Neutrophil apoptosis: a marker of disease severity in sepsis and sepsis-induced acute respiratory distress syndrome.
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The role of pro-resolution lipid mediators in infectious diseaseExtracellular administration of BCL2 protein reduces apoptosis and improves survival in a murine model of sepsisA Neutrophil Phenotype Model for Extracorporeal Treatment of SepsisEffect of siRNA against NF-κB on sepsis‑induced acute lung injury in a mouse model.Bacteria-specific neutrophil dysfunction associated with interferon-stimulated gene expression in the acute respiratory distress syndrome.Neutrophils are not less sensitive than other blood leukocytes to the genomic effects of glucocorticoids.Bcl3 prevents acute inflammatory lung injury in mice by restraining emergency granulopoiesis.Pathogenesis of indirect (secondary) acute lung injury.Increased serum soluble Fas after major trauma is associated with delayed neutrophil apoptosis and development of sepsisIncreased expression of neutrophil-related genes in patients with early sepsis-induced ARDS.Francisella tularensis inhibits the intrinsic and extrinsic pathways to delay constitutive apoptosis and prolong human neutrophil lifespan.High-dose ascorbate with low-dose amphotericin B attenuates severity of disease in a model of the reappearance of candidemia during sepsis in the mouse.The Interleukin-17 Induced Activation and Increased Survival of Equine Neutrophils Is Insensitive to Glucocorticoids.Extremes of Interferon-Stimulated Gene Expression Associate with Worse Outcomes in the Acute Respiratory Distress Syndrome.Serum α-1 Antitrypsin (AAT) antagonizes intrinsic apoptosis induction in neutrophils from patients with systemic inflammatory response syndrome.Resolvin D1 attenuates inflammation in lipopolysaccharide-induced acute lung injury through a process involving the PPARγ/NF-κB pathwayFlavones induce neutrophil apoptosis by down-regulation of Mcl-1 via a proteasomal-dependent pathwayInduction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.TGF-β1 limits the onset of innate lung inflammation by promoting mast cell-derived IL-6Epithelial cell apoptosis and neutrophil recruitment in acute lung injury-a unifying hypothesis? What we have learned from small interfering RNAs.Mitochondrial mechanisms of sepsis-induced organ failure.Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity.Toll-like receptor 9 signaling delays neutrophil apoptosis by increasing transcription of Mcl-1.Downregulation of Mcl-1 has anti-inflammatory pro-resolution effects and enhances bacterial clearance from the lungPathways mediating resolution of inflammation: when enough is too much.Targeting neutrophil apoptosis for enhancing the resolution of inflammation.Multifaceted effects of Francisella tularensis on human neutrophil function and lifespan.HSF1 regulates expression of G-CSF through the binding element for NF-IL6/CCAAT enhancer binding protein beta.Acute lung injury: apoptosis in effector and target cells of the upper and lower airway compartment.Effects of ghrelin on the apoptosis of human neutrophils in vitroEffect of rapamycin on early stage apoptosis of neutrophils in Sprague-Dawley rats with acute lung injury.Intranasal curcumin ameliorates lipopolysaccharide-induced acute lung injury in mice.The cyclin-dependent kinase inhibitor AT7519 accelerates neutrophil apoptosis in sepsis-related acute respiratory distress syndrome.Glycogen synthase kinase-3β inactivation is an intracellular marker and regulator for endotoxemic neutrophilia.Vitamin C and Immune Function.Caspase-1-dependent pyroptosis of peripheral blood mononuclear cells predicts the development of sepsis in severe trauma patients: A prospective observational study.New Insights into the Immune Molecular Regulation of the Pathogenesis of Acute Respiratory Distress Syndrome.Endogenous PGI2 signaling through IP inhibits neutrophilic lung inflammation in LPS-induced acute lung injury mice model.Targeting apoptosis for optical imaging of infection.Extracellular Vesicles as Markers and Mediators in Sepsis.
P2860
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P2860
Neutrophil apoptosis: a marker of disease severity in sepsis and sepsis-induced acute respiratory distress syndrome.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@ast
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@en
type
label
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@ast
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@en
prefLabel
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@ast
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@en
P2093
P2860
P356
P1433
P1476
Neutrophil apoptosis: a marker ...... respiratory distress syndrome.
@en
P2093
Adriana R Milani
Edison M Rodrigues Filho
Eric Vachon
Gregory P Downey
João C Prolla
Luciano Fochesatto Filho
Léa Fialkow
Mary C Bozzetti
Paula Pierozan
Rafaela M de Moura
P2860
P2888
P356
10.1186/CC5090
P577
2006-01-01T00:00:00Z
P5875
P6179
1020739857