Prostate cancer induces bone metastasis through Wnt-induced bone morphogenetic protein-dependent and independent mechanisms. - Wikidata - wikimedia - TriplyDB

Prostate cancer induces bone metastasis through Wnt-induced bone morphogenetic protein-dependent and independent mechanisms.

Prostate cancer induces bone metastasis through Wnt-induced bone morphogenetic protein-dependent and independent mechanisms.

http://www.wikidata.org/entity/Q35609965

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Wnt signaling pathways in urological cancers: past decades and still growing Mouse Models in Prostate Cancer Translational Research: From Xenograft to PDX The role of the BMP signaling antagonist noggin in the development of prostate cancer osteolytic bone metastasis The chemopreventive retinoid 4HPR impairs prostate cancer cell migration and invasion by interfering with FAK/AKT/GSK3beta pathway and beta-catenin stability Beta-catenin phosphorylated at threonine 120 antagonizes generation of active beta-catenin by spatial localization in trans-Golgi network Prostate cancer progression and metastasis: potential regulatory pathways for therapeutic targeting.Identification of differentially expressed subnetworks based on multivariate ANOVA.Metastatic bone disease: role of transcription factors and future targets.Bone Metastasis of Prostate Cancer Can Be Therapeutically Targeted at the TBX2-WNT Signaling Axis.The science and practice of bone health in oncology: managing bone loss and metastasis in patients with solid tumors p21CIP-1/WAF-1 induction is required to inhibit prostate cancer growth elicited by deficient expression of the Wnt inhibitor Dickkopf-1.The host microenvironment influences prostate cancer invasion, systemic spread, bone colonization, and osteoblastic metastasis.Role of non-canonical Wnt signaling in osteoblast maturation on microstructured titanium surfaces.A new murine model of osteoblastic/osteolytic lesions from human androgen-resistant prostate cancer The PCa Tumor Microenvironment.Effects of murine dermal cells on the regulation of hair growth is dependent on the cell number and post-natal age of newborn mice.Identification of NOG as a specific breast cancer bone metastasis-supporting gene Wnt3a: functions and implications in cancer.Cancer to bone: a fatal attraction.Integrin alpha2beta 1 (α2β1) promotes prostate cancer skeletal metastasis.Skeletal metastasis: treatments, mouse models, and the Wnt signaling.Expression of PGK1 by prostate cancer cells induces bone formation.CAPE suppresses migration and invasion of prostate cancer cells via activation of non-canonical Wnt signaling.Reciprocal cross-talk between Prostaglandin E2 and bone in prostate cancer: a current review.Prostate cancer bone metastases acquire resistance to androgen deprivation via WNT5A-mediated BMP-6 induction.Bone morphogenetic proteins and cancer: review of the literature.A novel non-canonical Wnt signature for prostate cancer aggressiveness.Steps in prostate cancer progression that lead to bone metastasis Bone metastasis in prostate cancer: emerging therapeutic strategies.Signaling between tumor cells and the host bone marrow microenvironment.Galectin-3 in bone tumor microenvironment: a beacon for individual skeletal metastasis management.Estrogen and estrogen receptor alpha promotes malignancy and osteoblastic tumorigenesis in prostate cancer.Prostate cancer cells induce osteoblastic differentiation via semaphorin 3A.Does Wnt/β-catenin pathway contribute to the stability of DNMT1 expression in urological cancer cell lines?Effects of the proteasome inhibitor, bortezomib, on cytodifferentiation and mineralization of periodontal ligament cells.Role of WNT7B-induced noncanonical pathway in advanced prostate cancer.Gene expression and epigenetic discovery screen reveal methylation of SFRP2 in prostate cancer.Vitamin D deficiency promotes prostate cancer growth in bone.Bone morphogenetic protein-7 is a MYC target with prosurvival functions in childhood medulloblastoma.Interactions between human osteoblasts and prostate cancer cells in a novel 3D in vitro model.

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P2860

Prostate cancer induces bone metastasis through Wnt-induced bone morphogenetic protein-dependent and independent mechanisms.

http://www.wikidata.org/entity/Q35609965

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2008 nî lūn-bûn

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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type

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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Cancer Research

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Prostate cancer induces bone m ...... nt and independent mechanisms.

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Atsushi Mizokami

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Christopher L Hall

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Evan T Keller

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Jill M Keller

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Jinlu Dai

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June Escara-Wilke

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P2860

Regulation of BMP7 expression during kidney development

Transcriptional mechanisms of bone morphogenetic protein-induced osteoprotegrin gene expression

Bone morphogenetic protein-6 expression in normal and malignant prostate

Wnt signaling: multiple pathways, multiple receptors, and multiple transcription factors

Immunolocalization and messenger RNA expression of bone morphogenetic protein-6 in human benign and malignant prostatic tissue

Expression of bone morphogenetic protein messenger RNAs by normal rat and human prostate and prostate cancer cells

BMP antagonists: their roles in development and involvement in pathophysiology

Molecular cloning and analysis of the 5'-flanking region of the human bone morphogenetic protein-6 (BMP-6)

Wnt but not BMP signaling is involved in the inhibitory action of sclerostin on BMP-stimulated bone formation

Sclerostin inhibition of Wnt-3a-induced C3H10T1/2 cell differentiation is indirect and mediated by bone morphogenetic proteins

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