse models of Alzheimer's disease and the effect of immunotherapy on these responses.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Diverse inflammatory responses ...... unotherapy on these responses.
@ast
Diverse inflammatory responses ...... unotherapy on these responses.
@en
type
label
Diverse inflammatory responses ...... unotherapy on these responses.
@ast
Diverse inflammatory responses ...... unotherapy on these responses.
@en
prefLabel
Diverse inflammatory responses ...... unotherapy on these responses.
@ast
Diverse inflammatory responses ...... unotherapy on these responses.
@en
P2093
P2860
P356
P1433
P1476
Diverse inflammatory responses ...... unotherapy on these responses.
@en
P2093
Angela Everhart
Dave Morgan
Donna M Wilcock
Jennifer E Lee
Joan G Wilson
Marcia N Gordon
P2860
P304
P356
10.1042/AN20110018
P577
2011-11-30T00:00:00Z